Hai‐Lin Dong

799 total citations
23 papers, 553 citations indexed

About

Hai‐Lin Dong is a scholar working on Cellular and Molecular Neuroscience, Molecular Biology and Neurology. According to data from OpenAlex, Hai‐Lin Dong has authored 23 papers receiving a total of 553 indexed citations (citations by other indexed papers that have themselves been cited), including 16 papers in Cellular and Molecular Neuroscience, 15 papers in Molecular Biology and 12 papers in Neurology. Recurrent topics in Hai‐Lin Dong's work include Neurological diseases and metabolism (12 papers), Hereditary Neurological Disorders (10 papers) and Genetic Neurodegenerative Diseases (9 papers). Hai‐Lin Dong is often cited by papers focused on Neurological diseases and metabolism (12 papers), Hereditary Neurological Disorders (10 papers) and Genetic Neurodegenerative Diseases (9 papers). Hai‐Lin Dong collaborates with scholars based in China and United States. Hai‐Lin Dong's co-authors include Zhi‐Ying Wu, Junfeng Zhang, Shengpu Gao, Chen‐Yu Zhang, Lei Huang, Qi Sun, Xi Chen, Xiao Hu, Li Liang and Haijin Li and has published in prestigious journals such as Brain, Gene and Neurobiology of Aging.

In The Last Decade

Hai‐Lin Dong

23 papers receiving 548 citations

Peers

Countries citing papers authored by Hai‐Lin Dong

Since Specialization

Citations

This map shows the geographic impact of Hai‐Lin Dong's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Hai‐Lin Dong with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Hai‐Lin Dong more than expected).

Fields of papers citing papers by Hai‐Lin Dong

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Hai‐Lin Dong. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Hai‐Lin Dong. The network helps show where Hai‐Lin Dong may publish in the future.

Co-authorship network of co-authors of Hai‐Lin Dong

This figure shows the co-authorship network connecting the top 25 collaborators of Hai‐Lin Dong. A scholar is included among the top collaborators of Hai‐Lin Dong based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Hai‐Lin Dong. Hai‐Lin Dong is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

