Gilles Edan

5.3k citations
66 papers · 1.8k indexed · h-index 21

Gilles Edan

61 papers receiving 1.7k citations

Peers

Gilles Edan
Comparison fields: 5 of 105
  • Pathology and Forensic Medicine 903
  • Neurology 668
  • Hematology 255
  • Rheumatology 318
  • Neurology 126
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Citations per field
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Citations per year

Countries citing papers authored by Gilles Edan

Since Specialization
Citations

This map shows the geographic impact of Gilles Edan's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Gilles Edan with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Gilles Edan more than expected).

Fields of papers citing papers by Gilles Edan

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Gilles Edan. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Gilles Edan. The network helps show where Gilles Edan may publish in the future.

Co-authorship network

The 25 scholars most cited alongside Gilles Edan, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Gilles Edan Line = papers co-authored together Gilles Edan links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown
#Work
1 20222
2 20200
3 20187
4 201811
5 20161
6 20165
7
Serum 25-hydroxyvitamin D concentrations among patients in BENEFIT predicts conversion to multiple sclerosis, MRI lesions, and brain volume loss
20122
8 201010
9 2010406
10 20063
11 200510
12 20056
13 200577
14 20033
15
[Treatment of progressive forms of multiple sclerosis].
20013
16 19984
17 199428
18
[Intrasynovial secretion of antibrucella antibodies].
19790
19
[Demonstration by iron overloading study and HLA genotyping of recessive transmission of idiopathic haemochromatosis in two pseudodominant pedigrees (author's transl)].
19793
20
Hérédité récessive de l'hémochromatose idiopathique: deux observations de transmission pseudo-dominante reconnue comme récessive par l'étude de la surcharge en fer et des génotypes HLA dan les familles.
19793

About Gilles Edan

Gilles Edan is a scholar working on Pathology and Forensic Medicine, Neurology and Rheumatology, having authored 66 papers that have together received 1.8k indexed citations. Recurring topics across this work include Multiple Sclerosis Research Studies (29 papers), Peripheral Neuropathies and Disorders (13 papers), Polyomavirus and related diseases (7 papers), Rheumatoid Arthritis Research and Therapies (6 papers), Iron Metabolism and Disorders (5 papers), Thyroid Disorders and Treatments (4 papers), Systemic Sclerosis and Related Diseases (4 papers) and Diabetes and associated disorders (3 papers). The work is most often cited by research in Pathology and Forensic Medicine (903 citations), Neurology (668 citations) and Hematology (255 citations). Gilles Edan has collaborated with scholars based in France, Switzerland and Spain. Frequent co-authors include Jacqueline Yaouanq, Emmanuelle Le Page, Marc Coustans, Emmanuelle Leray, David Laplaud, Joël Oger, Marc Vérin, R Fauchet, M Bourel and M. Simon. Their work appears in journals such as New England Journal of Medicine, Gastroenterology and Brain.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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