Fabiane Sônego
- Immunology top 2%
- Molecular Biology
- Epidemiology top 10%
- Surgery
- Physiology
- Co-authors
- Fernando Q. CunhaJosé C. Alves‐FilhoDaniele C. NascimentoFabrício Oliveira SoutoFoo Y. LiewAndressa de FreitasFernanda V. S. CastanheiraWaldiceu A. Verri
- Topics
- Immune Response and Inflammation (11 papers)Neutrophil, Myeloperoxidase and Oxidative Mechanisms (8 papers)Sepsis Diagnosis and Treatment (6 papers)
- Partner nations
- BrazilUnited KingdomFrance
In The Last Decade
Fabiane Sônego
33 papers receiving 1.6k citations
Peers
Comparison fields: 5 of 101
- Immunology 1.1k
- Molecular Biology 418
- Epidemiology 348
- Surgery 193
- Physiology 159
Countries citing papers authored by Fabiane Sônego
This map shows the geographic impact of Fabiane Sônego's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Fabiane Sônego with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Fabiane Sônego more than expected).
Fields of papers citing papers by Fabiane Sônego
This network shows the impact of papers produced by Fabiane Sônego. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Fabiane Sônego. The network helps show where Fabiane Sônego may publish in the future.
Co-authorship network of co-authors of Fabiane Sônego
This figure shows the co-authorship network connecting the top 25 collaborators of Fabiane Sônego. A scholar is included among the top collaborators of Fabiane Sônego based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Fabiane Sônego. Fabiane Sônego is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 0 | |
| 2 | 12 | |
| 3 | 0 | |
| 4 | 6 | |
| 5 | 2 | |
| 6 | 21 | |
| 7 | 286 | |
| 8 | 55 | |
| 9 | PPAR-γ/IL-10 axis inhibits MyD88 expression and ameliorates murine polymicrobial sepsis | 1 |
| 10 | 13 | |
| 11 | 55 | |
| 12 | 29 | |
| 13 | 15 | |
| 14 | 1 | |
| 15 | 30 | |
| 16 | 10 | |
| 17 | 388 | |
| 18 | 25 | |
| 19 | 80 | |
| 20 | 77 |
About Fabiane Sônego
Fabiane Sônego is a scholar working on Immunology, Critical Care and Intensive Care Medicine and Neurology, having authored 37 papers that have together received 1.6k indexed citations. Recurring topics across this work include Immune Response and Inflammation (11 papers), Neutrophil, Myeloperoxidase and Oxidative Mechanisms (8 papers) and Sepsis Diagnosis and Treatment (6 papers). The work is most often cited by research in Immunology (1.1k citations), Critical Care and Intensive Care Medicine (72 citations) and Neurology (94 citations). Fabiane Sônego has collaborated with scholars based in Brazil, United Kingdom and France. Frequent co-authors include Fernando Q. Cunha, José C. Alves‐Filho, Daniele C. Nascimento, Fabrício Oliveira Souto, Foo Y. Liew, Andressa de Freitas, Fernanda V. S. Castanheira, Waldiceu A. Verri, Aníbal Basile-Filho and Maria Auxiliadora‐Martins. Their work appears in journals such as Proceedings of the National Academy of Sciences, Nature Medicine and Nature Communications.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.