Fanglian Chen

3.3k total citations · 1 hit paper
64 papers, 2.5k citations indexed

About

Fanglian Chen is a scholar working on Molecular Biology, Neurology and Neurology. According to data from OpenAlex, Fanglian Chen has authored 64 papers receiving a total of 2.5k indexed citations (citations by other indexed papers that have themselves been cited), including 41 papers in Molecular Biology, 30 papers in Neurology and 19 papers in Neurology. Recurrent topics in Fanglian Chen's work include Traumatic Brain Injury and Neurovascular Disturbances (19 papers), Extracellular vesicles in disease (16 papers) and Neuroinflammation and Neurodegeneration Mechanisms (14 papers). Fanglian Chen is often cited by papers focused on Traumatic Brain Injury and Neurovascular Disturbances (19 papers), Extracellular vesicles in disease (16 papers) and Neuroinflammation and Neurodegeneration Mechanisms (14 papers). Fanglian Chen collaborates with scholars based in China, United States and Montenegro. Fanglian Chen's co-authors include Ping Lei, Xintong Ge, Zhaoli Han, Zhenyu Yin, Shan Huang, Jianning Zhang, Mengtian Guo, Ying Li, Ping Lei and Jianning Zhang and has published in prestigious journals such as Brain Research, The FASEB Journal and Critical Care Medicine.

In The Last Decade

Fanglian Chen

56 papers receiving 2.4k citations

Hit Papers

Increased miR‐124‐3p in microglial exosomes following tra... 2017 2026 2020 2023 2017 100 200 300

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Fanglian Chen China 25 1.6k 732 551 507 294 64 2.5k
Xintong Ge China 23 1.3k 0.8× 668 0.9× 486 0.9× 464 0.9× 245 0.8× 35 2.0k
Zhaoli Han China 22 1.1k 0.7× 633 0.9× 460 0.8× 400 0.8× 232 0.8× 38 1.8k
Renée J. Turner Australia 26 1.1k 0.7× 543 0.7× 655 1.2× 616 1.2× 549 1.9× 52 2.5k
Yaying Song China 23 1.0k 0.6× 522 0.7× 585 1.1× 301 0.6× 133 0.5× 50 1.7k
Erez Eitan United States 24 2.4k 1.5× 984 1.3× 540 1.0× 298 0.6× 207 0.7× 35 3.3k
Rongcai Jiang China 32 1.6k 1.0× 631 0.9× 478 0.9× 1.4k 2.8× 443 1.5× 143 3.5k
Qingyi Ma United States 28 1.1k 0.7× 347 0.5× 1.0k 1.9× 920 1.8× 329 1.1× 52 3.1k
Omolara O. Ogunshola Switzerland 28 1.1k 0.7× 541 0.7× 768 1.4× 241 0.5× 217 0.7× 55 2.8k
Yirong Yang United States 21 541 0.3× 325 0.4× 737 1.3× 267 0.5× 323 1.1× 60 1.9k
Woo Jean Kim South Korea 29 1.0k 0.6× 320 0.4× 594 1.1× 148 0.3× 169 0.6× 55 2.5k

Countries citing papers authored by Fanglian Chen

Since Specialization
Citations

This map shows the geographic impact of Fanglian Chen's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Fanglian Chen with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Fanglian Chen more than expected).

Fields of papers citing papers by Fanglian Chen

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Fanglian Chen. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Fanglian Chen. The network helps show where Fanglian Chen may publish in the future.

