Gary Sweeney
Impact in
- Endocrine and Autonomic Systems top 10%
- Regulation of Appetite and Obesity
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- Adipose Tissue and Metabolism
Papers in
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- Adipokines, Inflammation, and Metabolic Diseases 6
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- Adipose Tissue and Metabolism 5
- Co-authors
- David G. Muñoz (1 shared paper)Ren‐Ke Li (1 shared paper)S. Madani (1 shared paper)Yu Wang (3 shared papers)Ying Liu (1 shared paper)Mika Eguchi (1 shared paper)Fang Xu (1 shared paper)Anqi Xu (1 shared paper)
- Journals
- Cardiovascular Research (2 papers)Endocrine Research (1 paper)Biochemical Journal (1 paper)Journal of Cellular Physiology (1 paper)Diabetologia (1 paper)
- Partner nations
- CanadaHong KongSouth Korea
In The Last Decade
Gary Sweeney
8 papers receiving 339 citations
Peers
Comparison fields: 5 of 59
- Endocrine and Autonomic Systems 75
- Physiology 145
- Epidemiology 163
- Cardiology and Cardiovascular Medicine 86
- Biochemistry 18
Countries citing papers authored by Gary Sweeney
This map shows the geographic impact of Gary Sweeney's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Gary Sweeney with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Gary Sweeney more than expected).
Fields of papers citing papers by Gary Sweeney
This network shows the impact of papers produced by Gary Sweeney. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Gary Sweeney. The network helps show where Gary Sweeney may publish in the future.
Co-authors
The 25 scholars most cited alongside Gary Sweeney, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2005 | 95 | |
| 2 | 2007 | 94 | |
| 3 | 2011 | 60 | |
| 4 | 1998 | 35 | |
| 5 | Improved functional recovery to I/R injury in hearts from lipocalin-2 deficiency mice: restoration of mitochondrial function and phospholipids remodeling. | 2012 | 29 |
| 6 | 2005 | 17 | |
| 7 | 2021 | 12 | |
| 8 | 2009 | 2 |
About Gary Sweeney
Gary Sweeney is a scholar working on Epidemiology, Physiology, Molecular Biology, Endocrine and Autonomic Systems and Surgery, having authored 8 papers that have together received 344 indexed citations. Recurring topics across this work include Adipokines, Inflammation, and Metabolic Diseases (6 papers), Adipose Tissue and Metabolism (5 papers), Metabolism, Diabetes, and Cancer (2 papers), Regulation of Appetite and Obesity (2 papers), Atherosclerosis and Cardiovascular Diseases (1 paper), Signaling Pathways in Disease (1 paper), Pancreatic function and diabetes (1 paper) and Ion Transport and Channel Regulation (1 paper). The work is most often cited by research in Endocrine and Autonomic Systems (75 citations), Physiology (145 citations), Epidemiology (163 citations), Cardiology and Cardiovascular Medicine (86 citations) and Biochemistry (18 citations). Gary Sweeney has collaborated with scholars based in Canada, Hong Kong and South Korea. Frequent co-authors include David G. Muñoz, Ren‐Ke Li, S. Madani, Yu Wang, Ying Liu, Mika Eguchi, Fang Xu, Anqi Xu, Shelley Pallan and Palanivel Rajan. Their work appears in journals such as Cardiovascular Research, Endocrine Research, Biochemical Journal, Journal of Cellular Physiology and Diabetologia.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.