H. A. Jinnah

4.1k citations
55 papers · 2.8k indexed · h-index 32
Topics
Biochemical and Molecular Research (23 papers)Neurological disorders and treatments (21 papers)Genetic Neurodegenerative Diseases (18 papers)

In The Last Decade

H. A. Jinnah

55 papers receiving 2.7k citations

Peers

H. A. Jinnah
Comparison fields: 5 of 112
  • Neurology 1.1k
  • Molecular Biology 1.1k
  • Cellular and Molecular Neuroscience 930
  • Epidemiology 499
  • Infectious Diseases 268
Replace Takanori Yamagata with:
Takanori Yamagata Japan
Heinz Jungbluth United Kingdom
Bruria Ben‐Zeev Israel
Kouichi Ohta Japan
Guillermo M. Alexander United States
V. Ramaekers Germany
Pierangelo Veggiotti Italy
Christine Tranchant France
Gregor Kuhlenbäumer Germany
Nardo Nardocci Italy
H. A. Jinnah relative to Takanori Yamagata Japan Takanori Yamagata's profile →
Citations per field
00.5×3.3×
Takanori Yamagata · 1×
Citations per year

Countries citing papers authored by H. A. Jinnah

Since Specialization
Citations

This map shows the geographic impact of H. A. Jinnah's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by H. A. Jinnah with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites H. A. Jinnah more than expected).

Fields of papers citing papers by H. A. Jinnah

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by H. A. Jinnah. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by H. A. Jinnah. The network helps show where H. A. Jinnah may publish in the future.

Co-authorship network of co-authors of H. A. Jinnah

This figure shows the co-authorship network connecting the top 25 collaborators of H. A. Jinnah. A scholar is included among the top collaborators of H. A. Jinnah based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with H. A. Jinnah. H. A. Jinnah is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
#WorkIndexed citations
1 3
2 5
3 48
4 27
5 95
6 38
7 46
8 17
9 76
10 28
11 37
12 36
13 62
14 6
15 56
16 47
17 4
18 59
19 172
20 27

About H. A. Jinnah

H. A. Jinnah is a scholar working on Neurology, Cellular and Molecular Neuroscience and Physiology, having authored 55 papers that have together received 2.8k indexed citations. Recurring topics across this work include Biochemical and Molecular Research (23 papers), Neurological disorders and treatments (21 papers) and Genetic Neurodegenerative Diseases (18 papers). The work is most often cited by research in Neurology (1.1k citations), Cellular and Molecular Neuroscience (930 citations) and Physiology (178 citations). H. A. Jinnah has collaborated with scholars based in United States, Germany and France. Frequent co-authors include Ellen J. Hess, Theodore Friedmann, David J. Schretlen, James C. Harris, Vladimir Neychev, William L. Nyhan, Carolyn Pizoli, Xionglin Fan, Vanio Mitev and John P. O’Neill. Their work appears in journals such as Journal of Biological Chemistry, Journal of Neuroscience and Brain.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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