H Itoh
- Molecular Biology
- Cellular and Molecular Neuroscience
- Physiology
- Cardiology and Cardiovascular Medicine
- Surgery
- Co-authors
- Fumikazu OkajimaM UiTomohiko IshikawaK. LederisH HidakaGozoh TsujimotoKuniko HorieMorley D. Hollenberg
- Topics
- Protein Kinase Regulation and GTPase Signaling (3 papers)Cardiovascular, Neuropeptides, and Oxidative Stress Research (2 papers)Nitric Oxide and Endothelin Effects (2 papers)
In The Last Decade
H Itoh
15 papers receiving 407 citations
Peers
Comparison fields: 5 of 73
- Molecular Biology 284
- Cellular and Molecular Neuroscience 79
- Physiology 71
- Cardiology and Cardiovascular Medicine 66
- Surgery 45
Countries citing papers authored by H Itoh
This map shows the geographic impact of H Itoh's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by H Itoh with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites H Itoh more than expected).
Fields of papers citing papers by H Itoh
This network shows the impact of papers produced by H Itoh. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by H Itoh. The network helps show where H Itoh may publish in the future.
Co-authorship network of co-authors of H Itoh
This figure shows the co-authorship network connecting the top 25 collaborators of H Itoh. A scholar is included among the top collaborators of H Itoh based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with H Itoh. H Itoh is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 47 | |
| 2 | 45 | |
| 3 | Effects of antihypertensive treatment on endothelial structure and endothelium-dependent relaxation in aortae of stroke-prone spontaneously hypertensive rats | 10 |
| 4 | 5 | |
| 5 | 18 | |
| 6 | 9 | |
| 7 | 5 | |
| 8 | 13 | |
| 9 | 29 | |
| 10 | 3 | |
| 11 | 76 | |
| 12 | 22 | |
| 13 | ADP-ribosylation of the inhibitory guanine nucleotide regulatory protein (Ni) as a possible mechanism underlying development of beta-adrenergic responses during primary culture of rat hepatocytes. | 9 |
| 14 | 51 | |
| 15 | 79 |
About H Itoh
H Itoh is a scholar working on Endocrinology, Diabetes and Metabolism, Pharmacology and Gastroenterology, having authored 15 papers that have together received 421 indexed citations. Recurring topics across this work include Protein Kinase Regulation and GTPase Signaling (3 papers), Cardiovascular, Neuropeptides, and Oxidative Stress Research (2 papers) and Nitric Oxide and Endothelin Effects (2 papers). The work is most often cited by research in Cellular and Molecular Neuroscience (79 citations), Molecular Biology (284 citations) and Cardiology and Cardiovascular Medicine (66 citations). H Itoh has collaborated with scholars based in Canada and Japan. Frequent co-authors include Fumikazu Okajima, M Ui, Tomohiko Ishikawa, K. Lederis, H Hidaka, Gozoh Tsujimoto, Kuniko Horie, Morley D. Hollenberg, Hisashi Hidaka and M. Saitoh. Their work appears in journals such as Journal of Biological Chemistry, Journal of Pharmacology and Experimental Therapeutics and British Journal of Pharmacology.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.