David J. Elzi

2.6k total citations
30 papers, 2.1k citations indexed

About

David J. Elzi is a scholar working on Molecular Biology, Physiology and Immunology. According to data from OpenAlex, David J. Elzi has authored 30 papers receiving a total of 2.1k indexed citations (citations by other indexed papers that have themselves been cited), including 14 papers in Molecular Biology, 11 papers in Physiology and 9 papers in Immunology. Recurrent topics in David J. Elzi's work include Neutrophil, Myeloperoxidase and Oxidative Mechanisms (8 papers), Immune Response and Inflammation (6 papers) and Erythrocyte Function and Pathophysiology (5 papers). David J. Elzi is often cited by papers focused on Neutrophil, Myeloperoxidase and Oxidative Mechanisms (8 papers), Immune Response and Inflammation (6 papers) and Erythrocyte Function and Pathophysiology (5 papers). David J. Elzi collaborates with scholars based in United States, Slovakia and South Africa. David J. Elzi's co-authors include Christopher C. Silliman, Daniel R. Ambruso, Marguerite R. Kelher, Gwen Clarke, Linda Podlosky, Daniel R. Ambruso, Lynn K. Boshkov, Yuzuru Shiio, N. F. Voelkel and Rubin M. Tuder and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Clinical Investigation and The EMBO Journal.

In The Last Decade

David J. Elzi

30 papers receiving 2.0k citations

Peers

David J. Elzi
Wolfgang Bergmeier United States
Simon Allen United Kingdom
Mark E. Diebel United States
K. Vinod Vijayan United States
Anna Kobsar Germany
Yulia Kiyan Germany
Stephen J. Klaus United States
Josefin Ahnström United Kingdom
Wolfgang Bergmeier United States
David J. Elzi
Citations per year, relative to David J. Elzi David J. Elzi (= 1×) peers Wolfgang Bergmeier

Countries citing papers authored by David J. Elzi

Since Specialization
Citations

This map shows the geographic impact of David J. Elzi's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by David J. Elzi with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites David J. Elzi more than expected).

Fields of papers citing papers by David J. Elzi

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by David J. Elzi. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by David J. Elzi. The network helps show where David J. Elzi may publish in the future.

