Géza Schermann

1.4k total citations
18 papers, 985 citations indexed

About

Géza Schermann is a scholar working on Molecular Biology, Public Health, Environmental and Occupational Health and Cell Biology. According to data from OpenAlex, Géza Schermann has authored 18 papers receiving a total of 985 indexed citations (citations by other indexed papers that have themselves been cited), including 11 papers in Molecular Biology, 3 papers in Public Health, Environmental and Occupational Health and 3 papers in Cell Biology. Recurrent topics in Géza Schermann's work include Acute Lymphoblastic Leukemia research (3 papers), RNA and protein synthesis mechanisms (3 papers) and RNA Research and Splicing (3 papers). Géza Schermann is often cited by papers focused on Acute Lymphoblastic Leukemia research (3 papers), RNA and protein synthesis mechanisms (3 papers) and RNA Research and Splicing (3 papers). Géza Schermann collaborates with scholars based in Germany, Hungary and United States. Géza Schermann's co-authors include Tamás Fischer, Irmgard Sinning, Nikolay Dobrev, Carmen Ruiz de Almodóvar, Thomas Schmidt, Aı̈da Valls, Xiaohong Wang, Ying Shen, Gergely Solecki and Laura Castro and has published in prestigious journals such as Cell, Nature Communications and Neuron.

In The Last Decade

Géza Schermann

18 papers receiving 980 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Géza Schermann Germany 11 738 263 114 100 72 18 985
Idan Cohen Israel 18 669 0.9× 189 0.7× 135 1.2× 77 0.8× 58 0.8× 34 1.0k
Srinath C. Sampath United States 11 1.0k 1.4× 501 1.9× 99 0.9× 76 0.8× 143 2.0× 14 1.3k
Ramsay J. McFarlane United Kingdom 19 766 1.0× 179 0.7× 69 0.6× 102 1.0× 122 1.7× 40 888
Cynthia A. Sparks United States 9 912 1.2× 653 2.5× 137 1.2× 71 0.7× 79 1.1× 9 1.3k
Olga Ermakova Italy 16 440 0.6× 90 0.3× 66 0.6× 65 0.7× 62 0.9× 29 725
Jianguo Fan United States 17 545 0.7× 86 0.3× 155 1.4× 85 0.8× 49 0.7× 37 737
Irina Matos Portugal 13 546 0.7× 384 1.5× 173 1.5× 162 1.6× 90 1.3× 17 853
Madhvi B. Upender United States 15 587 0.8× 193 0.7× 152 1.3× 215 2.1× 87 1.2× 21 1000
Aude Robert France 15 706 1.0× 206 0.8× 116 1.0× 52 0.5× 52 0.7× 34 945
Cristina Claverı́a Spain 11 592 0.8× 316 1.2× 83 0.7× 71 0.7× 13 0.2× 11 835

Countries citing papers authored by Géza Schermann

Since Specialization
Citations

This map shows the geographic impact of Géza Schermann's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Géza Schermann with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Géza Schermann more than expected).

Fields of papers citing papers by Géza Schermann

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Géza Schermann. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Géza Schermann. The network helps show where Géza Schermann may publish in the future.

Co-authorship network of co-authors of Géza Schermann

This figure shows the co-authorship network connecting the top 25 collaborators of Géza Schermann. A scholar is included among the top collaborators of Géza Schermann based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Géza Schermann. Géza Schermann is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

