Yoe‐Sik Bae

5.7k total citations
175 papers, 4.6k citations indexed

About

Yoe‐Sik Bae is a scholar working on Molecular Biology, Immunology and Oncology. According to data from OpenAlex, Yoe‐Sik Bae has authored 175 papers receiving a total of 4.6k indexed citations (citations by other indexed papers that have themselves been cited), including 120 papers in Molecular Biology, 79 papers in Immunology and 37 papers in Oncology. Recurrent topics in Yoe‐Sik Bae's work include S100 Proteins and Annexins (43 papers), Immune Response and Inflammation (39 papers) and Sphingolipid Metabolism and Signaling (24 papers). Yoe‐Sik Bae is often cited by papers focused on S100 Proteins and Annexins (43 papers), Immune Response and Inflammation (39 papers) and Sphingolipid Metabolism and Signaling (24 papers). Yoe‐Sik Bae collaborates with scholars based in South Korea, United States and Japan. Yoe‐Sik Bae's co-authors include Ha Young Lee, Sung Ho Ryu, Sang Doo Kim, Suk‐Hwan Baek, Jeanho Yun, Pann‐Ghill Suh, Jong‐Young Kwak, Taehoon G. Lee, Jae Woong Shim and Mi-Kyoung Kim and has published in prestigious journals such as Journal of Biological Chemistry, Journal of Clinical Investigation and Nature Communications.

In The Last Decade

Yoe‐Sik Bae

174 papers receiving 4.6k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Yoe‐Sik Bae South Korea 39 2.8k 1.4k 604 475 435 175 4.6k
Baldur Sveinbjørnsson Norway 40 1.9k 0.7× 1.0k 0.7× 886 1.5× 620 1.3× 435 1.0× 109 4.3k
Suk‐Hwan Baek South Korea 33 1.6k 0.6× 836 0.6× 364 0.6× 390 0.8× 114 0.3× 92 3.0k
Wanghua Gong United States 46 3.3k 1.2× 2.9k 2.1× 1.6k 2.7× 671 1.4× 853 2.0× 103 6.5k
Rui Li China 43 2.3k 0.8× 1.9k 1.4× 926 1.5× 1.3k 2.6× 108 0.2× 244 6.3k
Patrice E. Poubelle Canada 36 1.6k 0.6× 1.4k 1.0× 364 0.6× 216 0.5× 81 0.2× 110 3.7k
Giorgio Berton Italy 42 2.1k 0.7× 3.1k 2.3× 615 1.0× 398 0.8× 111 0.3× 109 6.3k
Hydar Ali United States 42 2.4k 0.9× 3.0k 2.2× 783 1.3× 121 0.3× 268 0.6× 100 5.4k
Haiying Li China 35 2.5k 0.9× 628 0.5× 317 0.5× 697 1.5× 144 0.3× 157 4.2k
Ruaidhrı́ J. Carmody United Kingdom 28 2.0k 0.7× 1.6k 1.1× 488 0.8× 851 1.8× 62 0.1× 52 4.0k
Patrick P. McDonald Canada 38 1.3k 0.5× 2.4k 1.8× 538 0.9× 637 1.3× 89 0.2× 81 4.1k

Countries citing papers authored by Yoe‐Sik Bae

Since Specialization
Citations

This map shows the geographic impact of Yoe‐Sik Bae's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Yoe‐Sik Bae with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Yoe‐Sik Bae more than expected).

Fields of papers citing papers by Yoe‐Sik Bae

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Yoe‐Sik Bae. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Yoe‐Sik Bae. The network helps show where Yoe‐Sik Bae may publish in the future.

