Günter Schneider

10.4k total citations · 3 hit papers
133 papers, 5.8k citations indexed

About

Günter Schneider is a scholar working on Molecular Biology, Oncology and Cancer Research. According to data from OpenAlex, Günter Schneider has authored 133 papers receiving a total of 5.8k indexed citations (citations by other indexed papers that have themselves been cited), including 84 papers in Molecular Biology, 58 papers in Oncology and 29 papers in Cancer Research. Recurrent topics in Günter Schneider's work include Pancreatic and Hepatic Oncology Research (35 papers), Epigenetics and DNA Methylation (24 papers) and Histone Deacetylase Inhibitors Research (20 papers). Günter Schneider is often cited by papers focused on Pancreatic and Hepatic Oncology Research (35 papers), Epigenetics and DNA Methylation (24 papers) and Histone Deacetylase Inhibitors Research (20 papers). Günter Schneider collaborates with scholars based in Germany, United States and France. Günter Schneider's co-authors include Dieter Saur, Roland M. Schmid, Angelika Schnieke, Oliver H. Krämer, Barbara Seidler, Jens T. Siveke, Roland Rad, Matthias Wirth, Florian Eckel and Marc Schmidt‐Supprian and has published in prestigious journals such as Nature, Proceedings of the National Academy of Sciences and Nucleic Acids Research.

In The Last Decade

Günter Schneider

130 papers receiving 5.7k citations

Hit Papers

align trajectories sort by overperformance

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Oncogenic KRAS signalling in pancreatic cancer

2014 395 citations Angelika Schnieke, Günter Schneider et al. profile →
Pancreatic ductal adenocarcinoma: biological hallmarks, current status, and future perspectives of combined modality treatment approaches

2019 326 citations Günter Schneider et al. profile →
Context-Specific Determinants of the Immunosuppressive Tumor Microenvironment in Pancreatic Cancer

2023 111 citations Chiara Falcomatà, Stefanie Bärthel et al. Cancer Discovery profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Günter Schneider Germany	40	3.4k	2.4k	1.2k	687	599	133	5.8k
Paul Yaswen United States	44	3.7k 1.1×	2.3k 0.9×	1.1k 0.9×	430 0.6×	382 0.6×	85	6.2k
Erik Bruyneel Belgium	47	4.7k 1.4×	2.2k 0.9×	1.3k 1.1×	727 1.1×	720 1.2×	123	7.3k
Benjamin P.C. Chen United States	41	4.8k 1.4×	1.7k 0.7×	974 0.8×	955 1.4×	304 0.5×	85	7.1k
Ambra Pozzi United States	53	4.0k 1.2×	1.3k 0.5×	1.4k 1.2×	744 1.1×	642 1.1×	173	8.5k
Peter Jung Germany	23	2.3k 0.7×	1.9k 0.8×	1.3k 1.1×	434 0.6×	334 0.6×	67	4.6k
Kevin M. Haigis United States	42	3.9k 1.1×	2.5k 1.1×	1.0k 0.8×	609 0.9×	431 0.7×	105	6.8k
Bo R. Rueda United States	47	2.7k 0.8×	1.7k 0.7×	1.2k 1.0×	1.3k 1.9×	284 0.5×	155	6.5k
Zhaojun Liu China	38	3.2k 0.9×	1.3k 0.5×	843 0.7×	1.3k 1.8×	658 1.1×	177	6.5k
Hong‐Jian Zhu Australia	48	5.4k 1.6×	2.3k 0.9×	2.0k 1.7×	1.0k 1.5×	599 1.0×	163	8.5k
Marie Arsenian‐Henriksson Sweden	41	4.3k 1.2×	1.5k 0.6×	1.6k 1.3×	783 1.1×	220 0.4×	101	6.3k

Countries citing papers authored by Günter Schneider

Since Specialization

Citations

This map shows the geographic impact of Günter Schneider's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Günter Schneider with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Günter Schneider more than expected).

Fields of papers citing papers by Günter Schneider

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Günter Schneider. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Günter Schneider. The network helps show where Günter Schneider may publish in the future.

Co-authorship network of co-authors of Günter Schneider

This figure shows the co-authorship network connecting the top 25 collaborators of Günter Schneider. A scholar is included among the top collaborators of Günter Schneider based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Günter Schneider. Günter Schneider is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Oliveira, Tiago De, et al.. (2025). Integrated real-time imaging of executioner caspase dynamics, apoptosis-induced proliferation, and immunogenic cell death using a stable fluorescent reporter platform. Cell Death Discovery. 11(1). 368–368.

