He Zha

562 total citations
21 papers, 471 citations indexed

About

He Zha is a scholar working on Molecular Biology, Cancer Research and Epidemiology. According to data from OpenAlex, He Zha has authored 21 papers receiving a total of 471 indexed citations (citations by other indexed papers that have themselves been cited), including 14 papers in Molecular Biology, 6 papers in Cancer Research and 4 papers in Epidemiology. Recurrent topics in He Zha's work include S100 Proteins and Annexins (5 papers), Biomarkers in Disease Mechanisms (3 papers) and MicroRNA in disease regulation (3 papers). He Zha is often cited by papers focused on S100 Proteins and Annexins (5 papers), Biomarkers in Disease Mechanisms (3 papers) and MicroRNA in disease regulation (3 papers). He Zha collaborates with scholars based in China, United States and Thailand. He Zha's co-authors include Huimin Peng, Lan Zhou, Xueru Li, Liang Duan, Huan Li, Junxia Chen, Zheng Zhang, Tao Chen, Songtao Ding and Xiao-yan He and has published in prestigious journals such as FEBS Letters, Cellular and Molecular Life Sciences and Gene.

In The Last Decade

He Zha

19 papers receiving 465 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
He Zha China 13 330 184 74 51 40 21 471
Kenji Miyazaki Japan 11 268 0.8× 83 0.5× 71 1.0× 38 0.7× 34 0.8× 36 483
Haoneng Tang China 13 194 0.6× 69 0.4× 64 0.9× 65 1.3× 18 0.5× 35 440
Maryann Kaler United States 13 373 1.1× 133 0.7× 209 2.8× 43 0.8× 19 0.5× 19 706
Yafeng Zhang China 14 230 0.7× 47 0.3× 45 0.6× 46 0.9× 14 0.3× 40 520
Andrea Latini Italy 17 236 0.7× 143 0.8× 130 1.8× 44 0.9× 7 0.2× 47 659
Weijing Liu China 7 374 1.1× 59 0.3× 114 1.5× 66 1.3× 72 1.8× 15 543
Bangjie Chen China 13 231 0.7× 91 0.5× 133 1.8× 65 1.3× 12 0.3× 40 523
Gábor B. Brenner Hungary 9 295 0.9× 107 0.6× 45 0.6× 25 0.5× 9 0.2× 23 389
Kelly A. Conrads United States 9 223 0.7× 70 0.4× 24 0.3× 77 1.5× 88 2.2× 23 467
Zhuxian Zhu China 6 211 0.6× 92 0.5× 20 0.3× 75 1.5× 15 0.4× 13 310

Countries citing papers authored by He Zha

Since Specialization
Citations

This map shows the geographic impact of He Zha's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by He Zha with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites He Zha more than expected).

Fields of papers citing papers by He Zha

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by He Zha. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by He Zha. The network helps show where He Zha may publish in the future.

