G. G. Pegg
- Endocrine and Autonomic Systems top 1%
- Physiology top 5%
- Epidemiology top 10%
- Nutrition and Dietetics top 5%
- Molecular Biology
- Co-authors
- Carlo GazzolaRA HillMartin N. SillenceD. B. LindsayPo‐Chin HuangM. N. SillenceAndrew HoeyRodney A. Hill
- Topics
- Pharmacological Effects and Assays (8 papers)Adipose Tissue and Metabolism (6 papers)Receptor Mechanisms and Signaling (3 papers)
- Journals
- International Journal of ObesityLife SciencesAmerican Journal of Physiology-Endocrinology and Metabolism
- Partner nations
- AustraliaUnited States
In The Last Decade
G. G. Pegg
13 papers receiving 975 citations
Hit Papers
Peers
Comparison fields: 5 of 94
- Endocrine and Autonomic Systems 529
- Physiology 478
- Epidemiology 418
- Nutrition and Dietetics 209
- Molecular Biology 146
Countries citing papers authored by G. G. Pegg
This map shows the geographic impact of G. G. Pegg's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by G. G. Pegg with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites G. G. Pegg more than expected).
Fields of papers citing papers by G. G. Pegg
This network shows the impact of papers produced by G. G. Pegg. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by G. G. Pegg. The network helps show where G. G. Pegg may publish in the future.
Co-authorship network of co-authors of G. G. Pegg
This figure shows the co-authorship network connecting the top 25 collaborators of G. G. Pegg. A scholar is included among the top collaborators of G. G. Pegg based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with G. G. Pegg. G. G. Pegg is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | Leptin: a review of its peripheral actions and interactionsbreakdown → | 758 |
| 2 | 9 | |
| 3 | 33 | |
| 4 | 11 | |
| 5 | Passive Immunisation with Anti-IGF-1 Antibodies Increases Feed Intake during Nutritional Restriction | 1 |
| 6 | 4 | |
| 7 | 9 | |
| 8 | 76 | |
| 9 | 16 | |
| 10 | 42 | |
| 11 | 11 | |
| 12 | 44 | |
| 13 | 10 |
About G. G. Pegg
G. G. Pegg is a scholar working on Animal Science and Zoology, Endocrine and Autonomic Systems and Behavioral Neuroscience, having authored 13 papers that have together received 1.0k indexed citations. Recurring topics across this work include Pharmacological Effects and Assays (8 papers), Adipose Tissue and Metabolism (6 papers) and Receptor Mechanisms and Signaling (3 papers). The work is most often cited by research in Endocrine and Autonomic Systems (529 citations), Physiology (478 citations) and Nutrition and Dietetics (209 citations). G. G. Pegg has collaborated with scholars based in Australia and United States. Frequent co-authors include Carlo Gazzola, RA Hill, Martin N. Sillence, D. B. Lindsay, Po‐Chin Huang, M. N. Sillence, Andrew Hoey, Rodney A. Hill, Thomas F. Magner and Nicholas R. Staten. Their work appears in journals such as International Journal of Obesity, Life Sciences and American Journal of Physiology-Endocrinology and Metabolism.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.