Heather E. McDermid
- Genetics top 0.5%
- Genomic variations and chromosomal abnormalities 21
- Genetics and Neurodevelopmental Disorders 9
- Molecular Biology top 5%
- Congenital heart defects research 17
- Genomics and Chromatin Dynamics 14
- Chromatin Remodeling and Cancer 6
- Ubiquitin and proteasome pathways 4
- Plant Science top 5%
- Chromosomal and Genetic Variations 13
- Cognitive Neuroscience top 5%
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- Peptidase Inhibition and Analysis 5
Heather E. McDermid
64 papers receiving 3.2k citations
Peers
Comparison fields: 5 of 108
- Genetics 2.0k
- Molecular Biology 2.1k
- Plant Science 712
- Cognitive Neuroscience 333
- Pediatrics, Perinatology and Child Health 294
Countries citing papers authored by Heather E. McDermid
This map shows the geographic impact of Heather E. McDermid's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Heather E. McDermid with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Heather E. McDermid more than expected).
Fields of papers citing papers by Heather E. McDermid
This network shows the impact of papers produced by Heather E. McDermid. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Heather E. McDermid. The network helps show where Heather E. McDermid may publish in the future.
Co-authorship network
The 25 scholars most cited alongside Heather E. McDermid, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2021 | 2 | |
| 2 | 2020 | 5 | |
| 3 | 2011 | 25 | |
| 4 | 2011 | 21 | |
| 5 | 2011 | 345 | |
| 6 | 2006 | 3 | |
| 7 | 2005 | 170 | |
| 8 | 2005 | 56 | |
| 9 | 2003 | 12 | |
| 10 | 2002 | 182 | |
| 11 | 2001 | 206 | |
| 12 | 2000 | 52 | |
| 13 | 1999 | 28 | |
| 14 | 1999 | 10 | |
| 15 | 1998 | 76 | |
| 16 | 1998 | 13 | |
| 17 | 1996 | 24 | |
| 18 | 1996 | 11 | |
| 19 | 1991 | 27 | |
| 20 | 1991 | 31 |
About Heather E. McDermid
Heather E. McDermid is a scholar working on Genetics, Molecular Biology, Plant Science, Oncology and Genetics, having authored 64 papers that have together received 3.3k indexed citations. Recurring topics across this work include Genomic variations and chromosomal abnormalities (21 papers), Congenital heart defects research (17 papers), Genomics and Chromatin Dynamics (14 papers), Chromosomal and Genetic Variations (13 papers), Genetics and Neurodevelopmental Disorders (9 papers), Chromatin Remodeling and Cancer (6 papers), Peptidase Inhibition and Analysis (5 papers) and Ubiquitin and proteasome pathways (4 papers). The work is most often cited by research in Genetics (2.0k citations), Molecular Biology (2.1k citations), Plant Science (712 citations), Cognitive Neuroscience (333 citations) and Pediatrics, Perinatology and Child Health (294 citations). Heather E. McDermid has collaborated with scholars based in Canada, United States and Japan. Frequent co-authors include Katy Phelan, Bernice E. Morrow, Jonathan Flint, Veronica J. Buckle, Andrew O.M. Wilkie, Robin M. Winter, Marcia L. Budarf, Anthony Holland, Alessandra M.V. Duncan and John Locke. Their work appears in journals such as Genomics, Human Molecular Genetics, Journal of Medical Genetics, Physiological Genomics and Immunogenetics.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.