Co-authorship network of co-authors of He Zha

This figure shows the co-authorship network connecting the top 25 collaborators of He Zha. A scholar is included among the top collaborators of He Zha based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with He Zha. He Zha is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
2.
Huang, Xiaoxue, Xia An, Yan Wang, et al.. (2025). Development of a one-pot RPA-cas12a/13a assay for simultaneous detection of HPV16 and HPV18. Frontiers in Bioengineering and Biotechnology. 13. 1608301–1608301. 1 indexed citations
3.
Luo, Juan, Liang Li, Weiwei Zhang, et al.. (2025). Visual detection of Coxsackievirus A6 using a reverse transcription polymerase spiral reaction method. Frontiers in Cellular and Infection Microbiology. 15. 1563495–1563495.
4.
Zha, He, Tianhui Wu, Handeng Liu, et al.. (2024). Cytosolic Hmgb1 accumulation in mesangial cells aggravates diabetic kidney disease progression via NFκB signaling pathway. Cellular and Molecular Life Sciences. 81(1). 408–408. 9 indexed citations
5.
Zhang, Weiwei, Jie Wu, Xiaoqian Li, et al.. (2021). Molecular Epidemiology and Clinical Features of Enteroviruses-Associated Hand, Foot, and Mouth Disease and Herpangina Outbreak in Zunyi, China, 2019. Frontiers in Medicine. 8. 656699–656699. 15 indexed citations
6.
Zha, He, et al.. (2021). Elevated Serum Levels of Progranulin and Soluble Vascular Cell Adhesion Molecule-1 in Patients with COVID-19. Journal of Inflammation Research. Volume 14. 4785–4794. 15 indexed citations
7.
8.
Zha, He, Xueru Li, Hui Sun, et al.. (2019). S100A9 promotes the proliferation and migration of cervical cancer cells by inducing epithelial‑mesenchymal transition and activating the Wnt/β‑catenin pathway. International Journal of Oncology. 55(1). 35–44. 21 indexed citations
9.
Yu, Zhao, Kaifeng Wu, Yan Liu, et al.. (2018). Genotype patterns and prevalence of human papilloma virus in non-vaccinated women in Zunyi, China. Saudi Medical Journal. 39(6). 572–578. 5 indexed citations
10.
Gu, Yue, Aifang Li, Hui Sun, et al.. (2018). BCL6B suppresses proliferation and migration of colorectal carcinoma cells through inhibition of the PI3K/AKT signaling pathway. International Journal of Molecular Medicine. 41(5). 2660–2668. 11 indexed citations
11.
Li, Aifang, Yue Gu, Xueru Li, et al.. (2018). S100A6 promotes the proliferation and migration of cervical cancer cells via the PI3K/Akt signaling pathway. Oncology Letters. 15(4). 5685–5693. 31 indexed citations
12.
Zha, He, Hui Sun, Xueru Li, et al.. (2016). S100A8 facilitates the migration of colorectal cancer cells through regulating macrophages in the inflammatory microenvironment. Oncology Reports. 36(1). 279–290. 13 indexed citations
13.
Li, Huan, Min Yang, He Zha, et al.. (2016). High intensity focused ultrasound inhibits melanoma cell migration and metastasis through attenuating microRNA-21-mediated PTEN suppression. Oncotarget. 7(31). 50450–50460. 12 indexed citations
14.
Peng, Rui, Handeng Liu, Huimin Peng, et al.. (2015). Promoter hypermethylation of let-7a-3 is relevant to its down-expression in diabetic nephropathy by targeting UHRF1. Gene. 570(1). 57–63. 46 indexed citations
15.
Duan, Liang, Liwei Ye, Rui Wu, et al.. (2015). Inactivation of the Phosphatidylinositol 3‐Kinase/Akt Pathway is Involved in BMP9‐mediated Tumor‐suppressive Effects in Gastric Cancer Cells. Journal of Cellular Biochemistry. 116(6). 1080–1089. 20 indexed citations
16.
17.
Wang, Haiyan, Liang Duan, Zheng‐Yu Zou, et al.. (2014). Activation of the PI3K/Akt/mTOR/p70S6K Pathway is Involved in S100A4-induced Viability and Migration in Colorectal Cancer Cells. International Journal of Medical Sciences. 11(8). 841–849. 49 indexed citations
18.
Duan, Liang, Rui Wu, Zheng‐Yu Zou, et al.. (2013). S100A6 stimulates proliferation and migration of colorectal carcinoma cells through activation of the MAPK pathways. International Journal of Oncology. 44(3). 781–790. 37 indexed citations
19.
Peng, Rui, Huimin Peng, He Zha, et al.. (2013). A potentially functional polymorphism in the regulatory region of let-7a-2 is associated with an increased risk for diabetic nephropathy. Gene. 527(2). 456–461. 42 indexed citations
20.
Zhang, Zheng, Songtao Ding, Junxia Chen, et al.. (2011). MicroRNA‐451 regulates p38 MAPK signaling by targeting of Ywhaz and suppresses the mesangial hypertrophy in early diabetic nephropathy. FEBS Letters. 586(1). 20–26. 97 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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