Frederik Holst

1.3k total citations
24 papers, 732 citations indexed

About

Frederik Holst is a scholar working on Genetics, Cancer Research and Molecular Biology. According to data from OpenAlex, Frederik Holst has authored 24 papers receiving a total of 732 indexed citations (citations by other indexed papers that have themselves been cited), including 11 papers in Genetics, 9 papers in Cancer Research and 8 papers in Molecular Biology. Recurrent topics in Frederik Holst's work include Estrogen and related hormone effects (8 papers), Endometrial and Cervical Cancer Treatments (6 papers) and Cancer Genomics and Diagnostics (5 papers). Frederik Holst is often cited by papers focused on Estrogen and related hormone effects (8 papers), Endometrial and Cervical Cancer Treatments (6 papers) and Cancer Genomics and Diagnostics (5 papers). Frederik Holst collaborates with scholars based in Germany, Norway and United States. Frederik Holst's co-authors include Ronald Simon, Guido Sauter, Luigi Terracciano, Annette Lebeau, F. Jänicke, Olaf Hellwinkel, Khawla S. Al‐Kuraya, Phillip Stahl, Christian Ruiz and Zeenath Jehan and has published in prestigious journals such as Nature Genetics, Journal of Clinical Oncology and Nature reviews. Cancer.

In The Last Decade

Frederik Holst

23 papers receiving 720 citations

Peers

Frederik Holst

ONCOL

GENET

Russell Broaddus United States

C. Marcelo Sergio Australia

William N. Harwin United States

Seog-Nyeon Bae South Korea

Berno Tanner Germany

Karen J. Krag United States

McGuire Wl United States

Lorenzo Ferrando Italy

R.K. Gregory United Kingdom

Gianluigi Bresciani Italy

Russell Broaddus United States

Frederik Holst

610 ×1.7

489 ×2.0

ONCOL

272 ×1.1

214 ×0.9

GENET

155 ×1.3

Citations per year, relative to Frederik Holst Frederik Holst (= 1×) peers Russell Broaddus

Countries citing papers authored by Frederik Holst

Since Specialization

Citations

This map shows the geographic impact of Frederik Holst's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Frederik Holst with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Frederik Holst more than expected).

Fields of papers citing papers by Frederik Holst

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Frederik Holst. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Frederik Holst. The network helps show where Frederik Holst may publish in the future.

Co-authorship network of co-authors of Frederik Holst

This figure shows the co-authorship network connecting the top 25 collaborators of Frederik Holst. A scholar is included among the top collaborators of Frederik Holst based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Frederik Holst. Frederik Holst is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Singer, Christian F., Frederik Holst, Stefan Steurer, et al.. (2022). Estrogen Receptor Alpha Gene Amplification Is an Independent Predictor of Long-Term Outcome in Postmenopausal Patients with Endocrine-Responsive Early Breast Cancer. Clinical Cancer Research. 28(18). 4112–4120. 10 indexed citations

Holst, Frederik, Henrica M.J. Werner, Siv Mjøs, et al.. (2018). PIK3CA Amplification Associates with Aggressive Phenotype but Not Markers of AKT-MTOR Signaling in Endometrial Carcinoma. Clinical Cancer Research. 25(1). 334–345. 21 indexed citations

Halle, Mari K., Akinyemi I. Ojesina, Kathrine Woie, et al.. (2017). Clinicopathologic and molecular markers in cervical carcinoma: a prospective cohort study. American Journal of Obstetrics and Gynecology. 217(4). 432.e1–432.e17. 40 indexed citations

Mjøs, Siv, Henrica M.J. Werner, Even Birkeland, et al.. (2017). PIK3CA exon9 mutations associate with reduced survival, and are highly concordant between matching primary tumors and metastases in endometrial cancer. Scientific Reports. 7(1). 10240–10240. 23 indexed citations

Holst, Frederik, Erling A. Høivik, William J. Gibson, et al.. (2016). Recurrent hormone-binding domain truncated ESR1 amplifications in primary endometrial cancers suggest their implication in hormone independent growth. Scientific Reports. 6(1). 25521–25521. 12 indexed citations

Holst, Frederik. (2016). Estrogen receptor alpha gene amplification in breast cancer: 25 years of debate. World Journal of Clinical Oncology. 7(2). 160–160. 18 indexed citations

Holst, Frederik. (2015). FISH Analysis of ESR1 Amplification in Breast Cancer - A Video Illustration. Figshare.

