Erling A. Høivik

3.9k total citations
55 papers, 1.6k citations indexed

About

Erling A. Høivik is a scholar working on Molecular Biology, Cancer Research and Obstetrics and Gynecology. According to data from OpenAlex, Erling A. Høivik has authored 55 papers receiving a total of 1.6k indexed citations (citations by other indexed papers that have themselves been cited), including 26 papers in Molecular Biology, 22 papers in Cancer Research and 21 papers in Obstetrics and Gynecology. Recurrent topics in Erling A. Høivik's work include Endometrial and Cervical Cancer Treatments (21 papers), Ovarian cancer diagnosis and treatment (10 papers) and Cancer Genomics and Diagnostics (8 papers). Erling A. Høivik is often cited by papers focused on Endometrial and Cervical Cancer Treatments (21 papers), Ovarian cancer diagnosis and treatment (10 papers) and Cancer Genomics and Diagnostics (8 papers). Erling A. Høivik collaborates with scholars based in Norway, United States and Thailand. Erling A. Høivik's co-authors include Camilla Krakstad, Jone Trovik, Marit Bakke, Helga B. Salvesen, Aurélia E. Lewis, Mari K. Halle, Ingfrid S. Haldorsen, Kanthida Kusonmano, Anne M. Øyan and Henrica M.J. Werner and has published in prestigious journals such as PLoS ONE, Development and Cancer Research.

In The Last Decade

Erling A. Høivik

51 papers receiving 1.6k citations

Peers

Erling A. Høivik

GENET

ONCOL

Donghai Dai United States

Lydia W.T. Cheung Hong Kong

Sung Choe United States

Dawn R. Cochrane United States

Kirsten Vang Nielsen Denmark

Jan Oosting Netherlands

T. Yano Japan

Queeny K.Y. Chan Hong Kong

Svetlana Bajalica‐Lagercrantz Sweden

Hema Parmar United States

Donghai Dai United States

Erling A. Høivik

443 ×0.5

320 ×0.6

204 ×0.6

444 ×1.4

GENET

307 ×1.0

ONCOL

Citations per year, relative to Erling A. Høivik Erling A. Høivik (= 1×) peers Donghai Dai

Countries citing papers authored by Erling A. Høivik

Since Specialization

Citations

This map shows the geographic impact of Erling A. Høivik's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Erling A. Høivik with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Erling A. Høivik more than expected).

Fields of papers citing papers by Erling A. Høivik

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Erling A. Høivik. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Erling A. Høivik. The network helps show where Erling A. Høivik may publish in the future.

Co-authorship network of co-authors of Erling A. Høivik

This figure shows the co-authorship network connecting the top 25 collaborators of Erling A. Høivik. A scholar is included among the top collaborators of Erling A. Høivik based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Erling A. Høivik. Erling A. Høivik is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Tegnander, A., et al.. (2025). Tumor necrosis associates with aggressive breast cancer features, increased hypoxia signaling and reduced patient survival. Scientific Reports. 15(1). 45518–45518.

Aas, Turid, et al.. (2025). Distinct clinicopathological features and treatment differences in breast cancer patients of young age. Scientific Reports. 15(1). 5655–5655.

Aas, Turid, Ingeborg Winge, Karin Collett, et al.. (2025). Reduced GATA3 expression associates with immuno‐metabolic alterations and aggressive features in breast cancer. The Journal of Pathology Clinical Research. 11(6). e70050–e70050.

