Fred L. Robinson

4.9k total citations · 2 hit papers
4 papers, 4.2k citations indexed

About

Fred L. Robinson is a scholar working on Molecular Biology, Cell Biology and Cellular and Molecular Neuroscience. According to data from OpenAlex, Fred L. Robinson has authored 4 papers receiving a total of 4.2k indexed citations (citations by other indexed papers that have themselves been cited), including 3 papers in Molecular Biology, 2 papers in Cell Biology and 1 paper in Cellular and Molecular Neuroscience. Recurrent topics in Fred L. Robinson's work include Melanoma and MAPK Pathways (3 papers), Protein Kinase Regulation and GTPase Signaling (2 papers) and Autophagy in Disease and Therapy (1 paper). Fred L. Robinson is often cited by papers focused on Melanoma and MAPK Pathways (3 papers), Protein Kinase Regulation and GTPase Signaling (2 papers) and Autophagy in Disease and Therapy (1 paper). Fred L. Robinson collaborates with scholars based in United States. Fred L. Robinson's co-authors include Melanie H. Cobb, Tara Beers Gibson, Gray W. Pearson, Bing-e Xu, Mahesh Karandikar, Kevin Berman, Loraine Silvestro, Zhu Chen, Colleen A. Vanderbilt and Angélique Wright and has published in prestigious journals such as Chemical Reviews, Journal of Biological Chemistry and Endocrine Reviews.

In The Last Decade

Fred L. Robinson

4 papers receiving 4.2k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Mitogen-Activated Protein (MAP) Kinase Pathways: Regulation and Physiological Functions*

2001 3.4k citations Gray W. Pearson, Fred L. Robinson et al. Endocrine Reviews profile →
MAP Kinases

2001 704 citations Zhu Chen, Tara Beers Gibson et al. Chemical Reviews profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Fred L. Robinson United States	4	2.8k	509	508	447	420	4	4.2k
Bing-e Xu United States	10	3.1k 1.1×	464 0.9×	476 0.9×	463 1.0×	395 0.9×	10	4.4k
Kevin Berman United States	10	2.5k 0.9×	465 0.9×	485 1.0×	407 0.9×	412 1.0×	12	3.9k
Mahesh Karandikar United States	13	2.6k 0.9×	586 1.2×	514 1.0×	409 0.9×	424 1.0×	18	4.2k
Tara Beers Gibson United States	11	2.9k 1.1×	525 1.0×	642 1.3×	479 1.1×	473 1.1×	20	4.6k
William F. Matter United States	14	3.1k 1.1×	749 1.5×	689 1.4×	542 1.2×	432 1.0×	19	4.5k
Tsung-I Peng Taiwan	7	3.5k 1.3×	585 1.1×	556 1.1×	258 0.6×	429 1.0×	7	4.7k
Razvan Lapadat United States	9	2.3k 0.8×	685 1.3×	623 1.2×	306 0.7×	543 1.3×	22	4.1k
Mikio Nishizawa Japan	37	2.1k 0.8×	490 1.0×	565 1.1×	255 0.6×	565 1.3×	150	4.3k
Jongsun Park South Korea	35	4.0k 1.4×	657 1.3×	591 1.2×	445 1.0×	632 1.5×	126	5.7k
Violeta Yu United States	15	3.2k 1.1×	774 1.5×	768 1.5×	352 0.8×	483 1.1×	29	4.4k

Countries citing papers authored by Fred L. Robinson

Since Specialization

Citations

This map shows the geographic impact of Fred L. Robinson's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Fred L. Robinson with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Fred L. Robinson more than expected).

Fields of papers citing papers by Fred L. Robinson

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Fred L. Robinson. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Fred L. Robinson. The network helps show where Fred L. Robinson may publish in the future.

Co-authorship network of co-authors of Fred L. Robinson

This figure shows the co-authorship network connecting the top 25 collaborators of Fred L. Robinson. A scholar is included among the top collaborators of Fred L. Robinson based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Fred L. Robinson. Fred L. Robinson is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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4 of 4 papers shown

1.

Robinson, Fred L., et al.. (2017). Schwann cell‐specific deletion of the endosomal PI 3‐kinase Vps34 leads to delayed radial sorting of axons, arrested myelination, and abnormal ErbB2‐ErbB3 tyrosine kinase signaling. Glia. 65(9). 1452–1470. 21 indexed citations

2.

Stippec, Steve, et al.. (2001). Hydrophobic as Well as Charged Residues in Both MEK1 and ERK2 Are Important for Their Proper Docking. Journal of Biological Chemistry. 276(28). 26509–26515. 79 indexed citations

3.

Chen, Zhu, Tara Beers Gibson, Fred L. Robinson, et al.. (2001). MAP Kinases. Chemical Reviews. 101(8). 2449–2476. 704 indexed citations breakdown →

4.

Pearson, Gray W., Fred L. Robinson, Tara Beers Gibson, et al.. (2001). Mitogen-Activated Protein (MAP) Kinase Pathways: Regulation and Physiological Functions*. Endocrine Reviews. 22(2). 153–183. 3411 indexed citations breakdown →

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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