Ge Lu

2.2k total citations · 1 hit paper
36 papers, 1.6k citations indexed

About

Ge Lu is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience and Neurology. According to data from OpenAlex, Ge Lu has authored 36 papers receiving a total of 1.6k indexed citations (citations by other indexed papers that have themselves been cited), including 18 papers in Molecular Biology, 12 papers in Cellular and Molecular Neuroscience and 8 papers in Neurology. Recurrent topics in Ge Lu's work include Genetic Neurodegenerative Diseases (12 papers), Mitochondrial Function and Pathology (11 papers) and Muscle Physiology and Disorders (7 papers). Ge Lu is often cited by papers focused on Genetic Neurodegenerative Diseases (12 papers), Mitochondrial Function and Pathology (11 papers) and Muscle Physiology and Disorders (7 papers). Ge Lu collaborates with scholars based in Canada, China and United States. Ge Lu's co-authors include Blair R. Leavitt, Michael R. Hayden, Rona K. Graham, Nagat Bissada, Elizabeth Slow, Lynn A. Raymond, Jacqueline Pearson, Yu Deng, Zoe Murphy and Jacqueline Shehadeh and has published in prestigious journals such as Cell, Proceedings of the National Academy of Sciences and Journal of Neuroscience.

In The Last Decade

Ge Lu

33 papers receiving 1.6k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Cleavage at the Caspase-6 Site Is Required for Neuronal Dysfunction and Degeneration Due to Mutant Huntingtin

2006 505 citations Rona K. Graham, Yu Deng et al. Cell profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Ge Lu Canada	17	1.1k	1.0k	541	221	128	36	1.6k
Xavier Xifró Spain	22	638 0.6×	635 0.6×	284 0.5×	125 0.6×	96 0.8×	36	1.1k
Chuan-En Wang United States	17	1.3k 1.2×	1.2k 1.2×	498 0.9×	200 0.9×	232 1.8×	21	1.9k
Iga Wegorzewska United States	11	889 0.8×	371 0.4×	750 1.4×	222 1.0×	175 1.4×	12	1.5k
Omar L. Nelson United States	14	833 0.8×	817 0.8×	215 0.4×	656 3.0×	117 0.9×	23	1.6k
P. Elyse Schauwecker United States	20	729 0.7×	1.0k 1.0×	130 0.2×	137 0.6×	238 1.9×	32	1.6k
Yuxiang Xie United States	16	735 0.7×	632 0.6×	291 0.5×	259 1.2×	142 1.1×	18	1.5k
Michael Klinkenberg Germany	17	737 0.7×	333 0.3×	634 1.2×	313 1.4×	134 1.0×	19	1.4k
Sandro Alves France	21	1.1k 1.0×	810 0.8×	265 0.5×	328 1.5×	173 1.4×	33	1.8k
José Aguilera Spain	20	530 0.5×	562 0.6×	395 0.7×	177 0.8×	73 0.6×	60	1.0k
Isao Nishimura Japan	17	953 0.9×	548 0.5×	422 0.8×	545 2.5×	169 1.3×	25	1.7k

Countries citing papers authored by Ge Lu

Since Specialization

Citations

This map shows the geographic impact of Ge Lu's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Ge Lu with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Ge Lu more than expected).

Fields of papers citing papers by Ge Lu

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Ge Lu. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Ge Lu. The network helps show where Ge Lu may publish in the future.

Co-authorship network of co-authors of Ge Lu

This figure shows the co-authorship network connecting the top 25 collaborators of Ge Lu. A scholar is included among the top collaborators of Ge Lu based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Ge Lu. Ge Lu is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

Sort: Min cites: Since: Top N: Style:

20 of 20 papers shown

Zhu, Yaoyao, et al.. (2025). Interstitial fluid transport in linea alba is involved in acupuncture-induced attenuation of ovarian hypofunction in aged rats. Frontiers in Endocrinology. 16. 1579031–1579031.

Gomes, A., Terri L. Petkau, Oriol Fornés, et al.. (2024). New MiniPromoter Ple389 (ADORA2A) drives selective expression in medium spiny neurons in mice and non-human primates. Scientific Reports. 14(1). 28194–28194.