Geismann, Claudia, Charlotte A. E. Hauser, Christian Schneeweis, et al.. (2023). NF-κB/RelA controlled A20 limits TRAIL-induced apoptosis in pancreatic cancer. Cell Death and Disease. 14(1). 3–3. 18 indexed citations

Falcomatà, Chiara, Stefanie Bärthel, Günter Schneider, et al.. (2023). Context-Specific Determinants of the Immunosuppressive Tumor Microenvironment in Pancreatic Cancer. Cancer Discovery. 13(2). 278–297. 111 indexed citations breakdown →

Bamopoulos, Stefanos A., Chuanbing Zang, Alexander T. den Dekker, et al.. (2022). NOXA expression drives synthetic lethality to RUNX1 inhibition in pancreatic cancer. Proceedings of the National Academy of Sciences. 119(9). 22 indexed citations

Kiweler, Nicole, Christina Susanne Mullins, Andrea Piée‐Staffa, et al.. (2022). The epigenetic modifier HDAC2 and the checkpoint kinase ATM determine the responses of microsatellite instable colorectal cancer cells to 5-fluorouracil. Cell Biology and Toxicology. 39(5). 2401–2419. 13 indexed citations

Yen, Yun, et al.. (2021). Class 1 Histone Deacetylases and Ataxia-Telangiectasia Mutated Kinase Control the Survival of Murine Pancreatic Cancer Cells upon dNTP Depletion. Cells. 10(10). 2520–2520. 5 indexed citations

Kiweler, Nicole, Desirée Wünsch, Matthias Wirth, et al.. (2020). Histone deacetylase inhibitors dysregulate DNA repair proteins and antagonize metastasis-associated processes. Journal of Cancer Research and Clinical Oncology. 146(2). 343–356. 39 indexed citations

Lange, Sebastian, Thomas Engleitner, Sebastian Mueller, et al.. (2020). Analysis pipelines for cancer genome sequencing in mice. Nature Protocols. 15(2). 266–315. 16 indexed citations

Beyer, Mandy, A Romański, Andrea Pautz, et al.. (2019). HDAC3 Activity is Essential for Human Leukemic Cell Growth and the Expression of β-catenin, MYC, and WT1. Cancers. 11(10). 1436–1436. 28 indexed citations

10.

Nikolova, Teodora, Nicole Kiweler, Toni Kühl, et al.. (2018). HDAC1 and HDAC2 integrate checkpoint kinase phosphorylation and cell fate through the phosphatase-2A subunit PR130. Nature Communications. 9(1). 764–764. 57 indexed citations

11.

Nathan, Abinaya, Nicole Kiweler, Carol Stocking, et al.. (2018). Loss of Wilms tumor 1 protein is a marker for apoptosis in response to replicative stress in leukemic cells. Archives of Toxicology. 92(6). 2119–2135. 13 indexed citations

12.

Yu, Peng, Svenja Wiechmann, Mathias Wilhelm, et al.. (2017). Trimodal Mixed Mode Chromatography That Enables Efficient Offline Two-Dimensional Peptide Fractionation for Proteome Analysis. Analytical Chemistry. 89(17). 8884–8891. 17 indexed citations

13.

Burstin, Johannes von, et al.. (2015). Detection of Tumor Suppressor Genes in Cancer Development by a Novel shRNA-Based Method. Molecular Cancer Research. 13(5). 863–869. 5 indexed citations

14.

Wirth, Matthias, Roland H. Stauber, Angelika Schnieke, et al.. (2012). MYC directs transcription of MCL1 and eIF4E genes to control sensitivity of gastric cancer cells toward HDAC inhibitors. Cell Cycle. 11(8). 1593–1602. 51 indexed citations

15.

Eser, Stefan, Marlena Messer, Philipp Eser, et al.. (2011). In vivo diagnosis of murine pancreatic intraepithelial neoplasia and early-stage pancreatic cancer by molecular imaging. Proceedings of the National Academy of Sciences. 108(24). 9945–9950. 67 indexed citations

16.

Schneider, Günter, et al.. (2009). Targeting histone deacetylases in pancreatic ductal adenocarcinoma. Journal of Cellular and Molecular Medicine. 14(6a). 1255–1263. 25 indexed citations

17.

Seidler, Barbara, Angelika Schnieke, Martin Göttlicher, et al.. (2009). HDAC2 mediates therapeutic resistance of pancreatic cancer cells via the BH3-only protein NOXA. Gut. 58(10). 1399–1409. 133 indexed citations

18.

Reichert, Maximilian, Dieter Saur, Rainer Hamacher, Roland M. Schmid, & Günter Schneider. (2007). Phosphoinositide-3-Kinase Signaling Controls S-Phase Kinase–Associated Protein 2 Transcription via E2F1 in Pancreatic Ductal Adenocarcinoma Cells. Cancer Research. 67(9). 4149–4156. 87 indexed citations

19.

Wichert, Götz von, et al.. (2005). Epidermal growth factor activates nuclear factor-κB in human proximal tubule cells. American Journal of Physiology-Renal Physiology. 289(4). F808–F815. 30 indexed citations

20.

Schneider, Günter, et al.. (2000). Experimental Investigation of Breakup of Turbulent Liquid Jets. Nature. 183(4671). 1324–5. 3 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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