Günter Schneider
- Molecular Biology top 2%
- Oncology top 1%
- Cancer Research top 1%
- Immunology top 5%
- Surgery top 5%
- Co-authors
- Dieter SaurRoland M. SchmidAngelika SchniekeOliver H. KrämerBarbara SeidlerJens T. SivekeRoland RadMatthias Wirth
- Topics
- Pancreatic and Hepatic Oncology Research (35 papers)Epigenetics and DNA Methylation (24 papers)Histone Deacetylase Inhibitors Research (20 papers)
- Partner nations
- GermanyUnited StatesFrance
In The Last Decade
Günter Schneider
130 papers receiving 5.7k citations
Hit Papers
Peers
Comparison fields: 5 of 146
- Molecular Biology 3.4k
- Oncology 2.4k
- Cancer Research 1.2k
- Immunology 687
- Surgery 599
Countries citing papers authored by Günter Schneider
This map shows the geographic impact of Günter Schneider's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Günter Schneider with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Günter Schneider more than expected).
Fields of papers citing papers by Günter Schneider
This network shows the impact of papers produced by Günter Schneider. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Günter Schneider. The network helps show where Günter Schneider may publish in the future.
Co-authorship network of co-authors of Günter Schneider
This figure shows the co-authorship network connecting the top 25 collaborators of Günter Schneider. A scholar is included among the top collaborators of Günter Schneider based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Günter Schneider. Günter Schneider is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 0 | |
| 2 | 18 | |
| 3 | Context-Specific Determinants of the Immunosuppressive Tumor Microenvironment in Pancreatic Cancerbreakdown → | 111 |
| 4 | 22 | |
| 5 | 13 | |
| 6 | 5 | |
| 7 | 16 | |
| 8 | 39 | |
| 9 | 28 | |
| 10 | 57 | |
| 11 | 17 | |
| 12 | 5 | |
| 13 | Oncogenic KRAS signalling in pancreatic cancerbreakdown → | 395 |
| 14 | 51 | |
| 15 | 67 | |
| 16 | 25 | |
| 17 | 133 | |
| 18 | 87 | |
| 19 | 30 | |
| 20 | 3 |
About Günter Schneider
Günter Schneider is a scholar working on Oncology, Cancer Research and Molecular Biology, having authored 133 papers that have together received 5.8k indexed citations. Recurring topics across this work include Pancreatic and Hepatic Oncology Research (35 papers), Epigenetics and DNA Methylation (24 papers) and Histone Deacetylase Inhibitors Research (20 papers). The work is most often cited by research in Oncology (2.4k citations), Cancer Research (1.2k citations) and Molecular Biology (3.4k citations). Günter Schneider has collaborated with scholars based in Germany, United States and France. Frequent co-authors include Dieter Saur, Roland M. Schmid, Angelika Schnieke, Oliver H. Krämer, Barbara Seidler, Jens T. Siveke, Roland Rad, Matthias Wirth, Florian Eckel and Marc Schmidt‐Supprian. Their work appears in journals such as Nature, Proceedings of the National Academy of Sciences and Nucleic Acids Research.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.