Chuan-En Wang

2.3k total citations
21 papers, 1.9k citations indexed

About

Chuan-En Wang is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience and Neurology. According to data from OpenAlex, Chuan-En Wang has authored 21 papers receiving a total of 1.9k indexed citations (citations by other indexed papers that have themselves been cited), including 17 papers in Molecular Biology, 17 papers in Cellular and Molecular Neuroscience and 5 papers in Neurology. Recurrent topics in Chuan-En Wang's work include Genetic Neurodegenerative Diseases (16 papers), Mitochondrial Function and Pathology (14 papers) and Muscle Physiology and Disorders (7 papers). Chuan-En Wang is often cited by papers focused on Genetic Neurodegenerative Diseases (16 papers), Mitochondrial Function and Pathology (14 papers) and Muscle Physiology and Disorders (7 papers). Chuan-En Wang collaborates with scholars based in United States, China and Poland. Chuan-En Wang's co-authors include Xiao‐Jiang Li, Shihua Li, Ji‐Yeon Shin, Zhihui Fang, Zhao-Xue Yu, Adam L. Orr, Shihua Li, Xingshun Xu, Sen Yan and He Li and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and The Journal of Experimental Medicine.

In The Last Decade

Chuan-En Wang

20 papers receiving 1.8k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Chuan-En Wang United States 17 1.3k 1.2k 498 232 200 21 1.9k
Siddhartha Mitra United States 14 1.6k 1.2× 1.5k 1.2× 563 1.1× 130 0.6× 320 1.6× 24 2.6k
Jessica E. Young United States 20 1.2k 0.9× 772 0.6× 266 0.5× 207 0.9× 586 2.9× 52 1.9k
Ge Lu Canada 17 1.1k 0.8× 1.0k 0.8× 541 1.1× 128 0.6× 221 1.1× 36 1.6k
Wendou Yu United States 16 874 0.7× 577 0.5× 232 0.5× 126 0.5× 282 1.4× 20 1.3k
Till G.A. Mack Germany 16 670 0.5× 684 0.6× 216 0.4× 214 0.9× 371 1.9× 21 1.5k
Elisabetta Babetto United States 16 620 0.5× 760 0.6× 225 0.5× 211 0.9× 241 1.2× 24 1.4k
Ismael Al‐Ramahi United States 22 1.2k 0.9× 956 0.8× 269 0.5× 93 0.4× 229 1.1× 41 1.6k
Antoni Matilla‐Dueñas Spain 21 1.5k 1.1× 1.2k 1.0× 476 1.0× 146 0.6× 83 0.4× 37 1.8k
Sílvia Porta United States 20 1.4k 1.0× 423 0.3× 664 1.3× 228 1.0× 359 1.8× 27 2.0k
Paymaan Jafar‐Nejad United States 23 1.6k 1.2× 920 0.8× 610 1.2× 212 0.9× 243 1.2× 55 2.3k

Countries citing papers authored by Chuan-En Wang

Since Specialization
Citations

This map shows the geographic impact of Chuan-En Wang's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Chuan-En Wang with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Chuan-En Wang more than expected).

Fields of papers citing papers by Chuan-En Wang

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Chuan-En Wang. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Chuan-En Wang. The network helps show where Chuan-En Wang may publish in the future.

