G J Hämmerling

2.1k total citations
37 papers, 1.8k citations indexed

About

G J Hämmerling is a scholar working on Immunology, Molecular Biology and Radiology, Nuclear Medicine and Imaging. According to data from OpenAlex, G J Hämmerling has authored 37 papers receiving a total of 1.8k indexed citations (citations by other indexed papers that have themselves been cited), including 30 papers in Immunology, 13 papers in Molecular Biology and 12 papers in Radiology, Nuclear Medicine and Imaging. Recurrent topics in G J Hämmerling's work include T-cell and B-cell Immunology (19 papers), Immune Cell Function and Interaction (13 papers) and Monoclonal and Polyclonal Antibodies Research (12 papers). G J Hämmerling is often cited by papers focused on T-cell and B-cell Immunology (19 papers), Immune Cell Function and Interaction (13 papers) and Monoclonal and Polyclonal Antibodies Research (12 papers). G J Hämmerling collaborates with scholars based in Germany, France and United States. G J Hämmerling's co-authors include Frank Momburg, Marga Nijenhuis, Jacques Neefjes, Harald Kropshofer, Anne B. Vogt, Juergen Hammer, Jennifer Blum, G Moldenhauer, Robert Busch and Elisa Rusch and has published in prestigious journals such as Cell, Proceedings of the National Academy of Sciences and The Journal of Experimental Medicine.

In The Last Decade

G J Hämmerling

37 papers receiving 1.8k citations

Peers

Countries citing papers authored by G J Hämmerling

Since Specialization

Citations

This map shows the geographic impact of G J Hämmerling's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by G J Hämmerling with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites G J Hämmerling more than expected).

Fields of papers citing papers by G J Hämmerling

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by G J Hämmerling. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by G J Hämmerling. The network helps show where G J Hämmerling may publish in the future.

Co-authorship network of co-authors of G J Hämmerling

This figure shows the co-authorship network connecting the top 25 collaborators of G J Hämmerling. A scholar is included among the top collaborators of G J Hämmerling based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with G J Hämmerling. G J Hämmerling is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

