H. Stewart Hendrickson
- Molecular Biology top 5%
- Cell Biology top 5%
- Physiology top 10%
- Surgery
- Biochemistry top 2%
- Co-authors
- Oscar TousterNathan N. AronsonJohn T. DulaneyElizabeth K. HendricksonEdward A. DennisJ. G. FullingtonClinton E. BallouJames L. Reinertsen
- Topics
- Protein Kinase Regulation and GTPase Signaling (15 papers)Lipid Membrane Structure and Behavior (10 papers)Biomedical Research and Pathophysiology (8 papers)
- Partner nations
- United StatesRussiaNetherlands
In The Last Decade
H. Stewart Hendrickson
54 papers receiving 1.9k citations
Hit Papers
Peers
Comparison fields: 5 of 115
- Molecular Biology 1.3k
- Cell Biology 409
- Physiology 258
- Surgery 241
- Biochemistry 241
Countries citing papers authored by H. Stewart Hendrickson
This map shows the geographic impact of H. Stewart Hendrickson's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by H. Stewart Hendrickson with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites H. Stewart Hendrickson more than expected).
Fields of papers citing papers by H. Stewart Hendrickson
This network shows the impact of papers produced by H. Stewart Hendrickson. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by H. Stewart Hendrickson. The network helps show where H. Stewart Hendrickson may publish in the future.
Co-authorship network of co-authors of H. Stewart Hendrickson
This figure shows the co-authorship network connecting the top 25 collaborators of H. Stewart Hendrickson. A scholar is included among the top collaborators of H. Stewart Hendrickson based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with H. Stewart Hendrickson. H. Stewart Hendrickson is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 4 | |
| 2 | 4 | |
| 3 | 60 | |
| 4 | 3 | |
| 5 | 4 | |
| 6 | 8 | |
| 7 | 10 | |
| 8 | 9 | |
| 9 | 44 | |
| 10 | 26 | |
| 11 | 34 | |
| 12 | 20 | |
| 13 | 14 | |
| 14 | 12 | |
| 15 | 19 | |
| 16 | 8 | |
| 17 | 6 | |
| 18 | 4 | |
| 19 | 77 | |
| 20 | 4 |
About H. Stewart Hendrickson
H. Stewart Hendrickson is a scholar working on Filtration and Separation, Biochemistry and Cell Biology, having authored 54 papers that have together received 2.1k indexed citations. Recurring topics across this work include Protein Kinase Regulation and GTPase Signaling (15 papers), Lipid Membrane Structure and Behavior (10 papers) and Biomedical Research and Pathophysiology (8 papers). The work is most often cited by research in Biochemistry (241 citations), Cell Biology (409 citations) and Molecular Biology (1.3k citations). H. Stewart Hendrickson has collaborated with scholars based in United States, Russia and Netherlands. Frequent co-authors include Oscar Touster, Nathan N. Aronson, John T. Dulaney, Elizabeth K. Hendrickson, Edward A. Dennis, J. G. Fullington, Clinton E. Ballou, James L. Reinertsen, Iain Johnson and Steven Farber. Their work appears in journals such as Science, Journal of Biological Chemistry and The Journal of Cell Biology.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.