Stefan Pfaffenberger

2.2k total citations
42 papers, 1.7k citations indexed

About

Stefan Pfaffenberger is a scholar working on Cardiology and Cardiovascular Medicine, Molecular Biology and Radiology, Nuclear Medicine and Imaging. According to data from OpenAlex, Stefan Pfaffenberger has authored 42 papers receiving a total of 1.7k indexed citations (citations by other indexed papers that have themselves been cited), including 16 papers in Cardiology and Cardiovascular Medicine, 13 papers in Molecular Biology and 10 papers in Radiology, Nuclear Medicine and Imaging. Recurrent topics in Stefan Pfaffenberger's work include Cardiac Imaging and Diagnostics (8 papers), Atherosclerosis and Cardiovascular Diseases (8 papers) and Cardiovascular Function and Risk Factors (7 papers). Stefan Pfaffenberger is often cited by papers focused on Cardiac Imaging and Diagnostics (8 papers), Atherosclerosis and Cardiovascular Diseases (8 papers) and Cardiovascular Function and Risk Factors (7 papers). Stefan Pfaffenberger collaborates with scholars based in Austria, Slovakia and United Kingdom. Stefan Pfaffenberger's co-authors include Gerald Maurer, Kurt Huber, Johann Wojta, Christoph Kaun, Julia Mascherbauer, Stefan Kastl, Svitlana Demyanets, Diana Bonderman, Walter S. Speidl and Beatrice A. Marzluf and has published in prestigious journals such as Journal of the American College of Cardiology, PLoS ONE and Stroke.

In The Last Decade

Stefan Pfaffenberger

41 papers receiving 1.7k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Stefan Pfaffenberger Austria	27	684	453	388	332	281	42	1.7k
Stefan Kastl Austria	25	425 0.6×	608 1.3×	424 1.1×	327 1.0×	80 0.3×	69	1.7k
Hans‐Reiner Figulla Germany	24	804 1.2×	170 0.4×	562 1.4×	322 1.0×	261 0.9×	75	1.6k
Hiroto Tsujioka Japan	21	824 1.2×	427 0.9×	1.0k 2.7×	273 0.8×	732 2.6×	32	1.9k
Hideyuki Ikejima Japan	21	922 1.3×	420 0.9×	1.1k 2.9×	260 0.8×	610 2.2×	31	1.9k
K‐Raman Purushothaman United States	20	658 1.0×	411 0.9×	772 2.0×	523 1.6×	419 1.5×	44	2.2k
Yoshihisa Shimada Japan	14	697 1.0×	327 0.7×	695 1.8×	160 0.5×	488 1.7×	52	1.6k
Alexander Schmeißer Germany	27	865 1.3×	499 1.1×	448 1.2×	847 2.6×	91 0.3×	94	2.5k
Mercè Roqué Spain	26	880 1.3×	395 0.9×	831 2.1×	826 2.5×	258 0.9×	72	2.9k
R Ripa Denmark	27	723 1.1×	162 0.4×	667 1.7×	734 2.2×	424 1.5×	115	2.2k
Jason M. Tarkin United Kingdom	21	539 0.8×	223 0.5×	419 1.1×	179 0.5×	613 2.2×	72	1.5k

Countries citing papers authored by Stefan Pfaffenberger

Since Specialization

Citations

This map shows the geographic impact of Stefan Pfaffenberger's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Stefan Pfaffenberger with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Stefan Pfaffenberger more than expected).

Fields of papers citing papers by Stefan Pfaffenberger

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Stefan Pfaffenberger. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Stefan Pfaffenberger. The network helps show where Stefan Pfaffenberger may publish in the future.

Co-authorship network of co-authors of Stefan Pfaffenberger

This figure shows the co-authorship network connecting the top 25 collaborators of Stefan Pfaffenberger. A scholar is included among the top collaborators of Stefan Pfaffenberger based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Stefan Pfaffenberger. Stefan Pfaffenberger is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Hohensinner, Philipp J., Christoph Kaun, Barbara Thaler, et al.. (2017). Urokinase plasminogen activator protects cardiac myocytes from oxidative damage and apoptosis via hOGG1 induction. APOPTOSIS. 22(8). 1048–1055. 18 indexed citations

Karakuş, Gültekin, Andreas A. Kammerlander, Stefan Aschauer, et al.. (2015). Pulmonary artery to aorta ratio for the detection of pulmonary hypertension: cardiovascular magnetic resonance and invasive hemodynamics in heart failure with preserved ejection fraction. Journal of Cardiovascular Magnetic Resonance. 17(1). 79–79. 38 indexed citations

Kammerlander, Andreas A., Beatrice A. Marzluf, Caroline Zotter‐Tufaro, et al.. (2015). T1 Mapping by CMR Imaging. JACC. Cardiovascular imaging. 9(1). 14–23. 156 indexed citations

Krychtiuk, Konstantin A., Stefan Kastl, Sebastian Hofbauer, et al.. (2015). Monocyte subset distribution in patients with stable atherosclerosis and elevated levels of lipoprotein(a). Journal of clinical lipidology. 9(4). 533–541. 37 indexed citations

Aschauer, Stefan, Andreas A. Kammerlander, Caroline Zotter‐Tufaro, et al.. (2015). The Right Heart in Heart Failure with Preserved Ejection Fraction: Insights from Cardiac Magnetic Resonance Imaging and Invasive Haemodynamics. European Journal of Heart Failure. 18(1). 71–80. 115 indexed citations

