Ernest J. Freeman
- Molecular Biology
- Cardiology and Cardiovascular Medicine top 10%
- Endocrinology, Diabetes and Metabolism top 10%
- Physiology
- Mechanical Engineering
- Co-authors
- Jennifer McDonoughE. Ann TallantCarlos M. FerrarioRobert J. ClementsGuy ChisolmRobert V. DormanDavid M. TerrianNaveen Kumar Singhal
- Topics
- Protein Kinase Regulation and GTPase Signaling (6 papers)Renin-Angiotensin System Studies (6 papers)Receptor Mechanisms and Signaling (5 papers)
- Partner nations
- United StatesCanadaIndia
In The Last Decade
Ernest J. Freeman
27 papers receiving 1.1k citations
Peers
Comparison fields: 5 of 102
- Molecular Biology 464
- Cardiology and Cardiovascular Medicine 287
- Endocrinology, Diabetes and Metabolism 163
- Physiology 142
- Mechanical Engineering 136
Countries citing papers authored by Ernest J. Freeman
This map shows the geographic impact of Ernest J. Freeman's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Ernest J. Freeman with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Ernest J. Freeman more than expected).
Fields of papers citing papers by Ernest J. Freeman
This network shows the impact of papers produced by Ernest J. Freeman. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Ernest J. Freeman. The network helps show where Ernest J. Freeman may publish in the future.
Co-authorship network of co-authors of Ernest J. Freeman
This figure shows the co-authorship network connecting the top 25 collaborators of Ernest J. Freeman. A scholar is included among the top collaborators of Ernest J. Freeman based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Ernest J. Freeman. Ernest J. Freeman is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 5 | |
| 2 | 18 | |
| 3 | 23 | |
| 4 | 24 | |
| 5 | 6 | |
| 6 | 53 | |
| 7 | 54 | |
| 8 | 62 | |
| 9 | 4 | |
| 10 | 95 | |
| 11 | 6 | |
| 12 | 46 | |
| 13 | 60 | |
| 14 | 8 | |
| 15 | 23 | |
| 16 | 14 | |
| 17 | 14 | |
| 18 | 27 | |
| 19 | 18 | |
| 20 | 72 |
About Ernest J. Freeman
Ernest J. Freeman is a scholar working on Cardiology and Cardiovascular Medicine, Molecular Biology and Neurology, having authored 28 papers that have together received 1.1k indexed citations. Recurring topics across this work include Protein Kinase Regulation and GTPase Signaling (6 papers), Renin-Angiotensin System Studies (6 papers) and Receptor Mechanisms and Signaling (5 papers). The work is most often cited by research in Developmental Neuroscience (57 citations), Cardiology and Cardiovascular Medicine (287 citations) and Neurology (94 citations). Ernest J. Freeman has collaborated with scholars based in United States, Canada and India. Frequent co-authors include Jennifer McDonough, E. Ann Tallant, Carlos M. Ferrario, Robert J. Clements, Guy Chisolm, Robert V. Dorman, David M. Terrian, Naveen Kumar Singhal, Mary L. Ruehr and Shuo Li. Their work appears in journals such as Journal of Neuroscience, PLoS ONE and Annals of Neurology.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.