G. William Rebeck

3.3k total citations · 1 hit paper
42 papers, 2.7k citations indexed

About

G. William Rebeck is a scholar working on Physiology, Cellular and Molecular Neuroscience and Molecular Biology. According to data from OpenAlex, G. William Rebeck has authored 42 papers receiving a total of 2.7k indexed citations (citations by other indexed papers that have themselves been cited), including 33 papers in Physiology, 17 papers in Cellular and Molecular Neuroscience and 14 papers in Molecular Biology. Recurrent topics in G. William Rebeck's work include Alzheimer's disease research and treatments (32 papers), Neuroscience and Neuropharmacology Research (9 papers) and Neuroinflammation and Neurodegeneration Mechanisms (7 papers). G. William Rebeck is often cited by papers focused on Alzheimer's disease research and treatments (32 papers), Neuroscience and Neuropharmacology Research (9 papers) and Neuroinflammation and Neurodegeneration Mechanisms (7 papers). G. William Rebeck collaborates with scholars based in United States, China and South Korea. G. William Rebeck's co-authors include Bradley T. Hyman, Dudley K. Strickland, Hyang‐Sook Hoe, Preeti J. Khandelwal, Alexander M. Herman, Charbel Moussa, Steven M. Greenberg, Guojun Bu, Zhihua Qiu and Daniel T.S. Pak and has published in prestigious journals such as Journal of Biological Chemistry, Neuron and Journal of Neuroscience.

In The Last Decade

G. William Rebeck

42 papers receiving 2.6k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Apolipoprotein E in sporadic Alzheimer's disease: Allelic variation and receptor interactions

1993 914 citations G. William Rebeck, Dudley K. Strickland et al. Neuron profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
G. William Rebeck United States	25	1.9k	1.1k	663	431	394	42	2.7k
Liana Fidani Greece	18	1.3k 0.7×	927 0.9×	449 0.7×	258 0.6×	458 1.2×	74	2.2k
Ina Tesseur Belgium	25	2.3k 1.2×	1.6k 1.5×	974 1.5×	762 1.8×	316 0.8×	41	3.7k
Tsuneo Yamazaki Japan	23	1.7k 0.9×	1.0k 1.0×	419 0.6×	387 0.9×	603 1.5×	53	2.5k
Karelle Leroy Belgium	29	2.1k 1.1×	1.4k 1.3×	963 1.5×	639 1.5×	307 0.8×	60	3.2k
Estibaliz Capetillo‐Zarate United States	26	1.4k 0.8×	791 0.7×	797 1.2×	784 1.8×	263 0.7×	44	2.6k
Wen-Lang Lin United States	8	2.6k 1.4×	1.2k 1.1×	920 1.4×	696 1.6×	441 1.1×	11	3.1k
Bart Dermaut Belgium	31	1.3k 0.7×	1.9k 1.8×	540 0.8×	416 1.0×	872 2.2×	64	3.3k
Katie Hamm United States	7	1.3k 0.7×	555 0.5×	319 0.5×	565 1.3×	223 0.6×	7	1.9k
Lena Lilius Sweden	19	1.7k 0.9×	1.1k 1.0×	389 0.6×	240 0.6×	229 0.6×	42	2.2k
Janet van Eersel Australia	17	1.8k 1.0×	918 0.9×	980 1.5×	534 1.2×	364 0.9×	24	2.6k

Countries citing papers authored by G. William Rebeck

Since Specialization

Citations

This map shows the geographic impact of G. William Rebeck's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by G. William Rebeck with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites G. William Rebeck more than expected).

Fields of papers citing papers by G. William Rebeck

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by G. William Rebeck. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by G. William Rebeck. The network helps show where G. William Rebeck may publish in the future.

Co-authorship network of co-authors of G. William Rebeck

This figure shows the co-authorship network connecting the top 25 collaborators of G. William Rebeck. A scholar is included among the top collaborators of G. William Rebeck based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with G. William Rebeck. G. William Rebeck is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Gross, Phillip S., et al.. (2023). The chemotherapeutic agent doxorubicin induces brain senescence, with modulation by APOE genotype. Experimental Neurology. 371. 114609–114609. 14 indexed citations

Zhao, Wenjuan, et al.. (2014). Aging reduces glial uptake and promotes extracellular accumulation of AÎ² from a lentiviral vector. Frontiers in Aging Neuroscience. 6. 210–210. 23 indexed citations

Tamboli, Irfan Y., et al.. (2014). Extracellular Proteolysis of Apolipoprotein E (apoE) by Secreted Serine Neuronal Protease. PLoS ONE. 9(3). e93120–e93120. 30 indexed citations

