Emily M. Nakada
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- IL-33, ST2, and ILC Pathways 5
- Immune Cell Function and Interaction 4
- Immune Response and Inflammation 2
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- Allergic Rhinitis and Sensitization 3
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- Asthma and respiratory diseases 6
- Emergency Medical Services top 10%
- Pediatric health and respiratory diseases 4
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- Endoplasmic Reticulum Stress and Disease 5
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- Glioma Diagnosis and Treatment 2
- Co-authors
- Elizabeth D. FixmanJichuan ShanSalman T. QureshiJames G. MartinNicolas ChamberlainVikas AnathySidra M. HoffmanDavid G. Chapman
- Journals
- The Journal of Immunology (3 papers)The Journal of Physiology (1 paper)International Journal of Molecular Sciences (2 papers)
- Partner nations
- CanadaUnited StatesAustralia
In The Last Decade
Emily M. Nakada
17 papers receiving 351 citations
Peers
Comparison fields: 5 of 73
- Immunology 130
- Immunology and Allergy 36
- Physiology 145
- Emergency Medical Services 32
- Cell Biology 57
Countries citing papers authored by Emily M. Nakada
This map shows the geographic impact of Emily M. Nakada's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Emily M. Nakada with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Emily M. Nakada more than expected).
Fields of papers citing papers by Emily M. Nakada
This network shows the impact of papers produced by Emily M. Nakada. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Emily M. Nakada. The network helps show where Emily M. Nakada may publish in the future.
Co-authorship network
The 25 scholars most cited alongside Emily M. Nakada, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2025 | 0 | |
| 2 | 2025 | 1 | |
| 3 | 2023 | 3 | |
| 4 | 2023 | 1 | |
| 5 | 2022 | 3 | |
| 6 | 2022 | 7 | |
| 7 | 2021 | 13 | |
| 8 | 2021 | 15 | |
| 9 | 2019 | 49 | |
| 10 | 2019 | 49 | |
| 11 | 2019 | 12 | |
| 12 | 2019 | 1 | |
| 13 | 2016 | 35 | |
| 14 | 2014 | 33 | |
| 15 | 2014 | 17 | |
| 16 | 2013 | 55 | |
| 17 | 2013 | 40 | |
| 18 | 2012 | 21 |
About Emily M. Nakada
Emily M. Nakada is a scholar working on Emergency Medical Services, Immunology and Allergy and Immunology, having authored 18 papers that have together received 355 indexed citations. Recurring topics across this work include Asthma and respiratory diseases (6 papers), Endoplasmic Reticulum Stress and Disease (5 papers), IL-33, ST2, and ILC Pathways (5 papers), Pediatric health and respiratory diseases (4 papers), Immune Cell Function and Interaction (4 papers), Allergic Rhinitis and Sensitization (3 papers), Immune Response and Inflammation (2 papers) and Glioma Diagnosis and Treatment (2 papers). The work is most often cited by research in Immunology (130 citations), Immunology and Allergy (36 citations) and Physiology (145 citations). Emily M. Nakada has collaborated with scholars based in Canada, United States and Australia. Frequent co-authors include Elizabeth D. Fixman, Jichuan Shan, Salman T. Qureshi, James G. Martin, Nicolas Chamberlain, Vikas Anathy, Sidra M. Hoffman, David G. Chapman, Karim H. Shalaby and Brian J. Day. Their work appears in journals such as The Journal of Immunology, The Journal of Physiology and International Journal of Molecular Sciences.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.