Feng Qian

4.4k total citations
113 papers, 3.5k citations indexed

About

Feng Qian is a scholar working on Immunology, Molecular Biology and Pulmonary and Respiratory Medicine. According to data from OpenAlex, Feng Qian has authored 113 papers receiving a total of 3.5k indexed citations (citations by other indexed papers that have themselves been cited), including 49 papers in Immunology, 42 papers in Molecular Biology and 19 papers in Pulmonary and Respiratory Medicine. Recurrent topics in Feng Qian's work include Immune cells in cancer (13 papers), Interstitial Lung Diseases and Idiopathic Pulmonary Fibrosis (12 papers) and Immune Response and Inflammation (12 papers). Feng Qian is often cited by papers focused on Immune cells in cancer (13 papers), Interstitial Lung Diseases and Idiopathic Pulmonary Fibrosis (12 papers) and Immune Response and Inflammation (12 papers). Feng Qian collaborates with scholars based in China, United States and Macao. Feng Qian's co-authors include Richard D. Ye, Lei Sun, John W. Christman, Yaxian Wu, Zilong Wen, Huiqiong He, Kaikai Yu, Yudong Hu, Asrar B. Malik and Mei Huang and has published in prestigious journals such as Nature Communications, The Journal of Experimental Medicine and Journal of Clinical Oncology.

In The Last Decade

Feng Qian

106 papers receiving 3.5k citations

Peers

Feng Qian
Shanshan Wang China
Yufeng Tian United States
Marten Szibor Germany
Jun Guo China
Hui Liao China
Satoshi Ishii Japan
Laura Masuelli Italy
Manoj Kumar Barthwal India
Yoe‐Sik Bae South Korea
Albert Amberger Austria
Shanshan Wang China
Feng Qian
Citations per year, relative to Feng Qian Feng Qian (= 1×) peers Shanshan Wang

Countries citing papers authored by Feng Qian

Since Specialization
Citations

This map shows the geographic impact of Feng Qian's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Feng Qian with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Feng Qian more than expected).

Fields of papers citing papers by Feng Qian

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Feng Qian. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Feng Qian. The network helps show where Feng Qian may publish in the future.

