G. Preibisch

469 total citations
8 papers, 354 citations indexed

About

G. Preibisch is a scholar working on Molecular Biology, Endocrine and Autonomic Systems and Physiology. According to data from OpenAlex, G. Preibisch has authored 8 papers receiving a total of 354 indexed citations (citations by other indexed papers that have themselves been cited), including 3 papers in Molecular Biology, 3 papers in Endocrine and Autonomic Systems and 3 papers in Physiology. Recurrent topics in G. Preibisch's work include Regulation of Appetite and Obesity (3 papers), Adipose Tissue and Metabolism (3 papers) and Protein Kinase Regulation and GTPase Signaling (1 paper). G. Preibisch is often cited by papers focused on Regulation of Appetite and Obesity (3 papers), Adipose Tissue and Metabolism (3 papers) and Protein Kinase Regulation and GTPase Signaling (1 paper). G. Preibisch collaborates with scholars based in Germany and United States. G. Preibisch's co-authors include Hermann Ragg, H J Döring, Oliver Stehling, Inger Kappel Schmidt, Ulrich Schweiger, C. Wewetzer, Martin Klingenspor, Werner Blum, Cornelia Exner and Gerhard Heldmaier and has published in prestigious journals such as Journal of Biological Chemistry, Endocrinology and Diabetologia.

In The Last Decade

G. Preibisch

8 papers receiving 347 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
G. Preibisch Germany 7 205 112 112 100 43 8 354
Kohji Kiwaki Japan 12 134 0.7× 17 0.2× 89 0.8× 46 0.5× 193 4.5× 15 525
A. M. Sharma United States 6 199 1.0× 44 0.4× 156 1.4× 123 1.2× 113 2.6× 7 420
Yang-Ho Choi United States 6 193 0.9× 15 0.1× 186 1.7× 71 0.7× 71 1.7× 8 465
Eileen F. VanderLaan United States 9 83 0.4× 43 0.4× 86 0.8× 18 0.2× 73 1.7× 10 417
Hanspeter Goldschmidt Germany 11 298 1.5× 67 0.6× 191 1.7× 230 2.3× 102 2.4× 13 608
Agnieszka Olszewska Germany 7 146 0.7× 23 0.2× 115 1.0× 36 0.4× 154 3.6× 17 566
Cecilia E. Kim United States 11 101 0.5× 24 0.2× 125 1.1× 66 0.7× 178 4.1× 14 678
Gabriella Segal‐Lieberman Israel 12 310 1.5× 20 0.2× 181 1.6× 142 1.4× 42 1.0× 18 536
Jill S. Carmody United States 9 271 1.3× 8 0.1× 197 1.8× 94 0.9× 93 2.2× 9 501
Neil Inhaber United States 10 92 0.4× 8 0.1× 86 0.8× 17 0.2× 18 0.4× 14 386

Countries citing papers authored by G. Preibisch

Since Specialization
Citations

This map shows the geographic impact of G. Preibisch's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by G. Preibisch with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites G. Preibisch more than expected).

Fields of papers citing papers by G. Preibisch

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by G. Preibisch. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by G. Preibisch. The network helps show where G. Preibisch may publish in the future.

Co-authorship network of co-authors of G. Preibisch

This figure shows the co-authorship network connecting the top 25 collaborators of G. Preibisch. A scholar is included among the top collaborators of G. Preibisch based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with G. Preibisch. G. Preibisch is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

8 of 8 papers shown
1.
Exner, Cornelia, Johannes Hebebrand, Helmut Remschmidt, et al.. (2000). Leptin suppresses semi-starvation induced hyperactivity in rats: implications for anorexia nervosa. Molecular Psychiatry. 5(5). 476–481. 167 indexed citations
2.
Hamann, A., et al.. (1997). Chronic leptin treatment does not prevent the development of obesity in transgenic mice with brown fat deficiency. Diabetologia. 40(7). 810–815. 17 indexed citations
3.
Stehling, Oliver, et al.. (1996). Leptin reduces juvenile fat stores by altering the circadian cycle of energy expenditure. American Journal of Physiology-Regulatory, Integrative and Comparative Physiology. 271(6). R1770–R1774. 78 indexed citations
4.
Preibisch, G., et al.. (1995). Homocysteine-Induced Oxidative Damage: Mechanisms and Possible Roles in Neurodegenerative and Atherogenic Processes. Zeitschrift für Naturforschung C. 50(9-10). 699–707. 25 indexed citations
5.
Preibisch, G., et al.. (1993). Biochemical Model Reactions on the Prooxidative Activity of Homocysteine. Zeitschrift für Naturforschung C. 48(1-2). 58–62. 14 indexed citations
6.
Wildey, Gary, Alan J. Fischman, John T. Fallon, et al.. (1988). Cellular Processing of Pro-Atrial Natriuretic Factor (Pro-ANF): Studies Using an Antiserum that Selectively Binds ANF-(99–126) after Its Cleavage from Pro-ANF*. Endocrinology. 123(4). 2054–2061. 6 indexed citations
7.
Ragg, Hermann & G. Preibisch. (1988). Structure and expression of the gene coding for the human serpin hLS2.. Journal of Biological Chemistry. 263(24). 12129–12134. 31 indexed citations
8.
Bloch, Kenneth D., Steven W. Jones, G. Preibisch, et al.. (1987). Proatrial natriuretic factor is phosphorylated by rat cardiocytes in culture.. Journal of Biological Chemistry. 262(21). 9956–9961. 16 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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