Hannah M. Gibbons
- Neurology top 2%
- Molecular Biology
- Cellular and Molecular Neuroscience top 5%
- Immunology top 10%
- Developmental Neuroscience top 2%
- Co-authors
- Mike DragunowRichard L. M. FaullEdward MeeAmy M. SmithRobyn OldfieldMaurice A. CurtisJanusz LipskiMichael Dragunow
- Topics
- Neuroinflammation and Neurodegeneration Mechanisms (14 papers)Immune cells in cancer (5 papers)Neuroscience and Neuropharmacology Research (4 papers)
- Partner nations
- New ZealandSwedenSwitzerland
In The Last Decade
Hannah M. Gibbons
24 papers receiving 1.1k citations
Peers
Comparison fields: 5 of 94
- Neurology 482
- Molecular Biology 317
- Cellular and Molecular Neuroscience 298
- Immunology 246
- Developmental Neuroscience 190
Countries citing papers authored by Hannah M. Gibbons
This map shows the geographic impact of Hannah M. Gibbons's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Hannah M. Gibbons with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Hannah M. Gibbons more than expected).
Fields of papers citing papers by Hannah M. Gibbons
This network shows the impact of papers produced by Hannah M. Gibbons. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Hannah M. Gibbons. The network helps show where Hannah M. Gibbons may publish in the future.
Co-authorship network of co-authors of Hannah M. Gibbons
This figure shows the co-authorship network connecting the top 25 collaborators of Hannah M. Gibbons. A scholar is included among the top collaborators of Hannah M. Gibbons based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Hannah M. Gibbons. Hannah M. Gibbons is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 87 | |
| 2 | 7 | |
| 3 | 25 | |
| 4 | 46 | |
| 5 | 44 | |
| 6 | 38 | |
| 7 | 89 | |
| 8 | 22 | |
| 9 | 36 | |
| 10 | Tikanga in the Laboratory: Engaging Safe Practice | 3 |
| 11 | 112 | |
| 12 | 22 | |
| 13 | 59 | |
| 14 | 42 | |
| 15 | 64 | |
| 16 | 55 | |
| 17 | 3 | |
| 18 | 35 | |
| 19 | 24 | |
| 20 | 46 |
About Hannah M. Gibbons
Hannah M. Gibbons is a scholar working on Neurology, Developmental Neuroscience and Cellular and Molecular Neuroscience, having authored 24 papers that have together received 1.1k indexed citations. Recurring topics across this work include Neuroinflammation and Neurodegeneration Mechanisms (14 papers), Immune cells in cancer (5 papers) and Neuroscience and Neuropharmacology Research (4 papers). The work is most often cited by research in Neurology (482 citations), Developmental Neuroscience (190 citations) and Cellular and Molecular Neuroscience (298 citations). Hannah M. Gibbons has collaborated with scholars based in New Zealand, Sweden and Switzerland. Frequent co-authors include Mike Dragunow, Richard L. M. Faull, Edward Mee, Amy M. Smith, Robyn Oldfield, Maurice A. Curtis, Janusz Lipski, Michael Dragunow, Pritika Narayan and Alison M. Comer. Their work appears in journals such as Scientific Reports, Brain Research and Neuroscience.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.