Thomas Engleitner

6.2k total citations · 1 hit paper
41 papers, 1.4k citations indexed

About

Thomas Engleitner is a scholar working on Molecular Biology, Immunology and Oncology. According to data from OpenAlex, Thomas Engleitner has authored 41 papers receiving a total of 1.4k indexed citations (citations by other indexed papers that have themselves been cited), including 16 papers in Molecular Biology, 16 papers in Immunology and 11 papers in Oncology. Recurrent topics in Thomas Engleitner's work include Immune Cell Function and Interaction (6 papers), Immunotherapy and Immune Responses (4 papers) and Parasites and Host Interactions (4 papers). Thomas Engleitner is often cited by papers focused on Immune Cell Function and Interaction (6 papers), Immunotherapy and Immune Responses (4 papers) and Parasites and Host Interactions (4 papers). Thomas Engleitner collaborates with scholars based in Germany, United States and United Kingdom. Thomas Engleitner's co-authors include Roland Rad, Julia Weber, Julia Slotta‐Huspenina, Dirk H. Busch, Matthias Mann, Angela M. Krackhardt, Eva Bräunlein, Richard Klar, Rüdiger Hein and Melanie Straub and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Clinical Investigation and Nature Communications.

In The Last Decade

Thomas Engleitner

41 papers receiving 1.3k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Direct identification of clinically relevant neoepitopes presented on native human melanoma tissue by mass spectrometry

2016 469 citations Thomas Engleitner, Julia Weber et al. Nature Communications profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Thomas Engleitner Germany	19	690	669	422	154	134	41	1.4k
Nicole Müller Germany	17	577 0.8×	783 1.2×	241 0.6×	210 1.4×	96 0.7×	33	1.3k
Dale R. Taylor United Kingdom	9	892 1.3×	522 0.8×	285 0.7×	87 0.6×	102 0.8×	12	1.6k
Gary K. Koski United States	27	1.8k 2.6×	771 1.2×	935 2.2×	150 1.0×	198 1.5×	57	2.4k
Timo Burster Germany	23	677 1.0×	510 0.8×	201 0.5×	293 1.9×	74 0.6×	73	1.4k
Robert K. Bright United States	22	497 0.7×	670 1.0×	477 1.1×	202 1.3×	130 1.0×	60	1.5k
Xiaoyan Qiu China	18	562 0.8×	477 0.7×	138 0.3×	68 0.4×	258 1.9×	46	1.1k
Hans-Joachim Schoenfeld Switzerland	6	1.0k 1.5×	708 1.1×	247 0.6×	300 1.9×	177 1.3×	6	1.7k
Zeling Cai United States	21	1.3k 1.9×	613 0.9×	296 0.7×	245 1.6×	70 0.5×	36	1.9k
Chance John Luckey United States	21	923 1.3×	892 1.3×	260 0.6×	68 0.4×	88 0.7×	42	1.8k
Bao Duong United States	17	1.3k 1.9×	885 1.3×	156 0.4×	245 1.6×	174 1.3×	28	1.9k

Countries citing papers authored by Thomas Engleitner

Since Specialization

Citations

This map shows the geographic impact of Thomas Engleitner's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Thomas Engleitner with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Thomas Engleitner more than expected).

Fields of papers citing papers by Thomas Engleitner

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Thomas Engleitner. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Thomas Engleitner. The network helps show where Thomas Engleitner may publish in the future.

Co-authorship network of co-authors of Thomas Engleitner

This figure shows the co-authorship network connecting the top 25 collaborators of Thomas Engleitner. A scholar is included among the top collaborators of Thomas Engleitner based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Thomas Engleitner. Thomas Engleitner is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Pan, Qi, Thomas Engleitner, Rupert Öllinger, et al.. (2024). CDK4/6 inhibition initiates cell cycle arrest by nuclear translocation of RB and induces a multistep molecular response. Cell Death Discovery. 10(1). 453–453. 6 indexed citations

Montero, Juán José, M C Sugden, Rupert Öllinger, et al.. (2024). Genome-scale pan-cancer interrogation of lncRNA dependencies using CasRx. Nature Methods. 21(4). 584–596. 19 indexed citations

Andersen, Line Lykke, Yiqi Huang, Christian Urban, et al.. (2023). Systematic P2Y receptor survey identifies P2Y11 as modulator of immune responses and virus replication in macrophages. The EMBO Journal. 42(23). e113279–e113279. 3 indexed citations