Co-authorship network of co-authors of Feng Qian

This figure shows the co-authorship network connecting the top 25 collaborators of Feng Qian. A scholar is included among the top collaborators of Feng Qian based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Feng Qian. Feng Qian is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Yang, Xinyi, Ying Liu, Wen Li, et al.. (2025). Serum amyloid A3 aggravates bleomycin-induced pulmonary fibrosis through Krüppel-like factor 6-dependent interlukin-36α expression. Acta Pharmacologica Sinica. 46(12). 3244–3256.
2.
Sun, Lei, Haibo Zhang, Wen Li, et al.. (2025). LMO7 drives profibrotic fibroblast polarization and pulmonary fibrosis in mice through TGF-β signalling. Acta Pharmacologica Sinica. 46(7). 1930–1945. 4 indexed citations
3.
Zhao, Wenjuan, Feng Qian, Xinyi Yang, et al.. (2023). The Therapeutic Mechanism of Schisandrol A and Its Metabolites on Pulmonary Fibrosis Based on Plasma Metabonomics and Network Analysis. Drug Design Development and Therapy. Volume 17. 477–496. 4 indexed citations
4.
Wei, Ran, Xinyi Yang, Lu Xu, et al.. (2022). AMFR drives allergic asthma development by promoting alveolar macrophage–derived GM-CSF production. The Journal of Experimental Medicine. 219(5). 28 indexed citations
5.
Yan, Rui, et al.. (2022). Eucalyptol prevents bleomycin-induced pulmonary fibrosis and M2 macrophage polarization. European Journal of Pharmacology. 931. 175184–175184. 34 indexed citations
6.
Wu, Yaxian, Yingying Wang, Zhiqi Gao, et al.. (2021). Ethyl ferulate protects against lipopolysaccharide-induced acute lung injury by activating AMPK/Nrf2 signaling pathway. Acta Pharmacologica Sinica. 42(12). 2069–2081. 44 indexed citations
7.
Zhang, Yaping, et al.. (2020). Effects of Yangyin Liyan Decoction and Yuye Powder Blowing Throat on Serum Factors in Patients with Chronic Pharyngitis with Yin Deficiency and Pulmonary Dryness. 15(8). 1175–1178. 1 indexed citations
8.
Nie, Yunjuan, Dan Zhang, Feng Qian, & Yaxian Wu. (2019). Baccatin III ameliorates bleomycin-induced pulmonary fibrosis via suppression of TGF-β1 production and TGF-β1-induced fibroblast differentiation. International Immunopharmacology. 74. 105696–105696. 17 indexed citations
9.
Chen, Tingting, Yong Wang, Yang Yang, et al.. (2019). Gramicidin inhibits human gastric cancer cell proliferation, cell cycle and induced apoptosis. Biological Research. 52(1). 57–57. 24 indexed citations
10.
Wang, Zishu, Kaikai Yu, Yudong Hu, et al.. (2019). Schisantherin A induces cell apoptosis through ROS/JNK signaling pathway in human gastric cancer cells. Biochemical Pharmacology. 173. 113673–113673. 46 indexed citations
11.
12.
Hu, Yudong, Kaikai Yu, Gang Wang, et al.. (2018). Lanatoside C inhibits cell proliferation and induces apoptosis through attenuating Wnt/β-catenin/c-Myc signaling pathway in human gastric cancer cell. Biochemical Pharmacology. 150. 280–292. 66 indexed citations
13.
Liang, Qing, Ni Cheng, Yurong Liang, et al.. (2016). Identification of P-Rex1 as an anti-inflammatory and anti-fibrogenic target for pulmonary fibrosis. Scientific Reports. 6(1). 25785–25785. 16 indexed citations
14.
Lee, Yong Gyu, Sharmilee M. Nyenhuis, Evgeny Berdyshev, et al.. (2014). Recruited Alveolar Macrophages, in Response to Airway Epithelial–Derived Monocyte Chemoattractant Protein 1/CCL2, Regulate Airway Inflammation and Remodeling in Allergic Asthma. American Journal of Respiratory Cell and Molecular Biology. 52(6). 772–784. 142 indexed citations
15.
Chen, Mingjie, et al.. (2014). Serum amyloid A1 isoforms display different efficacy at Toll-like receptor 2 and formyl peptide receptor 2. Immunobiology. 219(12). 916–923. 49 indexed citations
16.
Di, Anke, Feng Qian, Takeshi Kawamura, et al.. (2011). The redox-sensitive cation channel TRPM2 modulates phagocyte ROS production and inflammation. Nature Immunology. 13(1). 29–34. 192 indexed citations
17.
Ren, Min, Qing Guo, Liang Guo, et al.. (2010). Polymerization of MIP‐1 chemokine (CCL3 and CCL4) and clearance of MIP‐1 by insulin‐degrading enzyme. The EMBO Journal. 29(23). 3952–3966. 134 indexed citations
18.
Qian, Feng, Ni Cheng, Emily J. Welch, et al.. (2009). A non‐redundant role for MKP5 in limiting ROS production and preventing LPS‐induced vascular injury. The EMBO Journal. 28(19). 2896–2907. 49 indexed citations
19.
Qian, Feng, Fenghua Zhen, Jin Xu, et al.. (2007). Distinct Functions for Different scl Isoforms in Zebrafish Primitive and Definitive Hematopoiesis. PLoS Biology. 5(5). e132–e132. 68 indexed citations
20.
Qian, Feng, Gerald M. Saidel, Damon Sutton, Agata A. Exner, & Jinming Gao. (2002). Combined modeling and experimental approach for the development of dual-release polymer millirods. Journal of Controlled Release. 83(3). 427–435. 23 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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