G. Travis Tabor

652 total citations
11 papers, 373 citations indexed

About

G. Travis Tabor is a scholar working on Molecular Biology, Neurology and Physiology. According to data from OpenAlex, G. Travis Tabor has authored 11 papers receiving a total of 373 indexed citations (citations by other indexed papers that have themselves been cited), including 6 papers in Molecular Biology, 5 papers in Neurology and 3 papers in Physiology. Recurrent topics in G. Travis Tabor's work include Neuroinflammation and Neurodegeneration Mechanisms (5 papers), Alzheimer's disease research and treatments (3 papers) and Immune cells in cancer (3 papers). G. Travis Tabor is often cited by papers focused on Neuroinflammation and Neurodegeneration Mechanisms (5 papers), Alzheimer's disease research and treatments (3 papers) and Immune cells in cancer (3 papers). G. Travis Tabor collaborates with scholars based in United States, Hungary and Pakistan. G. Travis Tabor's co-authors include David M. Holtzman, Jason D. Ulrich, Melissa Manis, Rudolph E. Tanzi, Tien‐Phat V. Huynh, Caroline Massad Francis, Carla M. Yuede, Yun Chen, Thomas E. Mahan and Mary Beth Finn and has published in prestigious journals such as Cell, Nature Communications and Neuron.

In The Last Decade

G. Travis Tabor

11 papers receiving 369 citations

Peers

Countries citing papers authored by G. Travis Tabor

Since Specialization

Citations

This map shows the geographic impact of G. Travis Tabor's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by G. Travis Tabor with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites G. Travis Tabor more than expected).

Fields of papers citing papers by G. Travis Tabor

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by G. Travis Tabor. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by G. Travis Tabor. The network helps show where G. Travis Tabor may publish in the future.

Co-authorship network of co-authors of G. Travis Tabor

This figure shows the co-authorship network connecting the top 25 collaborators of G. Travis Tabor. A scholar is included among the top collaborators of G. Travis Tabor based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with G. Travis Tabor. G. Travis Tabor is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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11 of 11 papers shown

#	Work	Indexed citations
1	TREM2 inhibition triggers antitumor cell activity of myeloid cells in glioblastoma 2023 ·Science Advances ·Rui Sun, Rowland H. Han, (unknown), Saad M. Khan, G. Travis Tabor, Yun Chen, Jinchao Hou, Haowu Jiang, Kathleen M. Schoch, Diane D. Mao, Ryan T. Cleary, Alicia Yang, Qin Liu, Jingqin Luo, Allegra A. Petti, Timothy M. Miller, Jason D. Ulrich, David M. Holtzman, Albert H. Kim	62
2	IMMU-28. TREM2 INHIBITION REPROGRAMS MYELOID CELLS TO STIMULATE ANTI-TUMOR IMMUNITY THROUGH BOTH DIRECT AND INDIRECT MECHANISMS 2023 ·Neuro-Oncology ·Rui Sun, Rowland H. Han, (unknown), G. Travis Tabor, Jinchao Hou, Ryan T. Cleary, Diane D. Mao, Qin Liu, Kathleen M. Schoch, Jingqin Luo, Allegra A. Petti, Timothy M. Miller, Jason D. Ulrich, David M. Holtzman, Albert Kim	1
3	Microglial REV-ERBα regulates inflammation and lipid droplet formation to drive tauopathy in male mice 2023 ·Nature Communications ·(unknown), Julie Dimitry, (unknown), (unknown), Patrick W. Sheehan, Melvin W. King, G. Travis Tabor, Young Ah Goo, Mitchell A. Lazar, Leonard Petrucelli, Erik S. Musiek	46
4	Amelioration of Tau and ApoE4-linked glial lipid accumulation and neurodegeneration with an LXR agonist 2023 ·Neuron ·Alexandra Litvinchuk, Jung H. Suh, Jing Guo, Karin Lin, Sonnet S. Davis, Nga Bien‐Ly, Eric Tycksen, G. Travis Tabor, Javier Remolina Serrano, Melissa Manis, Xin Bao, Choonghee Lee, Megan E. Bosch, Enmanuel J. Perez, Carla M. Yuede, Anil G. Cashikar, Jason D. Ulrich, Gilbert Di Paolo, David M. Holtzman	61
5	APOE3ch alters microglial response and suppresses Aβ-induced tau seeding and spread 2023 ·Cell ·Yun Chen, (unknown), Samira Parhizkar, (unknown), Yiyang Zhu, Michael R. Strickland, Chanung Wang, (unknown), G. Travis Tabor, Hong Jiang, (unknown), Albert A. Davis, Carla M. Yuede, Marco Colonna, Jason D. Ulrich, David M. Holtzman	54
6	Current status of amyloid-targeting immunotherapies for Alzheimer’s disease 2023 ·Science Translational Medicine ·G. Travis Tabor, David M. Holtzman	10
7	Activity-dependent isomerization of Kv4.2 by Pin1 regulates cognitive flexibility 2020 ·Nature Communications ·(unknown), (unknown), G. Travis Tabor, (unknown), Ying Liu, Daniel Abebe, Rose‐Marie Karlsson, Stewart R. Durell, Heather A. Cameron, Dax A. Hoffman	30
8	Lack of hepatic apoE does not influence early Aβ deposition: observations from a new APOE knock-in model 2019 ·Molecular Neurodegeneration ·Tien‐Phat V. Huynh, Chao Wang, (unknown), G. Travis Tabor, Thomas E. Mahan, Caroline Massad Francis, Mary Beth Finn, (unknown), Melissa Manis, Rudolph E. Tanzi, Jason D. Ulrich, David M. Holtzman	87
9	A novel bungarotoxin binding site-tagged construct reveals MAPK-dependent Kv4.2 trafficking 2019 ·Molecular and Cellular Neuroscience ·G. Travis Tabor, Jung M. Park, Jonathan G. Murphy, Jiahua Hu, Dax A. Hoffman	4
10	Disruption of GpI mGluR-Dependent Cav2.3 Translation in a Mouse Model of Fragile X Syndrome 2019 ·Journal of Neuroscience ·Erin E. Gray, Jonathan G. Murphy, Ying Liu, (unknown), G. Travis Tabor, Lin Lin, Dax A. Hoffman	16
11	Deciphering fact from artifact when using reporter assays to investigate the roles of host factors on L1 retrotransposition 2016 ·Mobile DNA ·(unknown), G. Travis Tabor	2

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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