Co-authorship network of co-authors of Chuan-En Wang

This figure shows the co-authorship network connecting the top 25 collaborators of Chuan-En Wang. A scholar is included among the top collaborators of Chuan-En Wang based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Chuan-En Wang. Chuan-En Wang is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Liu, Xudong, Chuan-En Wang, Yan Hong, et al.. (2016). N-terminal Huntingtin Knock-In Mice: Implications of Removing the N-terminal Region of Huntingtin for Therapy. PLoS Genetics. 12(5). e1006083–e1006083. 10 indexed citations
2.
Yang, Weili, Guohao Wang, Chuan-En Wang, et al.. (2015). Mutant Alpha-Synuclein Causes Age-Dependent Neuropathology in Monkey Brain. Journal of Neuroscience. 35(21). 8345–8358. 55 indexed citations
3.
Wang, Guohao, Huaqiang Yang, Sen Yan, et al.. (2015). Cytoplasmic mislocalization of RNA splicing factors and aberrant neuronal gene splicing in TDP-43 transgenic pig brain. Molecular Neurodegeneration. 10(1). 42–42. 48 indexed citations
4.
Wang, Chuan-En, et al.. (2014). Ubiquitin-Activating Enzyme Activity Contributes to Differential Accumulation of Mutant Huntingtin in Brain and Peripheral Tissues. Journal of Neuroscience Nursing. 34(25). 8411–8422.
5.
Wade, Barbara, Chuan-En Wang, Sen Yan, et al.. (2014). Ubiquitin-Activating Enzyme Activity Contributes to Differential Accumulation of Mutant Huntingtin in Brain and Peripheral Tissues. Journal of Neuroscience. 34(25). 8411–8422. 24 indexed citations
6.
Niu, Yuyu, Xiangyu Guo, Chuan-En Wang, et al.. (2014). Early Parkinson's disease symptoms in  -synuclein transgenic monkeys. Human Molecular Genetics. 24(8). 2308–2317. 79 indexed citations
7.
Yan, Sen, et al.. (2014). Differential ubiquitination and degradation of huntingtin fragments modulated by ubiquitin-protein ligase E3A. Proceedings of the National Academy of Sciences. 111(15). 5706–5711. 102 indexed citations
8.
Yan, Sen, Chuan-En Wang, Wenjie Wei, et al.. (2013). TDP-43 causes differential pathology in neuronal versus glial cells in the mouse brain. Human Molecular Genetics. 23(10). 2678–2693. 31 indexed citations
9.
Xu, Qiaoqiao, Shanshan Huang, Mingke Song, et al.. (2013). Synaptic mutant huntingtin inhibits synapsin-1 phosphorylation and causes neurological symptoms. The Journal of Cell Biology. 202(7). 1123–1138. 30 indexed citations
10.
Havel, Lauren S., et al.. (2011). Preferential accumulation of N-terminal mutant huntingtin in the nuclei of striatal neurons is regulated by phosphorylation. Human Molecular Genetics. 20(7). 1424–1437. 44 indexed citations
11.
Bradford, Jennifer W., Ji‐Yeon Shin, Meredith Roberts, et al.. (2010). Mutant Huntingtin in Glial Cells Exacerbates Neurological Symptoms of Huntington Disease Mice. Journal of Biological Chemistry. 285(14). 10653–10661. 126 indexed citations
12.
13.
Li, Xiang, Chuan-En Wang, Shanshan Huang, et al.. (2010). Inhibiting the ubiquitin–proteasome system leads to preferential accumulation of toxic N-terminal mutant huntingtin fragments. Human Molecular Genetics. 19(12). 2445–2455. 71 indexed citations
14.
Friedman, Meyer J., Chuan-En Wang, Xiao‐Jiang Li, & Shihua Li. (2008). Polyglutamine Expansion Reduces the Association of TATA-binding Protein with DNA and Induces DNA Binding-independent Neurotoxicity. Journal of Biological Chemistry. 283(13). 8283–8290. 57 indexed citations
15.
Orr, Adam L., Shihua Li, Chuan-En Wang, et al.. (2008). N-Terminal Mutant Huntingtin Associates with Mitochondria and Impairs Mitochondrial Trafficking. Journal of Neuroscience. 28(11). 2783–2792. 331 indexed citations
16.
17.
Wang, Chuan-En, Hui Zhou, John R. McGuire, et al.. (2008). Suppression of neuropil aggregates and neurological symptoms by an intracellular antibody implicates the cytoplasmic toxicity of mutant huntingtin. The Journal of Cell Biology. 181(5). 803–816. 95 indexed citations
18.
Wang, Jian‐Jun, et al.. (2008). Impaired ubiquitin–proteasome system activity in the synapses of Huntington's disease mice. The Journal of Cell Biology. 180(6). 1177–1189. 140 indexed citations
19.
Shin, Ji‐Yeon, Zhihui Fang, Zhao-Xue Yu, et al.. (2005). Expression of mutant huntingtin in glial cells contributes to neuronal excitotoxicity. The Journal of Cell Biology. 171(6). 1001–1012. 365 indexed citations
20.
Tissir, Fadel, Chuan-En Wang, & André M. Goffinet. (2004). Expression of the chemokine receptor Cxcr4 mRNA during mouse brain development. Developmental Brain Research. 149(1). 63–71. 97 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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