Co-authorship network of co-authors of David J. Elzi

This figure shows the co-authorship network connecting the top 25 collaborators of David J. Elzi. A scholar is included among the top collaborators of David J. Elzi based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with David J. Elzi. David J. Elzi is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Elzi, David J., et al.. (2021). Identification of a novel mechanism for meso‐tetra (4‐carboxyphenyl) porphyrin (TCPP) uptake in cancer cells. The FASEB Journal. 35(3). e21427–e21427. 9 indexed citations
2.
Zhou, Fuchun, David J. Elzi, Xiuye Ma, et al.. (2020). GDF6-CD99 Signaling Regulates Src and Ewing Sarcoma Growth. Cell Reports. 33(5). 108332–108332. 22 indexed citations
3.
Kelher, Marguerite R., Nathan McLaughlin, Anirban Banerjee, et al.. (2016). LysoPCs induce Hck- and PKCδ-mediated activation of PKCγ causing p47phox phosphorylation and membrane translocation in neutrophils. Journal of Leukocyte Biology. 101(1). 261–273. 15 indexed citations
4.
Elzi, David J., Meihua Song, & Yuzuru Shiio. (2015). Role of galactose in cellular senescence. Experimental Gerontology. 73. 1–4. 21 indexed citations
5.
Elzi, David J., Meihua Song, Kevin Hakala, Susan T. Weintraub, & Yuzuru Shiio. (2012). Wnt Antagonist SFRP1 Functions as a Secreted Mediator of Senescence. Molecular and Cellular Biology. 32(21). 4388–4399. 66 indexed citations
6.
Elzi, David J., Yanlai Lai, Meihua Song, et al.. (2012). Plasminogen activator inhibitor 1 - insulin-like growth factor binding protein 3 cascade regulates stress-induced senescence. Proceedings of the National Academy of Sciences. 109(30). 12052–12057. 118 indexed citations
7.
Silliman, Christopher C., Ernest E. Moore, Marguerite R. Kelher, et al.. (2011). Identification of lipids that accumulate during the routine storage of prestorage leukoreduced red blood cells and cause acute lung injury. Transfusion. 51(12). 2549–2554. 134 indexed citations
8.
Moore, Ernest E., Anirban Banerjee, Marguerite R. Kelher, et al.. (2010). Leukotriene B4 and its Metabolites Prime the Neutrophil Oxidase and Induce Proinflammatory Activation of Human Pulmonary Microvascular Endothelial Cells. Shock. 35(3). 240–244. 21 indexed citations
9.
Oh, Myungjin, Tae Beom Seo, Sung‐Jin Yoon, et al.. (2009). Axonal Outgrowth and Erk1/2 Activation by Training after Spinal Cord Injury in Rats. Journal of Neurotrauma. 26(11). 2071–2082. 33 indexed citations
10.
Oh, Myungjin, et al.. (2009). Axonal outgrowth and Erk1/2 activation by training after spinal cord injury in rats. Journal of Neurotrauma. 2852359181–2852359181. 3 indexed citations
11.
Xu, Mei, Weifeng Luo, David J. Elzi, Carla Grandori, & Denise A. Galloway. (2008). NFX1 Interacts with mSin3A/Histone Deacetylase To Repress hTERT Transcription in Keratinocytes. Molecular and Cellular Biology. 28(15). 4819–4828. 55 indexed citations
12.
Weber, Axel, David J. Elzi, Nicole Forster, et al.. (2008). Zbtb4 represses transcription of P21CIP1 and controls the cellular response to p53 activation. The EMBO Journal. 27(11). 1563–1574. 82 indexed citations
13.
Kelher, Marguerite R., Daniel R. Ambruso, David J. Elzi, et al.. (2003). Formyl-Met-Leu-Phe induces calcium-dependent tyrosine phosphorylation of Rel-1 in neutrophils. Cell Calcium. 34(6). 445–455. 18 indexed citations
14.
Silliman, Christopher C., Lynn K. Boshkov, David J. Elzi, et al.. (2002). Transfusion-related acute lung injury: epidemiology and a prospective analysis of etiologic factors. Blood. 101(2). 454–462. 451 indexed citations
15.
Wyman, Travis H., David J. Elzi, C. Wayne Smith, et al.. (2002). A two-insult in vitro model of PMN-mediated pulmonary endothelial damage: requirements for adherence and chemokine release. American Journal of Physiology-Cell Physiology. 283(6). C1592–C1603. 130 indexed citations
16.
Elzi, David J., et al.. (2001). Ionomycin causes activation of p38 and p42/44 mitogen-activated protein kinases in human neutrophils. American Journal of Physiology-Cell Physiology. 281(1). C350–C360. 55 indexed citations
17.
Silliman, Christopher C., N. F. Voelkel, David J. Elzi, et al.. (1998). Plasma and lipids from stored packed red blood cells cause acute lung injury in an animal model.. Journal of Clinical Investigation. 101(7). 1458–1467. 381 indexed citations
18.
Leavey, Patrick J., Karen S. Sellins, Gail Thurman, et al.. (1998). In Vivo Treatment With Granulocyte Colony-Stimulating Factor Results in Divergent Effects on Neutrophil Functions Measured In Vitro. Blood. 92(11). 4366–4374. 62 indexed citations
19.
Barnett, Carlton C., Ernest E. Moore, Gary W. Mierau, et al.. (1998). ICAM-1-CD18 interaction mediates neutrophil cytotoxicity through protease release. American Journal of Physiology-Cell Physiology. 274(6). C1634–C1644. 49 indexed citations
20.
Elzi, David J., Andrew Hiester, & Christopher C. Silliman. (1997). Receptor-Mediated Calcium Entry Is Required for Maximal Effects of Platelet Activating Factor Primed Responses in Human Neutrophils. Biochemical and Biophysical Research Communications. 240(3). 763–765. 24 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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