Alfons Meindl

1.3k citations
14 papers · 490 indexed · h-index 11
Topics
DNA Repair Mechanisms (2 papers)BRCA gene mutations in cancer (2 papers)Cell death mechanisms and regulation (2 papers)
Partner nations
GermanySpainNetherlands

In The Last Decade

Alfons Meindl

14 papers receiving 483 citations

Peers

Alfons Meindl
Comparison fields: 5 of 72
  • Molecular Biology 332
  • Genetics 121
  • Oncology 102
  • Genetics 65
  • Cardiology and Cardiovascular Medicine 41
Replace Luke C. Gandolfo with:
Luke C. Gandolfo Australia
Suna Önengüt United States
Qing Yan United States
Haruka Hirose Japan
Crystal G. Wheeler United States
Rachel Soemedi United States
Katrin Schwarze Germany
Pedro Cruz United States
Sigve Nakken Norway
Marta Albajar Spain
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Citations per field
00.5×4.1×
Luke C. Gandolfo · 1×
Citations per year

Countries citing papers authored by Alfons Meindl

Since Specialization
Citations

This map shows the geographic impact of Alfons Meindl's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Alfons Meindl with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Alfons Meindl more than expected).

Fields of papers citing papers by Alfons Meindl

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Alfons Meindl. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Alfons Meindl. The network helps show where Alfons Meindl may publish in the future.

Co-authorship network of co-authors of Alfons Meindl

This figure shows the co-authorship network connecting the top 25 collaborators of Alfons Meindl. A scholar is included among the top collaborators of Alfons Meindl based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Alfons Meindl. Alfons Meindl is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

14 of 14 papers shown
#WorkIndexed citations
1 28
2 11
3 36
4 34
5 5
6 11
7 124
8 28
9 8
10 42
11 49
12
The delta subunit of rod phosphodiesterase interacts with the RCC1 homologous domain of RPGR
1
13 53
14
Evidence for genetic heterogeneity of malignant hyperthermia susceptibility.
60

About Alfons Meindl

Alfons Meindl is a scholar working on Genetics, Molecular Biology and Neurology, having authored 14 papers that have together received 490 indexed citations. Recurring topics across this work include DNA Repair Mechanisms (2 papers), BRCA gene mutations in cancer (2 papers) and Cell death mechanisms and regulation (2 papers). The work is most often cited by research in Genetics (65 citations), Molecular Biology (332 citations) and Genetics (121 citations). Alfons Meindl has collaborated with scholars based in Germany, Spain and Netherlands. Frequent co-authors include Heide Hellebrand, Rita K. Schmutzler, Thomas Meitinger, Juliane Ramser, Eric P. Hoffman, Robin D. Clark, Mary Ellen Ahearn, Peter Lichtner, Lisa Baumbach‐Reardon and Nina Ditsch. Their work appears in journals such as Nature Genetics, PLoS ONE and Journal of Allergy and Clinical Immunology.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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