Aurélia E. Lewis

798 citations
27 papers · 598 indexed · h-index 15
Topics
Protein Kinase Regulation and GTPase Signaling (5 papers)Cancer-related Molecular Pathways (5 papers)PI3K/AKT/mTOR signaling in cancer (4 papers)

In The Last Decade

Aurélia E. Lewis

27 papers receiving 592 citations

Peers

Aurélia E. Lewis
Comparison fields: 5 of 78
  • Molecular Biology 428
  • Genetics 159
  • Oncology 90
  • Endocrinology, Diabetes and Metabolism 73
  • Cell Biology 65
Replace Gernot Langer with:
Gernot Langer Germany
A. Misra-Press United States
Stephan Ryser Switzerland
Youji Feng China
A. Silvina Nacht Spain
E. Pötter Germany
Kerstin W. Sinkevicius United States
Anthony J. Saporita United States
Christoph Geserick Spain
Michela Bossolasco Canada
Aurélia E. Lewis relative to Gernot Langer Germany Gernot Langer's profile →
Citations per field
00.5×2.8×
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Citations per year

Countries citing papers authored by Aurélia E. Lewis

Since Specialization
Citations

This map shows the geographic impact of Aurélia E. Lewis's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Aurélia E. Lewis with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Aurélia E. Lewis more than expected).

Fields of papers citing papers by Aurélia E. Lewis

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Aurélia E. Lewis. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Aurélia E. Lewis. The network helps show where Aurélia E. Lewis may publish in the future.

Co-authorship network of co-authors of Aurélia E. Lewis

This figure shows the co-authorship network connecting the top 25 collaborators of Aurélia E. Lewis. A scholar is included among the top collaborators of Aurélia E. Lewis based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Aurélia E. Lewis. Aurélia E. Lewis is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
#WorkIndexed citations
1 1
2 6
3 13
4 14
5 23
6 23
7 15
8 2
9 31
10 5
11 92
12 3
13 121
14 32
15 36
16 27
17 16
18 9
19 1
20 7

About Aurélia E. Lewis

Aurélia E. Lewis is a scholar working on Molecular Biology, Oncology and Cell Biology, having authored 27 papers that have together received 598 indexed citations. Recurring topics across this work include Protein Kinase Regulation and GTPase Signaling (5 papers), Cancer-related Molecular Pathways (5 papers) and PI3K/AKT/mTOR signaling in cancer (4 papers). The work is most often cited by research in Molecular Biology (428 citations), Reproductive Medicine (52 citations) and Genetics (159 citations). Aurélia E. Lewis has collaborated with scholars based in Norway, United Kingdom and United States. Frequent co-authors include Marit Bakke, Erling A. Høivik, Clive S. D’Santos, Judy L. Meinkoth, Magnus Ø. Arntzen, Nullin Divecha, Nicholas A. Morrice, Yvan Strahm, Marte Rusten and Camilla Krakstad. Their work appears in journals such as Journal of Biological Chemistry, Scientific Reports and Biochemical Journal.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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