Lu, Ge, et al.. (2024). Moxibustion prevents tripterygium glycoside-induced oligoasthenoteratozoospermia in rats via reduced oxidative stress and modulation of the Nrf2/HO-1 signaling pathway. Aging. 16(3). 2141–2160. 6 indexed citations

Lu, Ge, et al.. (2024). Combination of bone marrow mesenchymal stem cells and moxibustion restores cyclophosphamide-induced premature ovarian insufficiency by improving mitochondrial function and regulating mitophagy. Stem Cell Research & Therapy. 15(1). 102–102. 18 indexed citations

Raamsdonk, Jeremy M. Van, Timothy W. Bredy, Jacqueline Pearson, et al.. (2023). Huntingtin Decreases Susceptibility to a Spontaneous Seizure Disorder in FVN/B Mice. Aging and Disease. 14(6). 2249–2249. 1 indexed citations

Chen, Ting, et al.. (2022). Moxibustion mitigates mitochondrial dysfunction and NLRP3 inflammatory activation in cyclophosphamide-induced premature ovarian insufficiency rats. Life Sciences. 314. 121283–121283. 25 indexed citations

Shi, Ling, Ge Lu, Qian Wang, et al.. (2022). Moxibustion alleviates decreased ovarian reserve in rats by restoring the PI3K/AKT signaling pathway. Journal of Integrative Medicine. 20(2). 163–172. 26 indexed citations

Lu, Ge, et al.. (2022). Effects of acupuncture treatment on microRNAs expression in ovarian tissues from Tripterygium glycoside-induced diminished ovarian reserve rats. Frontiers in Genetics. 13. 968711–968711. 7 indexed citations

Galván, Adriana, Terri L. Petkau, Austin Hill, et al.. (2021). Intracerebroventricular Administration of AAV9-PHP.B SYN1-EmGFP Induces Widespread Transgene Expression in the Mouse and Monkey Central Nervous System. Human Gene Therapy. 32(11-12). 599–615. 22 indexed citations

10.

Petkau, Terri L., Ge Lu, Oriol Fornés, et al.. (2021). Human progranulin-expressing mice as a novel tool for the development of progranulin-modulating therapeutics. Neurobiology of Disease. 153. 105314–105314. 9 indexed citations

11.

Franciosi, Sonia, et al.. (2017). p35 hemizygosity activates Akt but does not improve motor function in the YAC128 mouse model of Huntington’s disease. Neuroscience. 352. 79–87. 6 indexed citations

12.

Petkau, Terri L., et al.. (2013). Sensitivity to neurotoxic stress is not increased in progranulin-deficient mice. Neurobiology of Aging. 34(11). 2548–2550. 8 indexed citations

13.

Budac, David, et al.. (2013). The absence of indoleamine 2,3-dioxygenase expression protects against NMDA receptor-mediated excitotoxicity in mouse brain. Experimental Neurology. 249. 144–148. 27 indexed citations

14.

Petkau, Terri L., Scott J. Neal, Austen J. Milnerwood, et al.. (2011). Synaptic dysfunction in progranulin-deficient mice. Neurobiology of Disease. 45(2). 711–722. 125 indexed citations

15.

Petkau, Terri L., Scott J. Neal, Paul C. Orban, et al.. (2010). Progranulin expression in the developing and adult murine brain. The Journal of Comparative Neurology. 518(19). 3931–3947. 112 indexed citations

16.

Metzler, Martina, Lu Gan, Rona K. Graham, et al.. (2010). Phosphorylation of Huntingtin at Ser⁴²¹in YAC128 Neurons Is Associated with Protection of YAC128 Neurons from NMDA-Mediated Excitotoxicity and Is Modulated by PP1 and PP2A. Journal of Neuroscience. 30(43). 14318–14329. 70 indexed citations

17.

Graham, Rona K., Mahmoud A. Pouladi, Prasad Joshi, et al.. (2009). Differential Susceptibility to Excitotoxic Stress in YAC128 Mouse Models of Huntington Disease between Initiation and Progression of Disease. Journal of Neuroscience. 29(7). 2193–2204. 98 indexed citations

18.

Graham, Rona K., Yu Deng, Elizabeth Slow, et al.. (2006). Cleavage at the Caspase-6 Site Is Required for Neuronal Dysfunction and Degeneration Due to Mutant Huntingtin. Cell. 125(6). 1179–1191. 505 indexed citations breakdown →

19.

Raamsdonk, Jeremy M. Van, William T. Gibson, Jacqueline Pearson, et al.. (2006). Body weight is modulated by levels of full-length Huntingtin. Human Molecular Genetics. 15(9). 1513–1523. 87 indexed citations

20.

Graham, Rona K., Elizabeth Slow, Yu Deng, et al.. (2005). Levels of mutant huntingtin influence the phenotypic severity of Huntington disease in YAC128 mouse models. Neurobiology of Disease. 21(2). 444–455. 69 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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