Jacqueline Heger

1.7k total citations
41 papers, 1.1k citations indexed

About

Jacqueline Heger is a scholar working on Molecular Biology, Cardiology and Cardiovascular Medicine and Physiology. According to data from OpenAlex, Jacqueline Heger has authored 41 papers receiving a total of 1.1k indexed citations (citations by other indexed papers that have themselves been cited), including 31 papers in Molecular Biology, 27 papers in Cardiology and Cardiovascular Medicine and 11 papers in Physiology. Recurrent topics in Jacqueline Heger's work include Cardiac Fibrosis and Remodeling (15 papers), Nitric Oxide and Endothelin Effects (9 papers) and Cardiac Ischemia and Reperfusion (8 papers). Jacqueline Heger is often cited by papers focused on Cardiac Fibrosis and Remodeling (15 papers), Nitric Oxide and Endothelin Effects (9 papers) and Cardiac Ischemia and Reperfusion (8 papers). Jacqueline Heger collaborates with scholars based in Germany, Hungary and Poland. Jacqueline Heger's co-authors include Gerhild Euler, Rainer Schulz, Axel Gödecke, Ulrich Flögel, Jürgen Schrader, H. M. Piper, Klaus‐Dieter Schlüter, Maryam Anwar, H. M. Piper and Andrei Molojavyi and has published in prestigious journals such as Journal of Biological Chemistry, Circulation Research and Scientific Reports.

In The Last Decade

Jacqueline Heger

41 papers receiving 1.1k citations

Peers

Jacqueline Heger
Scott M. MacDonnell United States
Attila Kiss Austria
Dongxing Zhu United Kingdom
Tang-Dong Liao United States
Federico Damilano United States
Jacqueline Heger
Citations per year, relative to Jacqueline Heger Jacqueline Heger (= 1×) peers Shinji Satoh

Countries citing papers authored by Jacqueline Heger

Since Specialization
Citations

This map shows the geographic impact of Jacqueline Heger's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Jacqueline Heger with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Jacqueline Heger more than expected).

Fields of papers citing papers by Jacqueline Heger

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Jacqueline Heger. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Jacqueline Heger. The network helps show where Jacqueline Heger may publish in the future.