Wik, Elisabeth, Even Birkeland, Jone Trovik, et al.. (2013). High Phospho-Stathmin(Serine38) Expression Identifies Aggressive Endometrial Cancer and Suggests an Association with PI3K Inhibition. Clinical Cancer Research. 19(9). 2331–2341. 35 indexed citations

Burandt, Eike, Frederik Holst, F. Jänicke, et al.. (2013). Prognostic relevance of AIB1 (NCoA3) amplification and overexpression in breast cancer. Breast Cancer Research and Treatment. 137(3). 745–753. 33 indexed citations

10.

Moelans, Cathy B., et al.. (2013). ESR1 Amplification in Breast Cancer by Optimized RNase FISH: Frequent but Low-Level and Heterogeneous. PLoS ONE 8(12): e84189. Optical Dataset S1. Figshare. 2 indexed citations

11.

Moelans, Cathy B., Frederik Holst, Olaf Hellwinkel, Ronald Simon, & P. J. van Diest. (2013). ESR1 Amplification in Breast Cancer by Optimized RNase FISH: Frequent but Low-Level and Heterogeneous. PLoS ONE. 8(12). e84189–e84189. 15 indexed citations

12.

Birkeland, Even, Elisabeth Wik, Siv Mjøs, et al.. (2012). KRAS gene amplification and overexpression but not mutation associates with aggressive and metastatic endometrial cancer. British Journal of Cancer. 107(12). 1997–2004. 66 indexed citations

13.

Choschzick, Matthias, Pierre Tennstedt, Frederik Holst, et al.. (2012). Role of cyclin D1 amplification and expression in vulvar carcinomas. Human Pathology. 43(9). 1386–1393. 16 indexed citations

14.

Singer, Christian F., Frederik Holst, Stefan Steurer, et al.. (2012). Estrogen receptor alpha (ESR1) gene amplification status and clinical outcome in tamoxifen-treated postmenopausal patients with endocrine-responsive early breast cancer: An analysis of the prospective ABCSG-6 trial.. Journal of Clinical Oncology. 30(15_suppl). 10501–10501. 3 indexed citations

15.

Holst, Frederik, Cathy B. Moelans, Martin Filipits, et al.. (2012). On the evidence for ESR1 amplification in breast cancer. Nature reviews. Cancer. 12(2). 149–149. 19 indexed citations

16.

Holst, Frederik. (2012). ESR1-Amplifikationen in humanen gynäkologischen Tumoren. 1 indexed citations

17.

Dancau, Ana‐Maria, Frederik Holst, Antje Krohn, et al.. (2009). PPFIA1 and CCND1 are frequently coamplified in breast cancer. Genes Chromosomes and Cancer. 49(1). 1–8. 21 indexed citations

18.

Lebeau, Annette, Tobias Grob, Frederik Holst, et al.. (2008). Estrogen receptor gene amplification occurs rarely in ovarian cancer. Modern Pathology. 22(2). 191–196. 23 indexed citations

19.

Lebeau, Annette, Tobias Grob, Frederik Holst, et al.. (2008). Oestrogen receptor gene (ESR1) amplification is frequent in endometrial carcinoma and its precursor lesions. The Journal of Pathology. 216(2). 151–157. 30 indexed citations

20.

Holst, Frederik, Phillip Stahl, Christian Ruiz, et al.. (2007). Estrogen receptor alpha (ESR1) gene amplification is frequent in breast cancer. Nature Genetics. 39(5). 655–660. 286 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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