#	Work	Indexed citations
1	Tetraspanin CD53 Promotes Lymphocyte Recirculation by Stabilizing L-Selectin Surface Expression 2020 ·iScience ·(unknown), (unknown), (unknown), Janet L. Wee, (unknown), (unknown), Zeyad Nasa, Frank Alderuccio, Pamela Hall, (unknown), Katrina J. Binger, G J Hämmerling, Hang Fai Kwok, Andrew J. Newman, Ann Ager, Annemiek B. van Spriel, Michael J. Hickey, Mark D. Wright	26
2	Dendritic Cell Migration and Antigen Presentation Are Coordinated by the Opposing Functions of the Tetraspanins CD82 and CD37 2016 ·The Journal of Immunology ·(unknown), Janet L. Wee, (unknown), (unknown), (unknown), Javier Vega‐Ramos, Jóse A. Villadangos, Annemiek B. van Spriel, Michael J. Hickey, G J Hämmerling, Mark D. Wright	38
3	Control of peripheral T-lymphocyte tolerance in neonates and adults 2005 ·Trends in Immunology ·Bernd Arnold, Thomas Schüler, G J Hämmerling	19
4	Stimulation of autoimmunity by toll-like receptor ligands 2005 ·Annals of the Rheumatic Diseases ·Andreas Limmer, (unknown), Natalio Garbi, Bernd Arnold, G J Hämmerling	2
5	Peptide binding α1α2 domain of HLA-B27 contributes to the disease pathogenesis in transgenic mice 1999 ·Human Immunology ·Sanjay D. Khare, (unknown), (unknown), Julie A. Hanson, Harvinder S. Luthra, G J Hämmerling, Chella S. David	6
6	Trimming of TAP-translocated peptides in the endoplasmic reticulum and in the cytosol during recycling. 1994 ·The Journal of Experimental Medicine ·(unknown), Monique Grommé, Frank Momburg, G J Hämmerling, Jacques Neefjes	125
7	Resistance to natural killer cell lysis conferred by TAP1/2 genes in human antigen-processing mutant cells. 1994 ·The Journal of Immunology ·Margarita Salcedo, Frank Momburg, G J Hämmerling, Hans‐Gustaf Ljunggren	37
8	Epitope-specific enhancement of antigen presentation by invariant chain. 1993 ·The Journal of Experimental Medicine ·Frank Momburg, Serge Y. Fuchs, Jan Felix Drexler, Robert Busch, Martin Post, G J Hämmerling, Luciano Adorini	44
9	Intracellular distribution of the MHC class II molecules and the associated invariant chain (li) in different cell lines 1993 ·International Immunology ·Anne Simonsen, Frank Momburg, (unknown), G J Hämmerling, Oddmund Bakke	52
10	Processing of endogenously synthesized hen egg-white lysozyme retained in the endoplasmic reticulum or in secretory form gives rise to a similar but not identical set of epitopes recognized by class II-restricted T cells. 1993 ·The Journal of Immunology ·Luciano Adorini, Jean‐Charles Guéry, Sebastian W. Fuchs, Vianney Ortiz‐Navarrete, G J Hämmerling, Frank Momburg	40
11	Folding and assembly of major histocompatibility complex class I heterodimers in the endoplasmic reticulum of intact cells precedes the binding of peptide. 1993 ·The Journal of Experimental Medicine ·Jacques Neefjes, G J Hämmerling, Frank Momburg	66
12	Gene transfer experiments imply instructive role of major histocompatibility complex class I molecules in cellular peptide processing 1992 ·European Journal of Immunology ·Hans‐Joachim Wallny, Olaf Rötzschke, Kirsten Falk, G J Hämmerling, Hans‐Georg Rammensee	19
13	Restoration of a tumorigenic phenotype by beta 2-microglobulin transfection to EL-4 mutant cells. 1992 ·The Journal of Experimental Medicine ·Rickard Glas, (unknown), G J Hämmerling, Klas Kärre, Hans‐Gustaf Ljunggren	58
14	Species-specific binding of CD4 to the beta 2 domain of major histocompatibility complex class II molecules. 1992 ·The Journal of Experimental Medicine ·Dario A.A. Vignali, José Moreno, Dorothea L. Schiller, G J Hämmerling	57
15	Uneven tissue distribution of minor histocompatibility proteins versus peptides is caused by MHC expression 1991 ·Cell ·Peter Griem, Hans‐Joachim Wallny, Kirsten Falk, Olaf Rötzschke, Bernd Arnold, Günther Schönrich, G J Hämmerling, (unknown)	57
16	Exogenous peptides compete for the presentation of endogenous antigens to major histocompatibility complex class II-restricted T cells. 1991 ·The Journal of Experimental Medicine ·Luciano Adorini, José Moreno, Frank Momburg, G J Hämmerling, Jean‐Charles Guéry, A Valli, Serge Y. Fuchs	50
17	Transfection of beta 2-microglobulin restores IFN-mediated protection from natural killer cell lysis in YAC-1 lymphoma variants. 1990 ·The Journal of Immunology ·Hans‐Gustaf Ljunggren, (unknown), Elisabeth Z. Wolpert, G J Hämmerling, K Kärre	48
18	NK SENSITIVITY, H‐2 EXPRESSION AND METASTATIC POTENTIAL: ANALYSIS OF H‐2D^k GENE TRANSFECTED FIBROSARCOMA CELLS 1989 ·International Journal of Immunogenetics ·Jacob Gopas, Bracha Rager‐Zisman, Iris Har‐Vardi, G J Hämmerling, Menashe Bar‐Eli, S. Segal	8
19	LOSS OF HLA‐A,B,C IN COLORECTAL CARCINOMA IS RELATED TO THE DEGREE OF DE‐DIFFERENTIATION 1986 ·International Journal of Immunogenetics ·Frank Momburg, Peter Möller, Gerhard Moldenhauer, G J Hämmerling	26
20	Recognition of polymorphic H-2 domains by T lymphocytes. I. Functional role of different H-2 domains for the generation of alloreactive cytotoxic T lymphocytes and determination of precursor frequencies. 1981 ·The Journal of Experimental Medicine ·Cornelia M. Weyand, Jörg J. Goronzy, G J Hämmerling	35

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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