Emily G. Atkinson

433 total citations
18 papers, 326 citations indexed

About

Emily G. Atkinson is a scholar working on Molecular Biology, Oncology and Orthopedics and Sports Medicine. According to data from OpenAlex, Emily G. Atkinson has authored 18 papers receiving a total of 326 indexed citations (citations by other indexed papers that have themselves been cited), including 14 papers in Molecular Biology, 8 papers in Oncology and 5 papers in Orthopedics and Sports Medicine. Recurrent topics in Emily G. Atkinson's work include Bone Metabolism and Diseases (11 papers), Bone health and treatments (8 papers) and Bone health and osteoporosis research (4 papers). Emily G. Atkinson is often cited by papers focused on Bone Metabolism and Diseases (11 papers), Bone health and treatments (8 papers) and Bone health and osteoporosis research (4 papers). Emily G. Atkinson collaborates with scholars based in United States, Spain and Australia. Emily G. Atkinson's co-authors include Jesús Delgado‐Calle, Lilian I. Plotkin, Rafael Pacheco‐Costa, Teresita Bellido, Hannah M. Davis, Mircea Ivan, Toshiyuki Yoneda, Arancha R. Gortázar, Lucas R. Brun and Angela Bruzzaniti and has published in prestigious journals such as Blood, Scientific Reports and Journal of Bone and Mineral Research.

In The Last Decade

Emily G. Atkinson

17 papers receiving 325 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Emily G. Atkinson United States 9 224 76 63 48 48 18 326
Jenny Fung Ling Chau Singapore 6 229 1.0× 125 1.6× 23 0.4× 44 0.9× 37 0.8× 7 324
Chung-Wen Kuo Taiwan 11 193 0.9× 41 0.5× 38 0.6× 67 1.4× 89 1.9× 18 357
Mitsuyasu Iwasawa Japan 9 227 1.0× 112 1.5× 60 1.0× 11 0.2× 66 1.4× 15 328
Barbara Haigl Austria 8 231 1.0× 80 1.1× 64 1.0× 16 0.3× 81 1.7× 10 324
Xiangde Zhao China 12 310 1.4× 99 1.3× 42 0.7× 16 0.3× 108 2.3× 18 423
Alanna C. Green Australia 11 165 0.7× 60 0.8× 41 0.7× 9 0.2× 24 0.5× 16 283
Shino Kondoh Japan 5 262 1.2× 130 1.7× 133 2.1× 36 0.8× 23 0.5× 8 406
Simin Lin China 9 137 0.6× 32 0.4× 15 0.2× 42 0.9× 43 0.9× 17 236
Jianguo Tao United States 9 128 0.6× 76 1.0× 19 0.3× 22 0.5× 16 0.3× 16 275
Claire-Sophie Devignes France 6 158 0.7× 77 1.0× 16 0.3× 23 0.5× 80 1.7× 8 308

Countries citing papers authored by Emily G. Atkinson

Since Specialization
Citations

This map shows the geographic impact of Emily G. Atkinson's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Emily G. Atkinson with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Emily G. Atkinson more than expected).

Fields of papers citing papers by Emily G. Atkinson

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Emily G. Atkinson. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Emily G. Atkinson. The network helps show where Emily G. Atkinson may publish in the future.

Co-authorship network of co-authors of Emily G. Atkinson

This figure shows the co-authorship network connecting the top 25 collaborators of Emily G. Atkinson. A scholar is included among the top collaborators of Emily G. Atkinson based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Emily G. Atkinson. Emily G. Atkinson is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

