Hava Ben‐David

887 total citations
12 papers, 313 citations indexed

About

Hava Ben‐David is a scholar working on Neurology, Immunology and Radiology, Nuclear Medicine and Imaging. According to data from OpenAlex, Hava Ben‐David has authored 12 papers receiving a total of 313 indexed citations (citations by other indexed papers that have themselves been cited), including 5 papers in Neurology, 5 papers in Immunology and 4 papers in Radiology, Nuclear Medicine and Imaging. Recurrent topics in Hava Ben‐David's work include Myasthenia Gravis and Thymoma (5 papers), Monoclonal and Polyclonal Antibodies Research (3 papers) and Coagulation, Bradykinin, Polyphosphates, and Angioedema (2 papers). Hava Ben‐David is often cited by papers focused on Myasthenia Gravis and Thymoma (5 papers), Monoclonal and Polyclonal Antibodies Research (3 papers) and Coagulation, Bradykinin, Polyphosphates, and Angioedema (2 papers). Hava Ben‐David collaborates with scholars based in Israel and United States. Hava Ben‐David's co-authors include Efrat Rorman, Chen Stein‐Zamir, Edna Mozes, Michael Sela, Amir Sharabi, Molly Dayan, Dror Luger, Heidy Zinger, Richard Bucala and Lin Leng and has published in prestigious journals such as Proceedings of the National Academy of Sciences, The Journal of Immunology and Cancer Research.

In The Last Decade

Hava Ben‐David

11 papers receiving 307 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Hava Ben‐David Israel 8 139 118 72 62 32 12 313
Norbert Blödorn‐Schlicht Germany 12 99 0.7× 35 0.3× 94 1.3× 66 1.1× 36 1.1× 20 335
Carole Leroy France 8 68 0.5× 73 0.6× 54 0.8× 110 1.8× 9 0.3× 18 342
Xiangshu Wen United States 12 334 2.4× 66 0.6× 99 1.4× 81 1.3× 30 0.9× 16 428
Stephen Nychay United States 9 101 0.7× 36 0.3× 29 0.4× 54 0.9× 28 0.9× 12 304
G.S. Baarsma Netherlands 10 60 0.4× 34 0.3× 88 1.2× 29 0.5× 69 2.2× 16 299
C. J. J. Brinkman Netherlands 8 24 0.2× 141 1.2× 95 1.3× 35 0.6× 81 2.5× 12 461
Elizabeth R. Bivins-Smith United States 6 251 1.8× 77 0.7× 285 4.0× 38 0.6× 26 0.8× 6 448
Maanasa Indaram United States 11 89 0.6× 56 0.5× 117 1.6× 95 1.5× 4 0.1× 24 420
Cristóbal Couto Argentina 11 21 0.2× 37 0.3× 64 0.9× 29 0.5× 93 2.9× 31 426
Jean‐Pierre Kusnierz France 8 181 1.3× 33 0.3× 29 0.4× 39 0.6× 59 1.8× 11 384

Countries citing papers authored by Hava Ben‐David

Since Specialization
Citations

This map shows the geographic impact of Hava Ben‐David's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Hava Ben‐David with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Hava Ben‐David more than expected).

Fields of papers citing papers by Hava Ben‐David

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Hava Ben‐David. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Hava Ben‐David. The network helps show where Hava Ben‐David may publish in the future.

Co-authorship network of co-authors of Hava Ben‐David

This figure shows the co-authorship network connecting the top 25 collaborators of Hava Ben‐David. A scholar is included among the top collaborators of Hava Ben‐David based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Hava Ben‐David. Hava Ben‐David is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

12 of 12 papers shown
1.
2.
Reuveni, Hadas, Mohammed Najeeb Al Hallak, Eliza W. Beal, et al.. (2024). Abstract 1939: NT219, a dual inhibitor of IRS1/2 and STAT3, suppresses cancer stem cell mediated resistance to KRASG12C and KRASG12D inhibitors in solid tumors. Cancer Research. 84(6_Supplement). 1939–1939. 1 indexed citations
3.
Ben‐David, Hava, Avi Livneh, Merav Lidar, et al.. (2018). Toll-like receptor 2 is overexpressed in Familial Mediterranean fever patients and is inhibited by colchicine treatment. Best Practice & Research Clinical Rheumatology. 32(5). 651–661. 14 indexed citations
4.
Ben‐David, Hava, Amir Sharabi, Heidy Zinger, et al.. (2010). A role for the B-cell CD74/macrophage migration inhibitory factor pathway in the immunomodulation of systemic lupus erythematosus by a therapeutic tolerogenic peptide. Immunology. 132(1). 87–95. 33 indexed citations
5.
Ben‐David, Hava, Amir Sharabi, Reshmi Parameswaran, Heidy Zinger, & Edna Mozes. (2009). A tolerogenic peptide down‐regulates mature B cells in bone marrow of lupus‐afflicted mice by inhibition of interleukin‐7, leading to apoptosis. Immunology. 128(2). 245–252. 6 indexed citations
6.
Sharabi, Amir, et al.. (2007). The Role of Apoptosis in the Ameliorating Effects of a CDR1-Based Peptide on Lupus Manifestations in a Mouse Model. The Journal of Immunology. 179(8). 4979–4987. 28 indexed citations
7.
Ben‐David, Hava, Amir Sharabi, Molly Dayan, Michael Sela, & Edna Mozes. (2007). The role of CD8+CD28−regulatory cells in suppressing myasthenia gravis-associated responses by a dual altered peptide ligand. Proceedings of the National Academy of Sciences. 104(44). 17459–17464. 47 indexed citations
8.
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Ben‐David, Hava, et al.. (2006). A 50-kDa ERK-like protein is up-regulated by a dual altered peptide ligand that suppresses myasthenia gravis-associated responses. Proceedings of the National Academy of Sciences. 103(48). 18232–18237. 9 indexed citations
11.
Rorman, Efrat, et al.. (2005). Congenital toxoplasmosis—prenatal aspects of Toxoplasma gondii infection. Reproductive Toxicology. 21(4). 458–472. 138 indexed citations
12.
Ben‐David, Hava, Michael Sela, & Edna Mozes. (2005). Down-regulation of myasthenogenic T cell responses by a dual altered peptide ligand via CD4+CD25+-regulated events leading to apoptosis. Proceedings of the National Academy of Sciences. 102(6). 2028–2033. 19 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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