Co-authorship network of co-authors of Yoe‐Sik Bae

This figure shows the co-authorship network connecting the top 25 collaborators of Yoe‐Sik Bae. A scholar is included among the top collaborators of Yoe‐Sik Bae based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Yoe‐Sik Bae. Yoe‐Sik Bae is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Zabel, Brian A., et al.. (2025). SiglecF Expressing Neutrophils Exacerbate Th17-Mediated Autoimmune Neuroinflammation. Immune Network. 25(3). e19–e19.
2.
Lee, Min Seok, Soohyun Kim, Jong‐Hwan Lee, Yoe‐Sik Bae, & Sung Kyun Lee. (2025). Synovium‐on‐a‐Chip Reveals Fibroblast–Macrophage Crosstalk Underpinning Joint Homeostasis and Evaluation of Gout Therapies. Advanced Healthcare Materials. 14(18). e2501471–e2501471. 1 indexed citations
4.
Lee, Mingyu, et al.. (2024). CD200Rhigh neutrophils with dysfunctional autophagy establish systemic immunosuppression by increasing regulatory T cells. Cellular and Molecular Immunology. 21(4). 349–361. 6 indexed citations
5.
Lee, Mingyu, Byung‐Hyun Park, Joon Seong Park, et al.. (2024). Sphingosylphosphorylcholine Promotes Th9 Cell Differentiation Through Regulation of Smad3, STAT5, and β-Catenin Pathways. Immune Network. 24(6). e45–e45. 1 indexed citations
6.
Bae, Yoe‐Sik, et al.. (2023). The role and regulation of phospholipase D in metabolic disorders. Advances in Biological Regulation. 91. 100988–100988. 6 indexed citations
7.
Lee, Mingyu, et al.. (2023). Functional roles of sphingolipids in immunity and their implication in disease. Experimental & Molecular Medicine. 55(6). 1110–1130. 82 indexed citations
8.
Bae, Yoe‐Sik, et al.. (2023). WKYMVm ameliorates obesity by improving lipid metabolism and leptin signalling. Journal of Cellular and Molecular Medicine. 27(18). 2782–2791. 2 indexed citations
9.
Kwon, Soie, Yong Chul Kim, Yoe‐Sik Bae, et al.. (2022). Siglec-F–expressing neutrophils are essential for creating a profibrotic microenvironment in renal fibrosis. Journal of Clinical Investigation. 132(12). 49 indexed citations
10.
Lee, Mingyu, Sung Kyun Lee, Ho Lee, et al.. (2022). Unique characteristics of lung-resident neutrophils are maintained by PGE2/PKA/Tgm2-mediated signaling. Blood. 140(8). 889–899. 23 indexed citations
11.
Kim, Mi Young, Je Kyung Seong, Minyoung Lee, et al.. (2021). Targeting PLD2 in adipocytes augments adaptive thermogenesis by improving mitochondrial quality and quantity in mice. The Journal of Experimental Medicine. 219(2). 5 indexed citations
12.
Kim, Jihyun, Seokjin Ham, Sun Mi Choi, et al.. (2021). A unique population of neutrophils generated by air pollutant–induced lung damage exacerbates airway inflammation. Journal of Allergy and Clinical Immunology. 149(4). 1253–1269.e8. 35 indexed citations
13.
Park, Byung‐Hyun, et al.. (2021). Activation of formyl peptide receptor 1 elicits therapeutic effects against collagen‐induced arthritis. Journal of Cellular and Molecular Medicine. 25(18). 8936–8946. 10 indexed citations
14.
Kim, Hye Young, et al.. (2020). Novel Sca-1+ macrophages modulate the pathogenic progress of endotoxemia. Biochemical and Biophysical Research Communications. 533(1). 83–89. 6 indexed citations
15.
Lee, Ha Young, Hyung Sik Kim, Yong‐Soo Bae, et al.. (2020). A membrane-tethering pepducin derived from formyl peptide receptor 3 shows strong therapeutic effects against sepsis. Biochemical and Biophysical Research Communications. 524(1). 156–162. 6 indexed citations
16.
Yun, Jeanho, et al.. (2007). Serum amyloid A induces WISH cell apoptosis. Acta Pharmacologica Sinica. 28(1). 73–80. 9 indexed citations
17.
Kim, Gi‐Young, Yong‐Il Shin, Young-Chul Park, et al.. (2004). Proteoglycan isolated from Phellinus linteus inhibits tumor growth through mechanisms leading to an activation of CD11c+CD8+ DC and type I helper T cell‐dominant immune state. FEBS Letters. 576(3). 391–400. 55 indexed citations
18.
Bae, Yoe‐Sik, Ha Young Lee, Eunjin Jo, et al.. (2004). Identification of Peptides That Antagonize Formyl Peptide Receptor-Like 1-Mediated Signaling. The Journal of Immunology. 173(1). 607–614. 146 indexed citations
19.
Bae, Yoe‐Sik, Eunjin Jo, Jung Im Kim, et al.. (2003). Differential Activation of Formyl Peptide Receptor-Like 1 by Peptide Ligands. The Journal of Immunology. 171(12). 6807–6813. 75 indexed citations
20.
Bae, Yoe‐Sik, Youndong Kim, Jae Ho Kim, et al.. (2000). Independent Functioning of Cytosolic Phospholipase A2 and Phospholipase D1 in Trp-Lys-Tyr-Met-Val-D-Met-Induced Superoxide Generation in Human Monocytes. The Journal of Immunology. 164(8). 4089–4096. 37 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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