Gersina Rega‐Kaun
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- Atherosclerosis and Cardiovascular Diseases 2
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- Protease and Inhibitor Mechanisms 3
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- Adipokines, Inflammation, and Metabolic Diseases 3
- Chronic Disease Management Strategies 2
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- Cytokine Signaling Pathways and Interactions 3
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- Bariatric Surgery and Outcomes 2
- Lipoproteins and Cardiovascular Health 2
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- Adipose Tissue and Metabolism 2
Gersina Rega‐Kaun
20 papers receiving 515 citations
Peers
Comparison fields: 5 of 73
- Immunology 147
- Cancer Research 92
- Immunology and Allergy 32
- Cardiology and Cardiovascular Medicine 104
- Internal Medicine 17
Countries citing papers authored by Gersina Rega‐Kaun
This map shows the geographic impact of Gersina Rega‐Kaun's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Gersina Rega‐Kaun with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Gersina Rega‐Kaun more than expected).
Fields of papers citing papers by Gersina Rega‐Kaun
This network shows the impact of papers produced by Gersina Rega‐Kaun. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Gersina Rega‐Kaun. The network helps show where Gersina Rega‐Kaun may publish in the future.
Co-authorship network
The 25 scholars most cited alongside Gersina Rega‐Kaun, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2025 | 0 | |
| 2 | 2024 | 8 | |
| 3 | 2024 | 1 | |
| 4 | 2021 | 6 | |
| 5 | 2020 | 15 | |
| 6 | 2019 | 23 | |
| 7 | 2019 | 15 | |
| 8 | 2019 | 19 | |
| 9 | 2018 | 35 | |
| 10 | 2018 | 16 | |
| 11 | 2013 | 35 | |
| 12 | Oncostatin M and IL-6 induce u-PA and VEGF in prostate cancer cells and correlate in vivo. | 2011 | 23 |
| 13 | 2010 | 53 | |
| 14 | 2009 | 51 | |
| 15 | 2007 | 27 | |
| 16 | 2006 | 39 | |
| 17 | The estrogen metabolite 17beta-dihydroequilenin counteracts interleukin-1alpha induced expression of inflammatory mediators in human endothelial cells in vitro via NF-kappaB pathway. | 2006 | 16 |
| 18 | 2006 | 49 | |
| 19 | 2005 | 18 | |
| 20 | 2004 | 27 |
About Gersina Rega‐Kaun
Gersina Rega‐Kaun is a scholar working on Internal Medicine, Immunology and Pharmacy, having authored 21 papers that have together received 516 indexed citations. Recurring topics across this work include Adipokines, Inflammation, and Metabolic Diseases (3 papers), Protease and Inhibitor Mechanisms (3 papers), Cytokine Signaling Pathways and Interactions (3 papers), Chronic Disease Management Strategies (2 papers), Bariatric Surgery and Outcomes (2 papers), Lipoproteins and Cardiovascular Health (2 papers), Atherosclerosis and Cardiovascular Diseases (2 papers) and Adipose Tissue and Metabolism (2 papers). The work is most often cited by research in Immunology (147 citations), Cancer Research (92 citations) and Immunology and Allergy (32 citations). Gersina Rega‐Kaun has collaborated with scholars based in Austria, United States and Slovakia. Frequent co-authors include Christoph Kaun, Johann Wojta, Gerald Maurer, Kurt Huber, Svitlana Demyanets, Philipp J. Hohensinner, Stefan Pfaffenberger, Stefan Kastl, Walter S. Speidl and Manfred Prager. Their work appears in journals such as Blood, FEBS Letters and International Journal of Molecular Sciences.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.