Fátima Valdés‐Mora

2.9k total citations · 1 hit paper
31 papers, 1.5k citations indexed

About

Fátima Valdés‐Mora is a scholar working on Molecular Biology, Oncology and Cancer Research. According to data from OpenAlex, Fátima Valdés‐Mora has authored 31 papers receiving a total of 1.5k indexed citations (citations by other indexed papers that have themselves been cited), including 27 papers in Molecular Biology, 9 papers in Oncology and 4 papers in Cancer Research. Recurrent topics in Fátima Valdés‐Mora's work include Epigenetics and DNA Methylation (9 papers), Genomics and Chromatin Dynamics (7 papers) and Single-cell and spatial transcriptomics (6 papers). Fátima Valdés‐Mora is often cited by papers focused on Epigenetics and DNA Methylation (9 papers), Genomics and Chromatin Dynamics (7 papers) and Single-cell and spatial transcriptomics (6 papers). Fátima Valdés‐Mora collaborates with scholars based in Australia, Spain and United States. Fátima Valdés‐Mora's co-authors include David Gallego‐Ortega, Andrew M. K. Law, Susan J. Clark, Juan Carlos Lacal, Clare Stirzaker, Aaron L. Statham, Teresa Gómez del Pulgar, Shalima S. Nair, Jenny Z. Song and Yolanda Colino‐Sanguino and has published in prestigious journals such as Nature Communications, The Journal of Experimental Medicine and PLoS ONE.

In The Last Decade

Fátima Valdés‐Mora

29 papers receiving 1.5k citations

Hit Papers

align trajectories sort by overperformance

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Myeloid-Derived Suppressor Cells as a Therapeutic Target for Cancer

2020 346 citations Andrew M. K. Law, Fátima Valdés‐Mora et al. Cells profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Fátima Valdés‐Mora Australia	19	975	415	334	277	128	31	1.5k
Min-Zu Wu United States	11	937 1.0×	193 0.5×	217 0.6×	234 0.8×	126 1.0×	15	1.3k
Geoffroy Andrieux Germany	18	665 0.7×	354 0.9×	197 0.6×	184 0.7×	106 0.8×	65	1.2k
Sylvia Kaden Germany	16	755 0.8×	319 0.8×	379 1.1×	173 0.6×	108 0.8×	23	1.6k
Sandra Cristea United States	8	906 0.9×	898 2.2×	344 1.0×	173 0.6×	207 1.6×	9	1.5k
Christian D. Young United States	23	1.1k 1.1×	790 1.9×	318 1.0×	420 1.5×	229 1.8×	52	1.9k
Huiyin Lan China	11	931 1.0×	508 1.2×	608 1.8×	529 1.9×	141 1.1×	18	1.7k
Andrea Lunardi United States	20	1.1k 1.2×	511 1.2×	186 0.6×	342 1.2×	242 1.9×	35	1.6k
Fiona Furlong Ireland	18	887 0.9×	326 0.8×	170 0.5×	369 1.3×	100 0.8×	34	1.3k
Simeon Santourlidis Germany	24	1.0k 1.1×	238 0.6×	468 1.4×	310 1.1×	158 1.2×	53	1.7k
Rosa Camerlingo Italy	21	605 0.6×	666 1.6×	247 0.7×	341 1.2×	208 1.6×	41	1.3k

Countries citing papers authored by Fátima Valdés‐Mora

Since Specialization

Citations

This map shows the geographic impact of Fátima Valdés‐Mora's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Fátima Valdés‐Mora with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Fátima Valdés‐Mora more than expected).

Fields of papers citing papers by Fátima Valdés‐Mora

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Fátima Valdés‐Mora. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Fátima Valdés‐Mora. The network helps show where Fátima Valdés‐Mora may publish in the future.

Co-authorship network of co-authors of Fátima Valdés‐Mora

This figure shows the co-authorship network connecting the top 25 collaborators of Fátima Valdés‐Mora. A scholar is included among the top collaborators of Fátima Valdés‐Mora based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Fátima Valdés‐Mora. Fátima Valdés‐Mora is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Colino‐Sanguino, Yolanda, et al.. (2025). The Bermuda Triangle of paediatric brain cancers: epigenetics, developmental timing window and cell of origin. Cancer and Metastasis Reviews. 44(4). 73–73.

