Gali Maor

495 total citations
13 papers, 394 citations indexed

About

Gali Maor is a scholar working on Physiology, Cell Biology and Neurology. According to data from OpenAlex, Gali Maor has authored 13 papers receiving a total of 394 indexed citations (citations by other indexed papers that have themselves been cited), including 10 papers in Physiology, 9 papers in Cell Biology and 5 papers in Neurology. Recurrent topics in Gali Maor's work include Lysosomal Storage Disorders Research (10 papers), Cellular transport and secretion (8 papers) and Parkinson's Disease Mechanisms and Treatments (5 papers). Gali Maor is often cited by papers focused on Lysosomal Storage Disorders Research (10 papers), Cellular transport and secretion (8 papers) and Parkinson's Disease Mechanisms and Treatments (5 papers). Gali Maor collaborates with scholars based in Israel, United States and Italy. Gali Maor's co-authors include Mia Horowitz, Mirella Filocamo, Hermann Steller, Daniel Segal, Sigal Rencus‐Lazar, Debora Rapaport, Ari Zimran, Metsada Pasmanik‐Chor, Johannes M. F. G. Aerts and Sumit Paul and has published in prestigious journals such as Journal of Neuroscience, International Journal of Molecular Sciences and Human Molecular Genetics.

In The Last Decade

Gali Maor

13 papers receiving 385 citations

Peers

Gali Maor

PHYSI

CB

NEURO

OC

MB

Emerson Maniwang United States

Petra Oliva United States

Lee Pellegrino United States

Jenny Do United States

Anna Meshcheriakova Israel

Михаил Николаев Russia

Samantha Cooper United States

Laura Smith United Kingdom

Lauren Morris United Kingdom

G.-J. Lee-Chen Taiwan

Emerson Maniwang United States

Gali Maor

258 ×0.9

PHYSI

151 ×0.7

CB

122 ×0.9

NEURO

54 ×0.8

OC

71 ×1.1

MB

Citations per year, relative to Gali Maor Gali Maor (= 1×) peers Emerson Maniwang

Countries citing papers authored by Gali Maor

Since Specialization

Citations

This map shows the geographic impact of Gali Maor's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Gali Maor with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Gali Maor more than expected).

Fields of papers citing papers by Gali Maor

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Gali Maor. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Gali Maor. The network helps show where Gali Maor may publish in the future.

Co-authorship network of co-authors of Gali Maor

This figure shows the co-authorship network connecting the top 25 collaborators of Gali Maor. A scholar is included among the top collaborators of Gali Maor based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Gali Maor. Gali Maor is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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13 of 13 papers shown

1.

Lai, Yunjia, et al.. (2025). Biotin mitigates the development of manganese-induced, Parkinson’s disease–related neurotoxicity in Drosophila and human neurons. Science Signaling. 18(870). eadn9868–eadn9868. 5 indexed citations

2.

Maor, Gali, Ronald R. Dubreuil, & Mel Β. Feany. (2023). α-Synuclein Promotes Neuronal Dysfunction and Death by Disrupting the Binding of Ankyrin to β-Spectrin. Journal of Neuroscience. 43(9). 1614–1626. 7 indexed citations

3.

Paul, Sumit, Gali Maor, Metsada Pasmanik‐Chor, et al.. (2021). The Uncovered Function of the Drosophila GBA1a-Encoded Protein. Cells. 10(3). 630–630. 5 indexed citations

4.

Maor, Gali, et al.. (2020). Misfolding of Lysosomal α-Galactosidase a in a Fly Model and Its Alleviation by the Pharmacological Chaperone Migalastat. International Journal of Molecular Sciences. 21(19). 7397–7397. 12 indexed citations

5.

Maor, Gali, Debora Rapaport, & Mia Horowitz. (2019). The effect of mutant GBA1 on accumulation and aggregation of α-synuclein. Human Molecular Genetics. 28(11). 1768–1781. 36 indexed citations

6.

Paul, Sumit, Gali Maor, Metsada Pasmanik‐Chor, et al.. (2019). Drosophila melanogaster Mutated in its GBA1b Ortholog Recapitulates Neuronopathic Gaucher Disease. Journal of Clinical Medicine. 8(9). 1420–1420. 28 indexed citations

7.

Maor, Gali, et al.. (2016). The contribution of mutantGBAto the development of Parkinson disease inDrosophila. Human Molecular Genetics. 25(13). ddw129–ddw129. 64 indexed citations

8.

Horowitz, Mia, et al.. (2016). Presence of mutant GBA allele leads to ER stress and development of Parkinson disease. Molecular Genetics and Metabolism. 117(2). S58–S58. 1 indexed citations

9.

Maor, Gali, et al.. (2016). UPR activation and CHOP mediated induction of GBA1 transcription in Gaucher disease. Blood Cells Molecules and Diseases. 68. 21–29. 26 indexed citations

10.

Horowitz, Mia & Gali Maor. (2015). Presence of mutant GBA allele leads to ER stress and development of Parkinson's disease. Molecular Genetics and Metabolism. 114(2). S55–S56. 1 indexed citations

11.

Horowitz, Mia, et al.. (2014). The connection between ERAD, UPR, Gaucher disease and Parkinson disease. Molecular Genetics and Metabolism. 111(2). S55–S55. 1 indexed citations

12.

Maor, Gali, Sigal Rencus‐Lazar, Mirella Filocamo, et al.. (2013). Unfolded protein response in Gaucher disease: from human to Drosophila. Orphanet Journal of Rare Diseases. 8(1). 140–140. 92 indexed citations

13.

Maor, Gali, et al.. (2012). Ambroxol as a pharmacological chaperone for mutant glucocerebrosidase. Blood Cells Molecules and Diseases. 50(2). 141–145. 116 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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