Eva Veselá

471 citations
9 papers · 349 · h-index 7

Impact in

Papers in

    • DNA Repair Mechanisms 5
    • Epigenetics and DNA Methylation 2
    • Cancer therapeutics and mechanisms 1
    • Cancer-related Molecular Pathways 1
    • Polyomavirus and related diseases 1

Eva Veselá

9 papers receiving 347 citations

Peers

Eva Veselá
Comparison fields: 5 of 53
  • Otorhinolaryngology 125
  • Periodontics 32
  • Oncology 113
  • Cell Biology 51
  • Cancer Research 46
Replace Marcia Davis with:
Marcia Davis Australia
Wei-Wen Jiang United States
Vali A. Papadimitrakopoulou United States
Shahla Bari United States
Natália R. Costa Portugal
Monique Ummelen Netherlands
Natalie Naranjo Galloni Canada
S Kiguwa United Kingdom
Anne-Katrin Emde Germany
Bo-Hua Su China
Eva Veselá relative to Marcia Davis Australia Marcia Davis's profile →
Citations per field
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Citations per year

Countries citing papers authored by Eva Veselá

Since Specialization
Citations

This map shows the geographic impact of Eva Veselá's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Eva Veselá with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Eva Veselá more than expected).

Fields of papers citing papers by Eva Veselá

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Eva Veselá. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Eva Veselá. The network helps show where Eva Veselá may publish in the future.

Co-authors

The 25 scholars most cited alongside Eva Veselá, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Eva Veselá Line = papers co-authored together Eva Veselá links everyone, so they are left out of the graph.

All Works

9 of 9 papers shown
#Work
1 200780
2 201775
3 201062
4 201362
5 202031
6 199719
7 201616
8 20163
9
Seroprevalence of hantavirus antibodies among chronic hemodialysis patients in the Czech Republic.
20101

About Eva Veselá

Eva Veselá is a scholar working on Molecular Biology, Oncology, Otorhinolaryngology, Infectious Diseases and Surgery, having authored 9 papers that have together received 349 indexed citations. Recurring topics across this work include DNA Repair Mechanisms (5 papers), Head and Neck Cancer Studies (2 papers), Epigenetics and DNA Methylation (2 papers), Cancer-related Molecular Pathways (1 paper), Cancer therapeutics and mechanisms (1 paper), Light effects on plants (1 paper), Polyomavirus and related diseases (1 paper) and Genetics and Neurodevelopmental Disorders (1 paper). The work is most often cited by research in Otorhinolaryngology (125 citations), Periodontics (32 citations), Oncology (113 citations), Cell Biology (51 citations) and Cancer Research (46 citations). Eva Veselá has collaborated with scholars based in Czechia, Denmark and United Kingdom. Frequent co-authors include Martin Mistrík, Eva Hamšíková, Ruth Tachezy, Jan Klozar, Martina Saláková, Jana Šmahelová, Jan Betka, Jiří Bártek, Bohumír Procházka and Viera Ludvı́ková. Their work appears in journals such as Genes & Development, Artificial Organs, Scientific Reports, Biomolecules and Nature Communications.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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