Ganguly, Debolina, Marcel O. Schmidt, Noah Sorrelle, et al.. (2023). Pleiotrophin drives a prometastatic immune niche in breast cancer. The Journal of Experimental Medicine. 220(5). 8 indexed citations

Law, Andrew M. K., et al.. (2021). Advancements in 3D Cell Culture Systems for Personalizing Anti-Cancer Therapies. Frontiers in Oncology. 11. 782766–782766. 61 indexed citations

Fuente, Laura Rodríguez de la, Andrew M. K. Law, David Gallego‐Ortega, & Fátima Valdés‐Mora. (2021). Tumor dissociation of highly viable cell suspensions for single-cell omic analyses in mouse models of breast cancer. STAR Protocols. 2(4). 100841–100841. 11 indexed citations

Law, Andrew M. K., Fátima Valdés‐Mora, & David Gallego‐Ortega. (2020). Myeloid-Derived Suppressor Cells as a Therapeutic Target for Cancer. Cells. 9(3). 561–561. 346 indexed citations breakdown →

Achinger-Kawecka, Joanna, Fátima Valdés‐Mora, Phuc‐Loi Luu, et al.. (2020). Epigenetic reprogramming at estrogen-receptor binding sites alters 3D chromatin landscape in endocrine-resistant breast cancer. Nature Communications. 11(1). 320–320. 96 indexed citations

Khoury, Amanda, Joanna Achinger-Kawecka, Grady C. Smith, et al.. (2020). Constitutively bound CTCF sites maintain 3D chromatin architecture and long-range epigenetically regulated domains. Nature Communications. 11(1). 54–54. 64 indexed citations

Colino‐Sanguino, Yolanda, Evan M. Cornett, Grady C. Smith, et al.. (2019). A Read/Write Mechanism Connects p300 Bromodomain Function to H2A.Z Acetylation. iScience. 21. 773–788. 18 indexed citations

Valdés‐Mora, Fátima, Kristina Handler, Andrew M. K. Law, et al.. (2018). Single-Cell Transcriptomics in Cancer Immunobiology: The Future of Precision Oncology. Frontiers in Immunology. 9. 2582–2582. 43 indexed citations

10.

Valdés‐Mora, Fátima, Cathryn M. Gould, Yolanda Colino‐Sanguino, et al.. (2017). Acetylated histone variant H2A.Z is involved in the activation of neo-enhancers in prostate cancer. Nature Communications. 8(1). 1346–1346. 64 indexed citations

11.

Stirzaker, Clare, Jenny Z. Song, Wai Tong Ng, et al.. (2016). Methyl-CpG-binding protein MBD2 plays a key role in maintenance and spread of DNA methylation at CpG islands and shores in cancer. Oncogene. 36(10). 1328–1338. 56 indexed citations

12.

Valdés‐Mora, Fátima & Susan J. Clark. (2014). Prostate cancer epigenetic biomarkers: next-generation technologies. Oncogene. 34(13). 1609–1618. 38 indexed citations

13.

Stone, Andrew, Mark J. Cowley, Fátima Valdés‐Mora, et al.. (2013). BCL-2 Hypermethylation Is a Potential Biomarker of Sensitivity to Antimitotic Chemotherapy in Endocrine-Resistant Breast Cancer. Molecular Cancer Therapeutics. 12(9). 1874–1885. 39 indexed citations

14.

Valdés‐Mora, Fátima, Jenny Z. Song, Aaron L. Statham, et al.. (2011). Acetylation of H2A.Z is a key epigenetic modification associated with gene deregulation and epigenetic remodeling in cancer. Genome Research. 22(2). 307–321. 141 indexed citations

15.

Valdés‐Mora, Fátima, et al.. (2011). TWIST1 (twist homolog 1 (Drosophila)). Atlas of Genetics and Cytogenetics in Oncology and Haematology.

16.

Lee, Heather, Rebecca A. Hinshelwood, Toula Bouras, et al.. (2011). Lineage Specific Methylation of the Elf5 Promoter in Mammary Epithelial Cells. Stem Cells. 29(10). 1611–1619. 36 indexed citations

17.

Gallego‐Ortega, David, Teresa Gómez del Pulgar, Fátima Valdés‐Mora, Arancha Cebrián, & Juan Carlos Lacal. (2010). Involvement of human choline kinase alpha and beta in carcinogenesis: A different role in lipid metabolism and biological functions. Advances in Enzyme Regulation. 51(1). 183–194. 52 indexed citations

18.

Gallego‐Ortega, David, Ana Ramı́rez de Molina, Fátima Valdés‐Mora, et al.. (2009). Differential Role of Human Choline Kinase α and β Enzymes in Lipid Metabolism: Implications in Cancer Onset and Treatment. PLoS ONE. 4(11). e7819–e7819. 86 indexed citations

19.

Gallego‐Ortega, David, et al.. (2008). Differential role of choline kinase alpha and beta isoforms in human carcinogenesis. European Journal of Cancer Supplements. 6(9). 26–26. 4 indexed citations

20.

Pulgar, Teresa Gómez del, Eva Bandrés, Carolina Espina, et al.. (2007). Differential expression of Rac1 identifies its target genes and its contribution to progression of colorectal cancer. The International Journal of Biochemistry & Cell Biology. 39(12). 2289–2302. 26 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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