Gwendalyn D. King
- Molecular Biology top 10%
- Genetics top 2%
- Immunology top 5%
- Nephrology top 1%
- Oncology top 5%
- Co-authors
- María G. CastroPedro R. LöwensteinJames F. CurtinRaymond Scott TurnerMarianela CandolfiKurt M. KroegerMariana PuntelCarlos Barcia
- Topics
- Parathyroid Disorders and Treatments (22 papers)Genetic Syndromes and Imprinting (16 papers)Virus-based gene therapy research (13 papers)
- Cited by
- NephrologyGenetics
- Journals
- Journal of Biological ChemistryThe Journal of Experimental MedicineThe Journal of Cell Biology
- Partner nations
- United StatesUnited KingdomFrance
In The Last Decade
Gwendalyn D. King
57 papers receiving 2.8k citations
Peers
Comparison fields: 5 of 112
- Molecular Biology 1.2k
- Genetics 1.1k
- Immunology 588
- Nephrology 540
- Oncology 495
Countries citing papers authored by Gwendalyn D. King
This map shows the geographic impact of Gwendalyn D. King's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Gwendalyn D. King with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Gwendalyn D. King more than expected).
Fields of papers citing papers by Gwendalyn D. King
This network shows the impact of papers produced by Gwendalyn D. King. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Gwendalyn D. King. The network helps show where Gwendalyn D. King may publish in the future.
Co-authorship network of co-authors of Gwendalyn D. King
This figure shows the co-authorship network connecting the top 25 collaborators of Gwendalyn D. King. A scholar is included among the top collaborators of Gwendalyn D. King based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Gwendalyn D. King. Gwendalyn D. King is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 9 | |
| 2 | 15 | |
| 3 | 76 | |
| 4 | 35 | |
| 5 | 57 | |
| 6 | 12 | |
| 7 | 38 | |
| 8 | 30 | |
| 9 | 62 | |
| 10 | 37 | |
| 11 | 1 | |
| 12 | 80 | |
| 13 | 39 | |
| 14 | 68 | |
| 15 | 81 | |
| 16 | 3 | |
| 17 | 42 | |
| 18 | 29 | |
| 19 | 50 | |
| 20 | 3 |
About Gwendalyn D. King
Gwendalyn D. King is a scholar working on Nephrology, Genetics and Immunology, having authored 57 papers that have together received 2.9k indexed citations. Recurring topics across this work include Parathyroid Disorders and Treatments (22 papers), Genetic Syndromes and Imprinting (16 papers) and Virus-based gene therapy research (13 papers). The work is most often cited by research in Nephrology (540 citations), Genetics (1.1k citations) and Genetics (347 citations). Gwendalyn D. King has collaborated with scholars based in United States, United Kingdom and France. Frequent co-authors include María G. Castro, Pedro R. Löwenstein, James F. Curtin, Raymond Scott Turner, Marianela Candolfi, Kurt M. Kroeger, Mariana Puntel, Carlos Barcia, Weidong Xiong and Carmela R. Abraham. Their work appears in journals such as Journal of Biological Chemistry, The Journal of Experimental Medicine and The Journal of Cell Biology.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.