Elisabetta Argenzio
- Molecular Biology top 10%
- Cell Biology top 2%
- Oncology
- Immunology and Allergy top 5%
- Radiology, Nuclear Medicine and Imaging
- Co-authors
- Simona PoloPier Paolo Di FioreSara SigismundDaniela TosoniElena CavallaroWouter H. MoolenaarKees JalinkDaniela Leyton-Puig
- Topics
- Ubiquitin and proteasome pathways (4 papers)Glycosylation and Glycoproteins Research (3 papers)Ion channel regulation and function (3 papers)
- Partner nations
- NetherlandsItalyUnited States
In The Last Decade
Elisabetta Argenzio
14 papers receiving 1.0k citations
Hit Papers
Peers
Comparison fields: 5 of 86
- Molecular Biology 723
- Cell Biology 461
- Oncology 183
- Immunology and Allergy 113
- Radiology, Nuclear Medicine and Imaging 110
Countries citing papers authored by Elisabetta Argenzio
This map shows the geographic impact of Elisabetta Argenzio's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Elisabetta Argenzio with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Elisabetta Argenzio more than expected).
Fields of papers citing papers by Elisabetta Argenzio
This network shows the impact of papers produced by Elisabetta Argenzio. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Elisabetta Argenzio. The network helps show where Elisabetta Argenzio may publish in the future.
Co-authorship network of co-authors of Elisabetta Argenzio
This figure shows the co-authorship network connecting the top 25 collaborators of Elisabetta Argenzio. A scholar is included among the top collaborators of Elisabetta Argenzio based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Elisabetta Argenzio. Elisabetta Argenzio is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 4 | |
| 2 | 13 | |
| 3 | 75 | |
| 4 | 77 | |
| 5 | 35 | |
| 6 | 12 | |
| 7 | 67 | |
| 8 | 46 | |
| 9 | 135 | |
| 10 | 52 | |
| 11 | Clathrin-Mediated Internalization Is Essential for Sustained EGFR Signaling but Dispensable for Degradationbreakdown → | 506 |
| 12 | 7 | |
| 13 | 1 | |
| 14 | 12 |
About Elisabetta Argenzio
Elisabetta Argenzio is a scholar working on Cell Biology, Sensory Systems and Molecular Biology, having authored 14 papers that have together received 1.0k indexed citations. Recurring topics across this work include Ubiquitin and proteasome pathways (4 papers), Glycosylation and Glycoproteins Research (3 papers) and Ion channel regulation and function (3 papers). The work is most often cited by research in Cell Biology (461 citations), Immunology and Allergy (113 citations) and Molecular Biology (723 citations). Elisabetta Argenzio has collaborated with scholars based in Netherlands, Italy and United States. Frequent co-authors include Simona Polo, Pier Paolo Di Fiore, Sara Sigismund, Daniela Tosoni, Elena Cavallaro, Wouter H. Moolenaar, Kees Jalink, Daniela Leyton-Puig, Metello Innocenti and Andrea Ciliberto. Their work appears in journals such as Journal of Biological Chemistry, Nature Communications and The EMBO Journal.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.