Mascherbauer, Julia, Beatrice A. Marzluf, Caroline Tufaro, et al.. (2013). EXTENT OF DIFFUSE MYOCARDIAL FIBROSIS DETERMINES OUTCOME IN PATIENTS WITH HEART FAILURE AND PRESERVED EJECTION FRACTION. Journal of the American College of Cardiology. 61(10). E827–E827. 1 indexed citations

Pfaffenberger, Stefan, Philipp E. Bartko, Alexandra Gráf, et al.. (2013). Size Matters! Impact of Age, Sex, Height, and Weight on the Normal Heart Size. Circulation Cardiovascular Imaging. 6(6). 1073–1079. 78 indexed citations

Marzluf, Beatrice A., Diana Bonderman, Caroline Tufaro, et al.. (2013). Diffuse myocardial fibrosis by post-contrast T1-time predicts outcome in heart failure with preserved ejection fraction. Journal of Cardiovascular Magnetic Resonance. 15. M6–M6. 2 indexed citations

Demyanets, Svitlana, Christoph Kaun, Richard Pentz, et al.. (2013). Components of the interleukin-33/ST2 system are differentially expressed and regulated in human cardiac cells and in cells of the cardiac vasculature. Journal of Molecular and Cellular Cardiology. 60. 16–26. 138 indexed citations

10.

Haidinger, Michael, Johannes Werzowa, Renate Kain, et al.. (2013). Hereditary amyloidosis caused by R554L fibrinogen Aα-chain mutation in a Spanish family and review of the literature. Amyloid. 20(2). 72–79. 15 indexed citations

11.

Demyanets, Svitlana, Christoph Kaun, Kathrin Rychli, et al.. (2010). Oncostatin M-enhanced vascular endothelial growth factor expression in human vascular smooth muscle cells involves PI3K-, p38 MAPK-, Erk1/2- and STAT1/STAT3-dependent pathways and is attenuated by interferon-γ. Basic Research in Cardiology. 106(2). 217–231. 53 indexed citations

12.

Demyanets, Svitlana, Christoph Kaun, Kathrin Rychli, et al.. (2007). The inflammatory cytokine oncostatin M induces PAI-1 in human vascular smooth muscle cells in vitro via PI 3-kinase and ERK1/2-dependent pathways. American Journal of Physiology-Heart and Circulatory Physiology. 293(3). H1962–H1968. 27 indexed citations

13.

Hohensinner, Philipp J., Christoph Kaun, Kathrin Rychli, et al.. (2007). Macrophage colony stimulating factor expression in human cardiac cells is upregulated by tumor necrosis factor‐α via an NF‐κB dependent mechanism. Journal of Thrombosis and Haemostasis. 5(12). 2520–2528. 29 indexed citations

14.

Demyanets, Svitlana, Stefan Pfaffenberger, Christoph Kaun, et al.. (2006). The estrogen metabolite 17beta-dihydroequilenin counteracts interleukin-1alpha induced expression of inflammatory mediators in human endothelial cells in vitro via NF-kappaB pathway.. PubMed. 95(1). 107–16. 16 indexed citations

15.

Demyanets, Svitlana, Christoph Kaun, Stefan Pfaffenberger, et al.. (2006). Hydroxymethylglutaryl-coenzyme A reductase inhibitors induce apoptosis in human cardiac myocytes in vitro. Biochemical Pharmacology. 71(9). 1324–1330. 49 indexed citations

16.

Weiss, T, Heda Kvakan, Christoph Kaun, et al.. (2006). No evidence for a direct role of Helicobacter pylori and Mycoplasma pneumoniae in carotid artery atherosclerosis. Journal of Clinical Pathology. 59(11). 1186–1190. 11 indexed citations

17.

Kaun, Christoph, Gersina Rega‐Kaun, Walter S. Speidl, et al.. (2005). The estrogen metabolite 17β-dihydroequilenin counteracts interleukin-1α induced expression of inflammatory mediators in human endothelial cells in vitro via NF-κB pathway. Thrombosis and Haemostasis. 95(1). 107–116. 18 indexed citations

18.

Weiss, Theresa R., Heda Kvakan, Christoph Kaun, et al.. (2005). The gp130 ligand oncostatin M regulates tissue inhibitor of metalloproteinases-1 through ERK1/2 and p38 in human adult cardiac myocytes and in human adult cardiac fibroblasts: A possible role for the gp130/gp130 ligand system in the modulation of extracellular matrix degradation in the human heart. Journal of Molecular and Cellular Cardiology. 39(3). 545–551. 35 indexed citations

19.

Pfaffenberger, Stefan, Stefan Kastl, Gerald Maurer, et al.. (2005). Ultrasound thrombolysis. Thrombosis and Haemostasis. 94(7). 26–36. 35 indexed citations

20.

Pfaffenberger, Stefan, Christian Kollmann, Stefan Kastl, et al.. (2004). CAN A COMMERCIAL DIAGNOSTIC ULTRASOUND DEVICE ACCELERATE THROMBOLYSIS?—AN IN VITRO STROKE MODEL. Cardiovascular Pathology. 13(3). 176–176. 1 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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