Entela Bua

1.3k total citations
8 papers, 1.0k citations indexed

About

Entela Bua is a scholar working on Molecular Biology, Clinical Biochemistry and Cellular and Molecular Neuroscience. According to data from OpenAlex, Entela Bua has authored 8 papers receiving a total of 1.0k indexed citations (citations by other indexed papers that have themselves been cited), including 8 papers in Molecular Biology, 3 papers in Clinical Biochemistry and 2 papers in Cellular and Molecular Neuroscience. Recurrent topics in Entela Bua's work include Mitochondrial Function and Pathology (7 papers), Muscle Physiology and Disorders (4 papers) and Metabolism and Genetic Disorders (3 papers). Entela Bua is often cited by papers focused on Mitochondrial Function and Pathology (7 papers), Muscle Physiology and Disorders (4 papers) and Metabolism and Genetic Disorders (3 papers). Entela Bua collaborates with scholars based in United States. Entela Bua's co-authors include Judd M. Aiken, Debbie McKenzie, Susan H. McKiernan, Allen Herbst, Jody Johnson, Shahriar Salamat, Jonathan Wanagat, Jeong W. Pak, Mohamed Bassiouni and Zhengjin Cao and has published in prestigious journals such as The FASEB Journal, Journal of Applied Physiology and Annals of the New York Academy of Sciences.

In The Last Decade

Entela Bua

8 papers receiving 989 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Entela Bua United States 8 788 508 192 138 133 8 1.0k
Giulia Favaro Italy 3 569 0.7× 353 0.7× 70 0.4× 18 0.1× 55 0.4× 4 719
Jonathan M. Memme Canada 11 520 0.7× 515 1.0× 27 0.1× 29 0.2× 40 0.3× 15 882
Matthew Triolo Canada 10 508 0.6× 430 0.8× 22 0.1× 28 0.2× 32 0.2× 15 780
Mette Cathrine Ørngreen Denmark 21 580 0.7× 344 0.7× 367 1.9× 6 0.0× 206 1.5× 52 1.1k
T. Bradley Willingham United States 13 410 0.5× 240 0.5× 60 0.3× 15 0.1× 62 0.5× 26 711
Arkan Abadi Canada 10 445 0.6× 269 0.5× 29 0.2× 18 0.1× 29 0.2× 14 715
Julianna Huang Canada 5 456 0.6× 408 0.8× 19 0.1× 20 0.1× 25 0.2× 7 603
Ashley N. Oliveira Canada 10 404 0.5× 396 0.8× 14 0.1× 22 0.2× 30 0.2× 17 675
Avigail T. Erlich Canada 11 478 0.6× 451 0.9× 20 0.1× 15 0.1× 25 0.2× 13 793
Hans P.M.M. Lauritzen United States 15 601 0.8× 502 1.0× 18 0.1× 11 0.1× 34 0.3× 17 998

Countries citing papers authored by Entela Bua

Since Specialization
Citations

This map shows the geographic impact of Entela Bua's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Entela Bua with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Entela Bua more than expected).

Fields of papers citing papers by Entela Bua

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Entela Bua. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Entela Bua. The network helps show where Entela Bua may publish in the future.

Co-authorship network of co-authors of Entela Bua

This figure shows the co-authorship network connecting the top 25 collaborators of Entela Bua. A scholar is included among the top collaborators of Entela Bua based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Entela Bua. Entela Bua is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

8 of 8 papers shown
1.
Herbst, Allen, Jeong W. Pak, Debbie McKenzie, et al.. (2007). Accumulation of Mitochondrial DNA Deletion Mutations in Aged Muscle Fibers: Evidence for a Causal Role in Muscle Fiber Loss. The Journals of Gerontology Series A. 62(3). 235–245. 153 indexed citations
2.
Bua, Entela, Jody Johnson, Allen Herbst, et al.. (2006). Mitochondrial DNA–Deletion Mutations Accumulate Intracellularly to Detrimental Levels in Aged Human Skeletal Muscle Fibers. The American Journal of Human Genetics. 79(3). 469–480. 337 indexed citations
3.
Bua, Entela, Susan H. McKiernan, & Judd M. Aiken. (2004). Calorie restriction limits the generation but not the progression of mitochondrial abnormalities in aging skeletal muscle. The FASEB Journal. 18(3). 582–584. 44 indexed citations
4.
McKiernan, Susan H., et al.. (2004). Early‐onset calorie restriction conserves fiber number in aging rat skeletal muscle. The FASEB Journal. 18(3). 580–581. 70 indexed citations
5.
Pak, Jeong W., Allen Herbst, Entela Bua, et al.. (2003). Mitochondrial DNA mutations as a fundamental mechanism in physiological declines associated with aging. Aging Cell. 2(1). 1–7. 68 indexed citations
6.
McKenzie, Debbie, et al.. (2002). Mitochondrial DNA deletion mutations. European Journal of Biochemistry. 269(8). 2010–2015. 93 indexed citations
7.
Aiken, Judd M., Entela Bua, Zhengjin Cao, et al.. (2002). Mitochondrial DNA Deletion Mutations and Sarcopenia. Annals of the New York Academy of Sciences. 959(1). 412–423. 70 indexed citations
8.
Bua, Entela, Susan H. McKiernan, Jonathan Wanagat, Debbie McKenzie, & Judd M. Aiken. (2002). Mitochondrial abnormalities are more frequent in muscles undergoing sarcopenia. Journal of Applied Physiology. 92(6). 2617–2624. 174 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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