Zhao, Wenchang, Sonya B. Dumanis, Irfan Y. Tamboli, et al.. (2013). Human APOE genotype affects intraneuronal A 1-42 accumulation in a lentiviral gene transfer model. Human Molecular Genetics. 23(5). 1365–1375. 27 indexed citations

Babus, Lenard W., Sakura Minami, Jung Min Song, et al.. (2011). Decreased dendritic spine density and abnormal spine morphology in Fyn knockout mice. Brain Research. 1415. 96–102. 30 indexed citations

Khandelwal, Preeti J., et al.. (2011). Parkin mediates beclin-dependent autophagic clearance of defective mitochondria and ubiquitinated A in AD models. Human Molecular Genetics. 20(11). 2091–2102. 150 indexed citations

Minami, Sakura, Elkhansa Sidahmed, Saba Aïd, et al.. (2010). Therapeutic versus neuroinflammatory effects of passive immunization is dependent on Aβ/amyloid burden in a transgenic mouse model of Alzheimer's disease. Journal of Neuroinflammation. 7(1). 57–57. 19 indexed citations

Khandelwal, Preeti J., Sonya B. Dumanis, Li Feng, et al.. (2010). Parkinson-related parkin reduces α-Synuclein phosphorylation in a gene transfer model. Molecular Neurodegeneration. 5(1). 47–47. 65 indexed citations

Rebeck, G. William, Hyang‐Sook Hoe, & Charbel Moussa. (2010). β-Amyloid1–42 Gene Transfer Model Exhibits Intraneuronal Amyloid, Gliosis, Tau Phosphorylation, and Neuronal Loss. Journal of Biological Chemistry. 285(10). 7440–7446. 48 indexed citations

10.

Burns, Mark P. & G. William Rebeck. (2010). Intracellular cholesterol homeostasis and amyloid precursor protein processing. Biochimica et Biophysica Acta (BBA) - Molecular and Cell Biology of Lipids. 1801(8). 853–859. 29 indexed citations

11.

Hoe, Hyang‐Sook, Kyung‐Jin Lee, Rosalind S.E. Carney, et al.. (2009). Interaction of Reelin with Amyloid Precursor Protein Promotes Neurite Outgrowth. Journal of Neuroscience. 29(23). 7459–7473. 174 indexed citations

12.

Hirata‐Fukae, Chiho, Huifang Li, Li Ma, et al.. (2008). Levels of soluble and insoluble tau reflect overall status of tau phosphorylation in vivo. Neuroscience Letters. 450(1). 51–55. 11 indexed citations

13.

Robbins, Elissa M., Rebecca A. Betensky, Sarah Domnitz, et al.. (2006). Kinetics of Cerebral Amyloid Angiopathy Progression in a Transgenic Mouse Model of Alzheimer Disease. Journal of Neuroscience. 26(2). 365–371. 65 indexed citations

14.

Rebeck, G. William, Mary Jo LaDu, Steven Estus, Guojun Bu, & Edwin J. Weeber. (2006). The generation and function of soluble apoE receptors in the CNS. Molecular Neurodegeneration. 1(1). 15–15. 52 indexed citations

15.

Hoe, Hyang‐Sook, et al.. (2005). Association of apolipoprotein J-positive β-amyloid plaques with dystrophic neurites in alzheimer’s disease brain. Neurotoxicity Research. 7(3). 231–241. 39 indexed citations

16.

Shin, Youngah, et al.. (2002). Vascular Changes in Iowa‐Type Hereditary Cerebral Amyloid Angiopathy. Annals of the New York Academy of Sciences. 977(1). 245–251. 19 indexed citations

17.

Hyman, Bradley T., et al.. (2001). Quantitation of apoE Domains in Alzheimer Disease Brain Suggests a Role for apoE in Aß Aggregation. Journal of Neuropathology & Experimental Neurology. 60(4). 342–349. 85 indexed citations

18.

Irizarry, Michael C., Bonnie Cheung, G. William Rebeck, et al.. (2000). Apolipoprotein E affects the amount, form, and anatomical distribution of amyloid β-peptide deposition in homozygous APP V717F transgenic mice. Acta Neuropathologica. 100(5). 451–458. 77 indexed citations

19.

Simonian, Nancy A., G. William Rebeck, & Bradley T. Hyman. (1994). Chapter 30 Functional integrity of neural systems related to memory in Alzheimer's disease. Progress in brain research. 100. 245–254. 5 indexed citations

20.

Rebeck, G. William, et al.. (1993). Apolipoprotein E in sporadic Alzheimer's disease: Allelic variation and receptor interactions. Neuron. 11(4). 575–580. 914 indexed citations breakdown →

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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