Eva Podstawa

673 total citations
8 papers, 495 citations indexed

About

Eva Podstawa is a scholar working on Pathology and Forensic Medicine, Rheumatology and Genetics. According to data from OpenAlex, Eva Podstawa has authored 8 papers receiving a total of 495 indexed citations (citations by other indexed papers that have themselves been cited), including 6 papers in Pathology and Forensic Medicine, 5 papers in Rheumatology and 4 papers in Genetics. Recurrent topics in Eva Podstawa's work include Autoimmune Bullous Skin Diseases (6 papers), Urticaria and Related Conditions (5 papers) and Coagulation, Bradykinin, Polyphosphates, and Angioedema (4 papers). Eva Podstawa is often cited by papers focused on Autoimmune Bullous Skin Diseases (6 papers), Urticaria and Related Conditions (5 papers) and Coagulation, Bradykinin, Polyphosphates, and Angioedema (4 papers). Eva Podstawa collaborates with scholars based in Germany, Italy and Romania. Eva Podstawa's co-authors include Michael Hertl, Angela Nagel, Rüdiger Eming, Dirk Debus, Christoph Becker, Christian H.K. Lehmann, Gheorghe Hundorfean, Raja Atreya, Stefan Fichtner‐Feigl and Tilman T. Rau and has published in prestigious journals such as Gut, Journal of Investigative Dermatology and Cell Reports.

In The Last Decade

Eva Podstawa

8 papers receiving 489 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Eva Podstawa Germany	8	273	227	200	163	64	8	495
Björn Löfström Sweden	8	195 0.7×	225 1.0×	80 0.4×	112 0.7×	23 0.4×	8	379
N. Hunzelmann Germany	14	303 1.1×	118 0.5×	78 0.4×	92 0.6×	53 0.8×	26	519
Marco Foppoli Italy	12	392 1.4×	82 0.4×	291 1.5×	159 1.0×	16 0.3×	30	738
Takeshi Echigo Japan	9	173 0.6×	158 0.7×	118 0.6×	87 0.5×	15 0.2×	18	366
Ayumi Okuyama Japan	11	95 0.3×	206 0.9×	100 0.5×	91 0.6×	14 0.2×	23	445
JL Huret France	6	301 1.1×	39 0.2×	128 0.6×	91 0.6×	24 0.4×	100	521
Trieneke C. G. Timmer Netherlands	6	74 0.3×	254 1.1×	31 0.2×	174 1.1×	23 0.4×	8	442
Jun Odawara Japan	12	226 0.8×	54 0.2×	117 0.6×	29 0.2×	29 0.5×	32	555
Stuart Weisman United States	5	68 0.2×	110 0.5×	71 0.4×	136 0.8×	44 0.7×	7	415
Le Huu Doanh Vietnam	10	170 0.6×	41 0.2×	37 0.2×	304 1.9×	42 0.7×	28	534

Countries citing papers authored by Eva Podstawa

Since Specialization

Citations

This map shows the geographic impact of Eva Podstawa's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Eva Podstawa with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Eva Podstawa more than expected).

Fields of papers citing papers by Eva Podstawa

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Eva Podstawa. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Eva Podstawa. The network helps show where Eva Podstawa may publish in the future.

Co-authorship network of co-authors of Eva Podstawa

This figure shows the co-authorship network connecting the top 25 collaborators of Eva Podstawa. A scholar is included among the top collaborators of Eva Podstawa based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Eva Podstawa. Eva Podstawa is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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8 of 8 papers shown

1.

Gámez‐Belmonte, Reyes, Mousumi Mahapatro, Lena Erkert, et al.. (2022). Epithelial presenilin-1 drives colorectal tumour growth by controlling EGFR-COX2 signalling. Gut. 72(6). 1155–1166. 8 indexed citations

2.

Martini, Eva, Nadine Wittkopf, Claudia Günther, et al.. (2016). Loss of Survivin in Intestinal Epithelial Progenitor Cells Leads to Mitotic Catastrophe and Breakdown of Gut Immune Homeostasis. Cell Reports. 14(5). 1062–1073. 22 indexed citations

3.

Chiriac, Mircea T., Eva Podstawa, Christian H.K. Lehmann, et al.. (2014). IL-9 and its receptor are predominantly involved in the pathogenesis of UC. Gut. 64(5). 743–755. 153 indexed citations

4.

Horváth, Barbara, A. Niedermeier, Eva Podstawa, et al.. (2010). IgA autoantibodies in the pemphigoids and linear IgA bullous dermatosis. Experimental Dermatology. 19(7). 648–653. 36 indexed citations

5.

Feliciani, Claudio, Giacomo Caldarola, Andrea Kneisel, et al.. (2009). IgG autoantibody reactivity against bullous pemphigoid (BP) 180 and BP230 in elderly patients with pruritic dermatoses. British Journal of Dermatology. 161(2). 306–312. 35 indexed citations

6.

Nagel, Angela, Eva Podstawa, Markus Eickmann, et al.. (2009). Rituximab Mediates a Strong Elevation of B-Cell-Activating Factor Associated with Increased Pathogen-Specific IgG but Not Autoantibodies in Pemphigus Vulgaris. Journal of Investigative Dermatology. 129(9). 2202–2210. 57 indexed citations

7.

Nagel, Angela, Eva Podstawa, Nicolas Hunzelmann, et al.. (2009). Clinical activity of pemphigus vulgaris relates to IgE autoantibodies against desmoglein 3. Clinical Immunology. 134(3). 320–330. 53 indexed citations

8.

Eming, Rüdiger, et al.. (2008). Rituximab Exerts a Dual Effect in Pemphigus Vulgaris. Journal of Investigative Dermatology. 128(12). 2850–2858. 131 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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