Franciska Haneveld

2.4k citations
4 papers · 368 indexed · h-index 4
  • Neurology top 5%
    • Neuroblastoma Research and Treatments 4
    • Cancer, Hypoxia, and Metabolism 1
    • Protein Degradation and Inhibitors 2
    • Cancer-related gene regulation 1
    • Signaling Pathways in Disease 1
    • Mechanisms of cancer metastasis 1
    • Ubiquitin and proteasome pathways 1
    • Hippo pathway signaling and YAP/TAZ 1

Franciska Haneveld

4 papers receiving 363 citations

Peers

Franciska Haneveld
Comparison fields: 5 of 37
  • Neurology 264
  • Cancer Research 131
  • Molecular Biology 247
  • Oncology 71
  • Cell Biology 35
Replace Nina Weichert‐Leahey with:
Nina Weichert‐Leahey United States
Anders Valind Sweden
Noémie Braekeveldt Sweden
Moritz Gartlgruber Germany
Deepa Khazi United States
Sally Malik United Kingdom
Niloufar Javanmardi Sweden
Stefanie Schlierf Germany
Verena Körber Germany
Tim van Groningen Netherlands
Franciska Haneveld relative to Nina Weichert‐Leahey United States Nina Weichert‐Leahey's profile →
Citations per field
00.5×2.8×
Nina Weichert‐Leahey · 1×
Citations per year

Countries citing papers authored by Franciska Haneveld

Since Specialization
Citations

This map shows the geographic impact of Franciska Haneveld's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Franciska Haneveld with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Franciska Haneveld more than expected).

Fields of papers citing papers by Franciska Haneveld

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Franciska Haneveld. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Franciska Haneveld. The network helps show where Franciska Haneveld may publish in the future.

Co-authorship network

The 20 scholars most cited alongside Franciska Haneveld, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Franciska Haneveld Line = papers co-authored together Franciska Haneveld links everyone, so they are left out of the graph.

All Works

About Franciska Haneveld

Franciska Haneveld is a scholar working on Neurology, Cancer Research and Cell Biology, having authored 4 papers that have together received 368 indexed citations. Recurring topics across this work include Neuroblastoma Research and Treatments (4 papers), Protein Degradation and Inhibitors (2 papers), Cancer-related gene regulation (1 paper), Signaling Pathways in Disease (1 paper), Mechanisms of cancer metastasis (1 paper), Cancer, Hypoxia, and Metabolism (1 paper), Ubiquitin and proteasome pathways (1 paper) and Hippo pathway signaling and YAP/TAZ (1 paper). The work is most often cited by research in Neurology (264 citations), Cancer Research (131 citations) and Molecular Biology (247 citations). Franciska Haneveld has collaborated with scholars based in Netherlands. Frequent co-authors include Linda J. Valentijn, Rogier Versteeg, Jan Köster, Peter van Sluis, Johan van Nes, Marloes E.C. Broekmans, Jan J. Molenaar, Arjen Koppen, Saskia Hopman and C. Dilara Savci‐Heijink. Their work appears in journals such as Proceedings of the National Academy of Sciences, Nature Communications and Cancer Research.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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