Winge, Ingeborg, Karen Toska, May Britt Kalvenes, et al.. (2024). Elevated expression of Aurora-A/AURKA in breast cancer associates with younger age and aggressive features. Breast Cancer Research. 26(1). 126–126. 3 indexed citations

Berg, Hege F., Tomasz Stokowy, Erling A. Høivik, et al.. (2024). Expression patterns of mismatch repair proteins in cervical cancer uncover independent prognostic value of MSH-2. International Journal of Gynecological Cancer. 34(7). 993–1000. 1 indexed citations

Tegnander, A., Kristi Krüger, Erling A. Høivik, et al.. (2024). Age‐related phenotypes in breast cancer: A population‐based study. International Journal of Cancer. 154(11). 2014–2024. 4 indexed citations

Stefansson, Ingunn M., et al.. (2024). Age-Related Clusters and Favorable Immune Phenotypes in Young Breast Cancer Patients. Modern Pathology. 37(8). 100529–100529. 1 indexed citations

Krakstad, Camilla, et al.. (2023). PIK3CA mutations and their impact on survival outcomes of patients with endometrial cancer: A systematic review and meta-analysis. PLoS ONE. 18(3). e0283203–e0283203. 11 indexed citations

Berg, Hege F., Mari K. Halle, Kathrine Woie, et al.. (2022). Mismatch repair markers in preoperative and operative endometrial cancer samples; expression concordance and prognostic value. British Journal of Cancer. 128(4). 647–655. 12 indexed citations

10.

Høivik, Erling A., Erlend Hodneland, Kari S. Wagner‐Larsen, et al.. (2021). A radiogenomics application for prognostic profiling of endometrial cancer. Communications Biology. 4(1). 1363–1363. 26 indexed citations

11.

Berg, Hege F., Heidi Espedal, Aashish Srivastava, et al.. (2021). Patient-derived organoids reflect the genetic profile of endometrial tumors and predict patient prognosis. Communications Medicine. 1(1). 20–20. 36 indexed citations

12.

Berg, Hege F., Mari K. Halle, Erling A. Høivik, et al.. (2020). Maintained survival outcome after reducing lymphadenectomy rates and optimizing adjuvant treatment in endometrial cancer. Gynecologic Oncology. 160(2). 396–404. 12 indexed citations

13.

Holst, Frederik, Henrica M.J. Werner, Siv Mjøs, et al.. (2018). PIK3CA Amplification Associates with Aggressive Phenotype but Not Markers of AKT-MTOR Signaling in Endometrial Carcinoma. Clinical Cancer Research. 25(1). 334–345. 21 indexed citations

14.

Aesöy, Reidun, Haruna Muwonge, Reidun Kristin Kopperud, et al.. (2018). Deletion of exchange proteins directly activated by cAMP (Epac) causes defects in hippocampal signaling in female mice. PLoS ONE. 13(7). e0200935–e0200935. 11 indexed citations

15.

Halle, Mari K., Akinyemi I. Ojesina, Kathrine Woie, et al.. (2017). Clinicopathologic and molecular markers in cervical carcinoma: a prospective cohort study. American Journal of Obstetrics and Gynecology. 217(4). 432.e1–432.e17. 40 indexed citations

16.

Zhao, Yu, Yihua Yang, Jone Trovik, et al.. (2014). A Novel Wnt Regulatory Axis in Endometrioid Endometrial Cancer. Cancer Research. 74(18). 5103–5117. 108 indexed citations

17.

Aesöy, Reidun, Katarina Gradin, Erling A. Høivik, et al.. (2014). Regulation of CDKN2B expression by interaction of Arnt with Miz-1 - a basis for functional integration between the HIF and Myc gene regulatory pathways. Molecular Cancer. 13(1). 54–54. 9 indexed citations

18.

Wik, Elisabeth, Maria B. Ræder, Camilla Krakstad, et al.. (2013). Lack of Estrogen Receptor-α Is Associated with Epithelial–Mesenchymal Transition and PI3K Alterations in Endometrial Carcinoma. Clinical Cancer Research. 19(5). 1094–1105. 115 indexed citations

19.

Wik, Elisabeth, Even Birkeland, Jone Trovik, et al.. (2013). High Phospho-Stathmin(Serine38) Expression Identifies Aggressive Endometrial Cancer and Suggests an Association with PI3K Inhibition. Clinical Cancer Research. 19(9). 2331–2341. 35 indexed citations

20.

Ellingsen, Staale, Mary Laplante, Melanie König, et al.. (2005). Large-scale enhancer detection in the zebrafish genome. Development. 132(17). 3799–3811. 133 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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