Éva Forgács

3.1k citations
39 papers · 2.4k indexed · 1 hit paper · h-index 21

Impact in

    • Proteoglycans and glycosaminoglycans research
    • Cellular Mechanics and Interactions
    • Epigenetics and DNA Methylation
    • Cancer-related gene regulation
    • RNA modifications and cancer
    • Muscle Physiology and Disorders
    • PI3K/AKT/mTOR signaling in cancer

Papers in

Éva Forgács

33 papers receiving 2.3k citations

Hit Papers

Epigenetic Inactivation of RASSF1A in Lung and Breast Cancers and Malignant Phenotype Suppression 2001 · 659 citations
6592001202620092017200400600

Peers

Éva Forgács
Comparison fields: 5 of 133
  • Cell Biology 481
  • Molecular Biology 1.7k
  • Cardiology and Cardiovascular Medicine 461
  • Cancer Research 256
  • Structural Biology 23
Replace Lisa D. Belmont with:
Lisa D. Belmont United States
Evelyne Friederich France
Mark Holt United Kingdom
Eugen Kerkhoff Germany
Joseph Loureiro United States
Alison J. North United States
Paul Mangeat France
Maryse Bailly United Kingdom
Steven J. Winder United Kingdom
James L. Lessard United States
Éva Forgács relative to Lisa D. Belmont United States Lisa D. Belmont's profile →
Citations per field
00.5×3.7×
Lisa D. Belmont · 1×
Citations per year

Countries citing papers authored by Éva Forgács

Since Specialization
Citations

This map shows the geographic impact of Éva Forgács's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Éva Forgács with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Éva Forgács more than expected).

Fields of papers citing papers by Éva Forgács

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Éva Forgács. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Éva Forgács. The network helps show where Éva Forgács may publish in the future.

Co-authors

The 25 scholars most cited alongside Éva Forgács, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Éva Forgács Line = papers co-authored together Éva Forgács links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown
#Work
1 20256
2 20221
3 201582
4 201567
5 20132
6 201119
7 200833
8 2008103
9 200742
10 200294
11 2001257
12 200161
13 200117
14
Epigenetic Inactivation of RASSF1A in Lung and Breast Cancers and Malignant Phenotype Suppression
Hit paper breakdown →
2001659
15 2000125
16 200030
17 1998166
18 199725
19 19952
20 19951

About Éva Forgács

Éva Forgács is a scholar working on Architecture, Visual Arts and Performing Arts, Cardiology and Cardiovascular Medicine, Cell Biology and Virology, having authored 39 papers that have together received 2.4k indexed citations. Recurring topics across this work include Cardiomyopathy and Myosin Studies (13 papers), Muscle Physiology and Disorders (7 papers), Cardiovascular Effects of Exercise (7 papers), Art, Politics, and Modernism (5 papers), Force Microscopy Techniques and Applications (4 papers), Genetic factors in colorectal cancer (4 papers), Cellular Mechanics and Interactions (4 papers) and Cancer-related gene regulation (3 papers). The work is most often cited by research in Cell Biology (481 citations), Molecular Biology (1.7k citations), Cardiology and Cardiovascular Medicine (461 citations), Cancer Research (256 citations) and Structural Biology (23 citations). Éva Forgács has collaborated with scholars based in United States, United Kingdom and Hungary. Frequent co-authors include John D. Minna, Yoshitaka Sekido, Howard D. White, Farida Latif, Adi F. Gazdar, Sara Milchgrub, Kwun M. Fong, Sabine Zöchbauer‐Müller, Donald A. Winkelmann and James R. Sellers. Their work appears in journals such as Journal of Biological Chemistry, Oncogene, Nature, Biochemistry and Proceedings of the National Academy of Sciences.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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