#	Work	Indexed citations
1	Biallelic variants in the COQ4 gene caused hereditary spastic paraplegia predominant phenotype 2023 ·CNS Neuroscience & Therapeutics ·(unknown), Hao Yu, (unknown), Juan‐Juan Xie, Hai‐Lin Dong, Zhi‐Ying Wu, Hong‐Fu Li	3
2	Identified novel heterozygous HTRA1 pathogenic variants in Chinese patients with HTRA1-associated dominant cerebral small vessel disease 2022 ·Frontiers in Genetics ·(unknown), Yi Zhang, (unknown), Hai‐Lin Dong, (unknown), Juan Zhang, (unknown), Ni Wang, Hong‐Fu Li	2
3	A novel frameshift ACTN2 variant causes a rare adult‐onset distal myopathy with multi‐minicores 2021 ·CNS Neuroscience & Therapeutics ·Lei Chen, (unknown), Hai‐Lin Dong, Gong‐Lu Liu, Zhi‐Ying Wu	10
4	Bi-allelic loss of function variants in COX20 gene cause autosomal recessive sensory neuronopathy 2021 ·Brain ·Hai‐Lin Dong, (unknown), Hao Yu, (unknown), Jiaqi Li, Gong‐Lu Liu, Hong‐Fu Li, Lei Chen, Dian-Fu Chen, Ge Bai, Zhi‐Ying Wu	15
5	Biallelic SORD pathogenic variants cause Chinese patients with distal hereditary motor neuropathy 2021 ·npj Genomic Medicine ·Hai‐Lin Dong, Jiaqi Li, Gong‐Lu Liu, Hao Yu, Zhi‐Ying Wu	18
6	Genetic spectrum and clinical features in a cohort of Chinese patients with autosomal recessive cerebellar ataxias 2021 ·Translational Neurodegeneration ·(unknown), (unknown), Yi Dong, Hai‐Lin Dong, (unknown), (unknown), Ying Shen, Zhi‐Ying Wu	13
7	A de novo variant of POLR3B causes demyelinating Charcot-Marie-Tooth disease in a Chinese patient: a case report 2021 ·BMC Neurology ·(unknown), (unknown), Hai‐Lin Dong, (unknown), Yun Yuan, Lingchao Meng, Zhi‐Ying Wu, Hao Yu	5
8	TGM6 might not be a specific causative gene for spinocerebellar ataxia resulting from genetic analysis and functional study 2021 ·Gene ·(unknown), Hai‐Lin Dong, Deshan Liu, Ni Wang, (unknown), (unknown), (unknown), Dian-Fu Chen, Yi Dong, Zhi‐Ying Wu	1
9	Genetic spectrum of MCM3AP and its relationship with phenotype of Charcot‐Marie‐Tooth disease 2020 ·Journal of the Peripheral Nervous System ·Hai‐Lin Dong, (unknown), Jiaqi Li, Hong‐Fu Li, Ge Bai, Huan Ma, Zhi‐Ying Wu	8
10	Genetic spectrum and clinical profiles in a southeast Chinese cohort of Charcot‐Marie‐Tooth disease 2019 ·Clinical Genetics ·(unknown), Hai‐Lin Dong, (unknown), (unknown), Hao Yu, Jiaqi Li, Gong‐Lu Liu, Hong‐Fu Li, Ge Bai, Huan Ma, Zhi‐Ying Wu	23
11	Functional study and pathogenicity classification of PRRT2 missense variants in PRRT2‐related disorders 2019 ·CNS Neuroscience & Therapeutics ·(unknown), Lixi Li, Yulan Chen, Yijun Chen, Gong‐Lu Liu, Hai‐Lin Dong, (unknown), Hong‐Fu Li, Zhi‐Ying Wu	13
12	Clinical and genetic characteristics of Chinese patients with cerebrotendinous xanthomatosis 2019 ·Orphanet Journal of Rare Diseases ·Qing‐Qing Tao, Yun Zhang, (unknown), Hai‐Lin Dong, Ni Wang, Zhi‐Ying Wu	14
13	Clinical features and genetic spectrum in Chinese patients with recessive hereditary spastic paraplegia 2019 ·Translational Neurodegeneration ·(unknown), Hai‐Lin Dong, (unknown), (unknown), Yang‐Tian Yan, (unknown), Hong‐Fu Li, Zhijun Liu, Qing‐Qing Tao, Zhi‐Ying Wu	35
14	Identification and functional characterization of mutations within HADHB associated with mitochondrial trifunctional protein deficiency 2019 ·Mitochondrion ·Zhirong Liu, Hai‐Lin Dong, (unknown), Zhi‐Ying Wu	5
15	Spastic paraplegia as the predominant phenotype in a cohort of Chinese patients with adrenoleukodystrophy 2019 ·Molecular Genetics & Genomic Medicine ·(unknown), (unknown), Hai‐Lin Dong, Yang‐Tian Yan, (unknown), Hong‐Fu Li	5
16	Genetic and clinical features of Chinese patients with mitochondrial ataxia identified by targeted next‐generation sequencing 2018 ·CNS Neuroscience & Therapeutics ·Hai‐Lin Dong, (unknown), (unknown), (unknown), (unknown), Sheng Chen, Zhi‐Ying Wu	5
17	Targeted next-generation sequencing improves diagnosis of hereditary spastic paraplegia in Chinese patients 2018 ·Journal of Molecular Medicine ·(unknown), Lixi Li, Hai‐Lin Dong, (unknown), Zhijun Liu, Ni Wang, Aaron D. Gitler, Zhi‐Ying Wu	23
18	A Novel Missense Mutation in Peripheral Myelin Protein-22 Causes Charcot-Marie-Tooth Disease 2017 ·Chinese Medical Journal ·Lixi Li, Hai‐Lin Dong, Bao‐Guo Xiao, Zhi‐Ying Wu	10
19	Clinical features of Chinese patients with Gerstmann–Sträussler–Scheinker identified by targeted next-generation sequencing 2016 ·Neurobiology of Aging ·Hong‐Fu Li, Zhijun Liu, Hai‐Lin Dong, Juan‐Juan Xie, (unknown), Ni Wang, Yi Dong, Zhi‐Ying Wu	13
20	Identification and characterization of microRNAs in raw milk during different periods of lactation, commercial fluid, and powdered milk products 2010 ·Cell Research ·Xi Chen, Chao Gao, Haijin Li, Lei Huang, Qi Sun, (unknown), Chunliang Tian, Shengpu Gao, Hai‐Lin Dong, (unknown), Xiao Hu, Shujian Zhao, Li Liang, Lin Zhu, Yan Qiao, Junfeng Zhang, Ke Zen, Chen‐Yu Zhang	310

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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