Bergant, Valter, Daniel Schnepf, Philipp Hubel, et al.. (2023). mRNA 3’UTR lengthening by alternative polyadenylation attenuates inflammatory responses and correlates with virulence of Influenza A virus. Nature Communications. 14(1). 4906–4906. 9 indexed citations

Linnerbauer, Mathias, Tobias A. Beyer, Hania Kébir, et al.. (2023). PD-L1 positive astrocytes attenuate inflammatory functions of PD-1 positive microglia in models of autoimmune neuroinflammation. Nature Communications. 14(1). 5555–5555. 33 indexed citations

Ecker, Veronika, Piero Giansanti, Aida Varela-Moreira, et al.. (2023). Negative feedback regulation of MAPK signaling is an important driver of chronic lymphocytic leukemia progression. Cell Reports. 42(10). 113017–113017. 7 indexed citations

Sie, Christopher, Ravi Kant, Christian Peter, et al.. (2022). IL-24 intrinsically regulates Th17 cell pathogenicity in mice. The Journal of Experimental Medicine. 219(8). 16 indexed citations

Mitsdoerffer, Meike, Lilian Aly, Melanie Barz, et al.. (2022). The glioblastoma multiforme tumor site promotes the commitment of tumor-infiltrating lymphocytes to the T_H17 lineage in humans. Proceedings of the National Academy of Sciences. 119(34). e2206208119–e2206208119. 16 indexed citations

Ecker, Veronika, Thomas Engleitner, Ingo Ringshausen, et al.. (2021). Targeted PI3K/AKT-hyperactivation induces cell death in chronic lymphocytic leukemia. Nature Communications. 12(1). 3526–3526. 50 indexed citations

10.

Lechner, Markus, Thomas Engleitner, Marc Schmidt‐Supprian, et al.. (2021). Notch2-mediated plasticity between marginal zone and follicular B cells. Nature Communications. 12(1). 1111–1111. 31 indexed citations

11.

Fischer, Julius, Erik Thiele Orberg, Dirk H. Busch, et al.. (2021). IFN-Gamma Producing Regulatory T Cells Counterbalance T Cell-Mediated Injury to the Intestinal Stem Cell Compartment in Mice and Humans. Blood. 138(Supplement 1). 89–89. 1 indexed citations

12.

Lange, Sebastian, Thomas Engleitner, Sebastian Mueller, et al.. (2020). Analysis pipelines for cancer genome sequencing in mice. Nature Protocols. 15(2). 266–315. 16 indexed citations

13.

Kumar, Dilip, Maciej Lech, Thomas Engleitner, et al.. (2020). c-Rel gain in B cells drives germinal center reactions and autoantibody production. Journal of Clinical Investigation. 130(6). 3270–3286. 10 indexed citations

14.

Rosenbaum, Marc, Andreas Gewies, Konstanze Pechloff, et al.. (2019). Bcl10-controlled Malt1 paracaspase activity is key for the immune suppressive function of regulatory T cells. Nature Communications. 10(1). 2352–2352. 60 indexed citations

15.

Heidegger, Simon, Alexander Wintges, Sarah Bek, et al.. (2019). RIG-I activation is critical for responsiveness to checkpoint blockade. Science Immunology. 4(39). 96 indexed citations

16.

Wang, Baocai, Benedikt Kaufmann, Thomas Engleitner, et al.. (2019). Brg1 promotes liver regeneration after partial hepatectomy via regulation of cell cycle. Scientific Reports. 9(1). 2320–2320. 25 indexed citations

17.

Knier, Benjamin, Michael Hiltensperger, Christopher Sie, et al.. (2018). Myeloid-derived suppressor cells control B cell accumulation in the central nervous system during autoimmunity. Nature Immunology. 19(12). 1341–1351. 81 indexed citations

18.

Preussner, Jens, Jiasheng Zhong, Stefan Günther, et al.. (2018). Oncogenic Amplification of Zygotic Dux Factors in Regenerating p53-Deficient Muscle Stem Cells Defines a Molecular Cancer Subtype. Cell stem cell. 23(6). 794–805.e4. 18 indexed citations

19.

Friedrich, Mathias, Lena Rad, Iraad F. Bronner, et al.. (2017). Genome-wide transposon screening and quantitative insertion site sequencing for cancer gene discovery in mice. Nature Protocols. 12(2). 289–309. 26 indexed citations

20.

Lalremruata, Albert, Sankarganesh Jeyaraj, Thomas Engleitner, et al.. (2017). Species and genotype diversity of Plasmodium in malaria patients from Gabon analysed by next generation sequencing. Malaria Journal. 16(1). 398–398. 48 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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