18 of 18 papers shown
1.
Schermann, Géza, Christian E. Zimmerli, Nikola Kellner, et al.. (2023). Identification and characterization of sugar-regulated promoters in Chaetomium thermophilum. BMC Biotechnology. 23(1). 19–19. 1 indexed citations
2.
3.
Shah, Bhavin, Sebastián Dupraz, Patricia Himmels, et al.. (2022). Endothelial PlexinD1 signaling instructs spinal cord vascularization and motor neuron development. Neuron. 110(24). 4074–4089.e6. 14 indexed citations
4.
Singh, Amit, et al.. (2021). Global Transcriptome Characterization and Assembly of the Thermophilic Ascomycete Chaetomium thermophilum. Genes. 12(10). 1549–1549. 6 indexed citations
5.
Shen, Ying, Xiaohong Wang, Yi Liu, et al.. (2021). STAT3-YAP/TAZ signaling in endothelial cells promotes tumor angiogenesis. Science Signaling. 14(712). eabj8393–eabj8393. 90 indexed citations
6.
Luck, Robert, Andromachi Karakatsani, Bhavin Shah, et al.. (2021). The angiopoietin-Tie2 pathway regulates Purkinje cell dendritic morphogenesis in a cell-autonomous manner. Cell Reports. 36(7). 109522–109522. 7 indexed citations
7.
Shostak, Anton, Géza Schermann, Axel Diernfellner, & Michael Brunner. (2021). MXD/MIZ1 transcription regulatory complexes activate the expression of MYC‐repressed genes. FEBS Letters. 595(12). 1639–1655. 3 indexed citations
8.
Shah, Bhavin, Heike Adler, Melanie Richter, et al.. (2021). Oligodendrocyte precursor cell specification is regulated by bidirectional neural progenitor–endothelial cell crosstalk. Nature Neuroscience. 24(4). 478–488. 21 indexed citations
9.
Wang, Xiaohong, Aı̈da Valls, Géza Schermann, et al.. (2017). YAP/TAZ Orchestrate VEGF Signaling during Developmental Angiogenesis. Developmental Cell. 42(5). 462–478.e7. 273 indexed citations
10.
Ha, Nati, et al.. (2017). The coding and noncoding transcriptome of Neurospora crassa. BMC Genomics. 18(1). 978–978. 22 indexed citations
11.
Schermann, Géza, Klemens Wild, Tamás Fischer, et al.. (2017). Structural basis for 5'-ETS recognition by Utp4 at the early stages of ribosome biogenesis. PLoS ONE. 12(6). e0178752–e0178752. 4 indexed citations
12.
Yang, Xiaoqing, András Gézsi, Géza Schermann, et al.. (2017). HLA-DRB1*07:01–HLA-DQA1*02:01–HLA-DQB1*02:02 haplotype is associated with a high risk of asparaginase hypersensitivity in acute lymphoblastic leukemia. Haematologica. 102(9). 1578–1586. 29 indexed citations
13.
Schermann, Géza, et al.. (2016). A Novel Epigenetic Silencing Pathway Involving the Highly Conserved 5’-3’ Exoribonuclease Dhp1/Rat1/Xrn2 in Schizosaccharomyces pombe. PLoS Genetics. 12(2). e1005873–e1005873. 31 indexed citations
14.
Schermann, Géza, et al.. (2016). Transient RNA-DNA Hybrids Are Required for Efficient Double-Strand Break Repair. Cell. 167(4). 1001–1013.e7. 306 indexed citations
15.
Gumiero, Andrea, Charlotte Conz, Ying Zhang, et al.. (2016). Interaction of the cotranslational Hsp70 Ssb with ribosomal proteins and rRNA depends on its lid domain. Nature Communications. 7(1). 13563–13563. 42 indexed citations
16.
Zhou, Yang, et al.. (2015). The fission yeast MTREC complex targets CUTs and unspliced pre-mRNAs to the nuclear exosome. Nature Communications. 6(1). 7050–7050. 90 indexed citations
17.
Schermann, Géza, Orsolya Lautner‐Csorba, Ferenc Rárosi, et al.. (2015). Impact of single nucleotide polymorphisms of cytarabine metabolic genes on drug toxicity in childhood acute lymphoblastic leukemia. Pediatric Blood & Cancer. 62(4). 622–628. 7 indexed citations
18.
Lautner‐Csorba, Orsolya, András Gézsi, Ágnes F. Semsei, et al.. (2012). Candidate gene association study in pediatric acute lymphoblastic leukemia evaluated by Bayesian network based Bayesian multilevel analysis of relevance. BMC Medical Genomics. 5(1). 42–42. 36 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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