Co-authorship network of co-authors of Jacqueline Heger

This figure shows the co-authorship network connecting the top 25 collaborators of Jacqueline Heger. A scholar is included among the top collaborators of Jacqueline Heger based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Jacqueline Heger. Jacqueline Heger is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Seidl, Matthias D., Hans-Gerd Kehl, Anne Schänzer, et al.. (2024). Cardiomyocyte maturation alters molecular stress response capacities and determines cell survival upon mitochondrial dysfunction. Free Radical Biology and Medicine. 213. 248–265. 5 indexed citations
2.
Heger, Jacqueline, Zoltán V. Varga, Tamás Baranyai, et al.. (2023). YB-1 Is a Novel Target for the Inhibition of α-Adrenergic-Induced Hypertrophy. International Journal of Molecular Sciences. 25(1). 401–401. 2 indexed citations
3.
Schreckenberg, Rolf, et al.. (2023). Monoamine Oxidase A Contributes to Serotonin—But Not Norepinephrine-Dependent Damage of Rat Ventricular Myocytes. Biomolecules. 13(6). 1013–1013. 1 indexed citations
4.
Bornbaum, Julia, Klaus‐Dieter Schlüter, Alessandra Ghigo, et al.. (2021). PI3K as Mediator of Apoptosis and Contractile Dysfunction in TGFβ1-Stimulated Cardiomyocytes. Biology. 10(7). 670–670. 4 indexed citations
5.
Euler, Gerhild, Jacqueline Heger, Rolf Schreckenberg, et al.. (2021). Matrix Metalloproteinases Repress Hypertrophic Growth in Cardiac Myocytes. Cardiovascular Drugs and Therapy. 35(2). 353–365. 15 indexed citations
6.
7.
Heger, Jacqueline, Julia Bornbaum, Akylbek Sydykov, et al.. (2021). Cardiomyocytes-specific deletion of monoamine oxidase B reduces irreversible myocardial ischemia/reperfusion injury. Free Radical Biology and Medicine. 165. 14–23. 24 indexed citations
8.
Lampe, Paul D., Jacqueline Heger, Klaus‐Dieter Schlüter, et al.. (2021). Connexin 43 phosphorylation by casein kinase 1 is essential for the cardioprotection by ischemic preconditioning. Basic Research in Cardiology. 116(1). 21–21. 32 indexed citations
9.
Parahuleva, Mariana S., Jens Kockskämper, Jacqueline Heger, et al.. (2020). Structural, Pro-Inflammatory and Calcium Handling Remodeling Underlies Spontaneous Onset of Paroxysmal Atrial Fibrillation in JDP2-Overexpressing Mice. International Journal of Molecular Sciences. 21(23). 9095–9095. 9 indexed citations
10.
Heger, Jacqueline, Julia Bornbaum, Renáta Gáspár, et al.. (2018). JDP2 overexpression provokes cardiac dysfunction in mice. Scientific Reports. 8(1). 7647–7647. 15 indexed citations
11.
Kleinbongard, Petra, Daniel Soetkamp, Jacqueline Heger, et al.. (2015). Interaction between Connexin 43 and nitric oxide synthase in mice heart mitochondria. Journal of Cellular and Molecular Medicine. 19(4). 815–825. 28 indexed citations
12.
Heger, Jacqueline, Y. Abdallah, Tayyab Shahzad, et al.. (2012). Transgenic overexpression of the adenine nucleotide translocase 1 protects cardiomyocytes against TGFβ1-induced apoptosis by stabilization of the mitochondrial permeability transition pore. Journal of Molecular and Cellular Cardiology. 53(1). 73–81. 17 indexed citations
13.
Vetter, Roland, et al.. (2011). Transgenic Overexpression of Heart-specific Adenine Nucleotide Translocase 1 Positively Affects Contractile Function in Cardiomyocytes. Cellular Physiology and Biochemistry. 27(2). 121–128. 7 indexed citations
14.
Wenzel, Sibylle, et al.. (2010). TGF-β1 improves cardiac performance via up-regulation of laminin receptor 37/67 in adult ventricular cardiomyocytes. Basic Research in Cardiology. 105(5). 621–629. 22 indexed citations
15.
Heger, Jacqueline, et al.. (2010). Growth differentiation factor 15 acts anti‐apoptotic and pro‐hypertrophic in adult cardiomyocytes. Journal of Cellular Physiology. 224(1). 120–126. 98 indexed citations
16.
Heger, Jacqueline, et al.. (2009). SMAD‐proteins as a molecular switch from hypertrophy to apoptosis induction in adult ventricular cardiomyocytes. Journal of Cellular Physiology. 220(2). 515–523. 13 indexed citations
17.
Schlüter, K.‐D., et al.. (2008). Enhanced SERCA2A expression improves contractile performance of ventricular cardiomyocytes of rat under adrenergic stimulation. Pflügers Archiv - European Journal of Physiology. 457(2). 485–491. 15 indexed citations
18.
Anwar, Maryam, et al.. (2006). Repression of anti-apoptotic genes via AP-1 as a mechanism of apoptosis induction in ventricular cardiomyocytes. Pflügers Archiv - European Journal of Physiology. 454(1). 53–61. 10 indexed citations
19.
Schröder, Dirk, Jacqueline Heger, H. M. Piper, & Gerhild Euler. (2006). Angiotensin II stimulates apoptosis via TGF-β1 signaling in ventricular cardiomyocytes of rat. Journal of Molecular Medicine. 84(11). 975–983. 51 indexed citations
20.
Heger, Jacqueline, et al.. (2005). The complex pattern of SMAD signaling in the cardiovascular system☆. Cardiovascular Research. 69(1). 15–25. 132 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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