18 of 18 papers shown
1.
Atkinson, Emily G., Daniel J. Horan, Teresita Bellido, et al.. (2023). Loss of Nmp4 enhances bone gain from sclerostin antibody administration. Bone. 177. 116891–116891. 4 indexed citations
2.
Atkinson, Emily G., Ronald C. Wek, Deepak Vashishth, et al.. (2023). NMP4, an Arbiter of Bone Cell Secretory Capacity and Regulator of Skeletal Response to PTH Therapy. Calcified Tissue International. 113(1). 110–125. 1 indexed citations
3.
Nguyen, Ngoc Khoi, Jue Chen, Emily G. Atkinson, et al.. (2022). Facile de Novo Sequencing of Tetrazine‐Cyclized Peptides through UV‐Induced Ring‐Opening and Cleavage from the Solid Phase. ChemBioChem. 24(4). e202200590–e202200590. 7 indexed citations
4.
Bidwell, Joseph P., Sarah A. Tersey, Robert N. Bone, et al.. (2021). Nmp4, a Regulator of Induced Osteoanabolism, Also Influences Insulin Secretion and Sensitivity. Calcified Tissue International. 110(2). 244–259. 3 indexed citations
5.
Atkinson, Emily G., et al.. (2021). Neural bases of risky decisions involving nicotine vapor versus monetary reward. NeuroImage Clinical. 32. 102869–102869.
6.
Atkinson, Emily G., Daniel J. Horan, Teresita Bellido, et al.. (2020). Conditional Loss of Nmp4 in Mesenchymal Stem Progenitor Cells Enhances PTH-Induced Bone Formation. Journal of Bone and Mineral Research. 38(1). 70–85. 8 indexed citations
7.
Atkinson, Emily G. & Jesús Delgado‐Calle. (2019). The Emerging Role of Osteocytes in Cancer in Bone. PMC. 1 indexed citations
8.
Aguilar‐Perez, Alexandra, Rafael Pacheco‐Costa, Emily G. Atkinson, et al.. (2019). Age- and sex-dependent role of osteocytic pannexin1 on bone and muscle mass and strength. Scientific Reports. 9(1). 13903–13903. 18 indexed citations
9.
Davis, Hannah M., Rafael Pacheco‐Costa, Emily G. Atkinson, et al.. (2019). Osteocytic miR21 deficiency improves bone strength independent of sex despite having sex divergent effects on osteocyte viability and bone turnover. FEBS Journal. 287(5). 941–963. 13 indexed citations
10.
Atkinson, Emily G. & Jesús Delgado‐Calle. (2019). The Emerging Role of Osteocytes in Cancer in Bone. JBMR Plus. 3(3). e10186–e10186. 43 indexed citations
11.
Delgado‐Calle, Jesús, Noriyoshi Kurihara, Emily G. Atkinson, et al.. (2019). Aplidin (plitidepsin) is a novel anti-myeloma agent with potent anti-resorptive activity mediated by direct effects on osteoclasts. Oncotarget. 10(28). 2709–2721. 28 indexed citations
12.
Davis, Hannah M., Mohammad W. Aref, Alexandra Aguilar‐Perez, et al.. (2018). Cx43 Overexpression in Osteocytes Prevents Osteocyte Apoptosis and Preserves Cortical Bone Quality in Aging Mice. JBMR Plus. 2(4). 206–216. 45 indexed citations
13.
Pacheco‐Costa, Rafael, Hannah M. Davis, Emily G. Atkinson, et al.. (2018). Reversal of loss of bone mass in old mice treated with mefloquine. Bone. 114. 22–31. 9 indexed citations
15.
Delgado‐Calle, Jesús, et al.. (2017). Aplidin (Plitidepsin) Regulates Viability and Function of Myeloma Cells and Bone Cells in Combination with Other Anti-MM Drugs. Blood. 130. 3065–3065. 1 indexed citations
16.
Pacheco‐Costa, Rafael, et al.. (2017). Connexin37 deficiency alters organic bone matrix, cortical bone geometry, and increases Wnt/β-catenin signaling. Bone. 97. 105–113. 18 indexed citations
17.
Davis, Hannah M., Rafael Pacheco‐Costa, Emily G. Atkinson, et al.. (2017). Disruption of the Cx43/miR21 pathway leads to osteocyte apoptosis and increased osteoclastogenesis with aging. Aging Cell. 16(3). 551–563. 114 indexed citations
18.
Pacheco‐Costa, Rafael, Hannah M. Davis, Emily G. Atkinson, et al.. (2016). Osteocytic connexin 43 is not required for the increase in bone mass induced by intermittent PTH administration in male mice.. PubMed. 16(1). 45–57. 12 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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