Co-authorship network of co-authors of Fanglian Chen

This figure shows the co-authorship network connecting the top 25 collaborators of Fanglian Chen. A scholar is included among the top collaborators of Fanglian Chen based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Fanglian Chen. Fanglian Chen is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Cao, Yiyao, Jianye Xu, Bo Chen, et al.. (2025). Inhibition of S100A8/A9 ameliorates neuroinflammation by blocking NET formation following traumatic brain injury. Redox Biology. 81. 103532–103532. 4 indexed citations
3.
Dai, Li, Wei Zhang, Zihan Zhang, et al.. (2025). DP1 Receptor Blockade Attenuates Microglial Senescence and Cognitive Decline Caused by PTGDS in Exosomes From Aged Brains. Aging Cell. 24(11). e70228–e70228.
4.
Cheng, Fangyuan, et al.. (2025). Neuronal PD-L1 suppression attenuates ischemic stroke injury via PD-1/RFX1 axis-mediated microglial polarization. International Journal of Surgery. 111(11). 7711–7726.
5.
Cheng, Fangyuan, Conglin Wang, Jieying Zhang, et al.. (2024). Identification of toll-like receptor 2 as a key regulator of neuronal apoptosis in vascular dementia by bioinformatics analysis and experimental validation. Experimental Gerontology. 193. 112464–112464.
6.
Wang, Conglin, et al.. (2024). Meta‐Analysis of the Therapeutic Effects of Stem Cell‐Derived Extracellular Vesicles in Rodent Models of Hemorrhagic Stroke. Stem Cells International. 2024(1). 3390446–3390446. 2 indexed citations
8.
Liu, Xilei, et al.. (2024). Gut microbiota–astrocyte axis: new insights into age-related cognitive decline. Neural Regeneration Research. 20(4). 990–1008. 13 indexed citations
9.
Liao, Pan, et al.. (2024). Therapeutic potential of microglia-derived extracellular vesicles in ischemic stroke. International Immunopharmacology. 139. 112712–112712. 6 indexed citations
10.
Guo, Mengtian, et al.. (2023). IL-33/ST2 Axis: A Potential Therapeutic Target in Neurodegenerative Diseases. Biomolecules. 13(10). 1494–1494. 9 indexed citations
11.
Shi, Mingming, Yiyao Cao, Xu Zhang, et al.. (2023). Inhibition of S100a8/A9 Ameliorates Neuroinflammation by Blocking NET Formation Via AMPK/Nrf2/HO-1 Pathway after Traumatic Brain Injury in Mice. SSRN Electronic Journal. 1 indexed citations
12.
Jia, Hao‐Ran, Xilei Liu, Yiyao Cao, et al.. (2023). Deferoxamine ameliorates neurological dysfunction by inhibiting ferroptosis and neuroinflammation after traumatic brain injury. Brain Research. 1812. 148383–148383. 38 indexed citations
14.
Ge, Xintong, Wenzhu Li, Fanglian Chen, et al.. (2021). Red Cell Distribution Width to Platelet Count Ratio: A Promising Routinely Available Indicator of Mortality for Acute Traumatic Brain Injury. Journal of Neurotrauma. 39(1-2). 159–171. 12 indexed citations
15.
Guo, Mengtian, Zhenyu Yin, Fanglian Chen, & Ping Lei. (2020). Mesenchymal stem cell-derived exosome: a promising alternative in the therapy of Alzheimer’s disease. Alzheimer s Research & Therapy. 12(1). 109–109. 138 indexed citations
16.
Li, Dai, Shan Huang, Zhenyu Yin, et al.. (2019). Increases in miR-124-3p in Microglial Exosomes Confer Neuroprotective Effects by Targeting FIP200-Mediated Neuronal Autophagy Following Traumatic Brain Injury. Neurochemical Research. 44(8). 1903–1923. 108 indexed citations
17.
Yin, Zhenyu, Zhaoli Han, Tianpeng Hu, et al.. (2019). Neuron-derived exosomes with high miR-21-5p expression promoted polarization of M1 microglia in culture. Brain Behavior and Immunity. 83. 270–282. 128 indexed citations
18.
Han, Zhaoli, Xintong Ge, Jin Tan, et al.. (2015). Establishment of Lipofection Protocol for Efficient miR-21 Transfection into Cortical Neurons In Vitro. DNA and Cell Biology. 34(12). 703–709. 23 indexed citations
20.
Zhang, Yongqiang, Ying Li, Shaobo Wang, et al.. (2013). Transplantation of expanded endothelial colony-forming cells improved outcomes of traumatic brain injury in a mouse model. Journal of Surgical Research. 185(1). 441–449. 42 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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