Giovanni Defazio
- Neurology top 1%
- Cellular and Molecular Neuroscience top 10%
- Physiology
- Cognitive Neuroscience
- Pathology and Forensic Medicine
- Co-authors
- Alfredo BerardelliMark HallettHyder A. JinnahGiovanni FabbriniAngelo Fabio GiganteMichèle TinazziJoseph JankovicMatteo Bologna
- Topics
- Neurological disorders and treatments (25 papers)Botulinum Toxin and Related Neurological Disorders (19 papers)Parkinson's Disease Mechanisms and Treatments (15 papers)
- Partner nations
- ItalyUnited StatesUnited Kingdom
In The Last Decade
Giovanni Defazio
31 papers receiving 903 citations
Peers
Comparison fields: 5 of 69
- Neurology 807
- Cellular and Molecular Neuroscience 247
- Physiology 106
- Cognitive Neuroscience 77
- Pathology and Forensic Medicine 68
Countries citing papers authored by Giovanni Defazio
This map shows the geographic impact of Giovanni Defazio's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Giovanni Defazio with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Giovanni Defazio more than expected).
Fields of papers citing papers by Giovanni Defazio
This network shows the impact of papers produced by Giovanni Defazio. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Giovanni Defazio. The network helps show where Giovanni Defazio may publish in the future.
Co-authorship network of co-authors of Giovanni Defazio
This figure shows the co-authorship network connecting the top 25 collaborators of Giovanni Defazio. A scholar is included among the top collaborators of Giovanni Defazio based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Giovanni Defazio. Giovanni Defazio is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 0 | |
| 2 | 3 | |
| 3 | 0 | |
| 4 | 0 | |
| 5 | 0 | |
| 6 | 1 | |
| 7 | 0 | |
| 8 | 29 | |
| 9 | 1 | |
| 10 | 3 | |
| 11 | 3 | |
| 12 | 6 | |
| 13 | 9 | |
| 14 | 1 | |
| 15 | 1 | |
| 16 | 61 | |
| 17 | 54 | |
| 18 | 61 | |
| 19 | 42 | |
| 20 | 121 |
About Giovanni Defazio
Giovanni Defazio is a scholar working on Neurology, Genetics and Cellular and Molecular Neuroscience, having authored 37 papers that have together received 919 indexed citations. Recurring topics across this work include Neurological disorders and treatments (25 papers), Botulinum Toxin and Related Neurological Disorders (19 papers) and Parkinson's Disease Mechanisms and Treatments (15 papers). The work is most often cited by research in Neurology (807 citations), Cellular and Molecular Neuroscience (247 citations) and Sensory Systems (46 citations). Giovanni Defazio has collaborated with scholars based in Italy, United States and United Kingdom. Frequent co-authors include Alfredo Berardelli, Mark Hallett, Hyder A. Jinnah, Giovanni Fabbrini, Angelo Fabio Gigante, Michèle Tinazzi, Joseph Jankovic, Matteo Bologna, Luca Marsili and Jennifer L. Giel. Their work appears in journals such as Neurology, Journal of Neurology Neurosurgery & Psychiatry and Movement Disorders.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.