G J Eaton

485 citations
16 papers · 370 indexed · h-index 11
Topics
Inflammatory mediators and NSAID effects (3 papers)Caveolin-1 and cellular processes (3 papers)Bone health and treatments (2 papers)

In The Last Decade

G J Eaton

16 papers receiving 348 citations

Peers

G J Eaton
Comparison fields: 5 of 73
  • Molecular Biology 175
  • Cell Biology 82
  • Immunology 69
  • Oncology 68
  • Cancer Research 61
Replace Kenji Miyazaki with:
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Countries citing papers authored by G J Eaton

Since Specialization
Citations

This map shows the geographic impact of G J Eaton's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by G J Eaton with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites G J Eaton more than expected).

Fields of papers citing papers by G J Eaton

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by G J Eaton. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by G J Eaton. The network helps show where G J Eaton may publish in the future.

Co-authorship network of co-authors of G J Eaton

This figure shows the co-authorship network connecting the top 25 collaborators of G J Eaton. A scholar is included among the top collaborators of G J Eaton based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with G J Eaton. G J Eaton is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

16 of 16 papers shown
#WorkIndexed citations
1 25
2 3
3 5
4 23
5 14
6
Apoptosis Signal-Regulating Kinase 1 Modulates Endochondral Bone Formation and Osteoarthritis Progression
1
7 28
8 2
9 95
10 46
11
Abstract 14567: Caveolin-1 Deficiency Exacerbates Cardiac Dysfunction and Reduces Survival in Mice with Myocardial Infarction
1
12
Influence of Sendai virus on carcinogenesis in strain A mice.
11
13
Spontaneous and induced tumor incidence in germfree "nude" mice.
65
14
Husbandry of the "nude" mouse in conventional and germfree environments.
11
15
Does the absence of immunologic surveillance affect the tumor incidence in "nude" mice? First recorded spontaneous lymphoma in a "nude" mouse.
11
16 29

About G J Eaton

G J Eaton is a scholar working on Clinical Biochemistry, Rheumatology and Cell Biology, having authored 16 papers that have together received 370 indexed citations. Recurring topics across this work include Inflammatory mediators and NSAID effects (3 papers), Caveolin-1 and cellular processes (3 papers) and Bone health and treatments (2 papers). The work is most often cited by research in Cell Biology (82 citations), Cancer Research (61 citations) and Immunology (69 citations). G J Eaton has collaborated with scholars based in United States, Jordan and United Kingdom. Frequent co-authors include Henry C. Outzen, R P Custer, Richmond T. Prehn, M. M. Green, Michael P. Lisanti, Theresa A. Freeman, Federica Sotgia, Ubaldo Martinez‐Outschoorn, Diana Whitaker‐Menezes and Stephanos Pavlides. Their work appears in journals such as Circulation, SHILAP Revista de lepidopterología and American Journal of Physiology-Heart and Circulatory Physiology.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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