Fei-Feng Li
Impact in
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- MicroRNA in disease regulation
- Cancer-related molecular mechanisms research
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- Connexins and lens biology
- Circular RNAs in diseases
- Congenital heart defects research
Papers in
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- Congenital heart defects research 6
- Connexins and lens biology 4
- Oncology 6
- Co-authors
- Shu‐Lin Liu (17 shared papers)Xu Ma (5 shared papers)Meng Zhang (4 shared papers)Shuzhen Wang (3 shared papers)Peng Yan (6 shared papers)Yi Cui (1 shared paper)Yi Hu (1 shared paper)Xiaochen Han (1 shared paper)
In The Last Decade
Fei-Feng Li
27 papers receiving 374 citations
Peers
Comparison fields: 5 of 68
- Cancer Research 73
- Molecular Biology 256
- Ophthalmology 33
- Neurology 24
- Genetics 77
Countries citing papers authored by Fei-Feng Li
This map shows the geographic impact of Fei-Feng Li's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Fei-Feng Li with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Fei-Feng Li more than expected).
Fields of papers citing papers by Fei-Feng Li
This network shows the impact of papers produced by Fei-Feng Li. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Fei-Feng Li. The network helps show where Fei-Feng Li may publish in the future.
Co-authors
The 25 scholars most cited alongside Fei-Feng Li, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
Showing the 20 most-cited of 29 papers — load more, or switch the sort, to bring in the rest.
| # | Work | ||
|---|---|---|---|
| 1 | 2012 | 63 | |
| 2 | Nonsense mutation in the CRYBB2 gene causing autosomal dominant progressive polymorphic congenital coronary cataracts. | 2008 | 30 |
| 3 | 2011 | 29 | |
| 4 | 2011 | 24 | |
| 5 | 2014 | 21 | |
| 6 | 2012 | 20 | |
| 7 | A new mutation in BFSP2 (G1091A) causes autosomal dominant congenital lamellar cataracts. | 2008 | 19 |
| 8 | 2015 | 17 | |
| 9 | 2015 | 16 | |
| 10 | 2016 | 16 | |
| 11 | 2010 | 14 | |
| 12 | 2020 | 13 | |
| 13 | 2016 | 13 | |
| 14 | 2020 | 13 | |
| 15 | 2015 | 12 | |
| 16 | 2015 | 12 | |
| 17 | 2015 | 11 | |
| 18 | 2016 | 10 | |
| 19 | 2020 | 6 | |
| 20 | 2016 | 6 |
About Fei-Feng Li
Fei-Feng Li is a scholar working on Molecular Biology, Oncology, Epidemiology, Neurology and Rheumatology, having authored 29 papers that have together received 377 indexed citations. Recurring topics across this work include Congenital heart defects research (6 papers), Congenital Heart Disease Studies (5 papers), Connexins and lens biology (4 papers), Cerebrovascular and genetic disorders (4 papers), Moyamoya disease diagnosis and treatment (3 papers), Intraocular Surgery and Lenses (3 papers), Nonlinear Photonic Systems (2 papers) and Yersinia bacterium, plague, ectoparasites research (2 papers). The work is most often cited by research in Cancer Research (73 citations), Molecular Biology (256 citations), Ophthalmology (33 citations), Neurology (24 citations) and Genetics (77 citations). Fei-Feng Li has collaborated with scholars based in China and Canada. Frequent co-authors include Shu‐Lin Liu, Xu Ma, Meng Zhang, Shuzhen Wang, Peng Yan, Yi Cui, Yi Hu, Xiaochen Han, Hong-Fei Xia and Chang Gao. Their work appears in journals such as PLoS ONE, Oncotarget, Bioscience Reports, Gene and The European Physical Journal D.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.