Edward T. W. Bampton
- Physiology top 5%
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- Nerve injury and regeneration 6
- Neuroscience and Neuropharmacology Research 2
- Developmental Neuroscience top 10%
- Epidemiology top 5%
- Autophagy in Disease and Therapy 3
- Cell Biology top 10%
- Endoplasmic Reticulum Stress and Disease 2
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- Cell death mechanisms and regulation 3
- Extracellular vesicles in disease 2
- Mitochondrial Function and Pathology 2
- ATP Synthase and ATPases Research 2
- Co-authors
- Aviva M. TolkovskyGerald M. CohenChristoph G. GoemansDhevahi NiranjanNoboru MizushimaJ. S. H. TaylorLaura F. GumyDavid Dinsdale
- Partner nations
- United KingdomUnited StatesItaly
In The Last Decade
Edward T. W. Bampton
18 papers receiving 1.2k citations
Peers
Comparison fields: 5 of 105
- Physiology 103
- Cellular and Molecular Neuroscience 269
- Developmental Neuroscience 60
- Epidemiology 495
- Cell Biology 214
Countries citing papers authored by Edward T. W. Bampton
This map shows the geographic impact of Edward T. W. Bampton's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Edward T. W. Bampton with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Edward T. W. Bampton more than expected).
Fields of papers citing papers by Edward T. W. Bampton
This network shows the impact of papers produced by Edward T. W. Bampton. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Edward T. W. Bampton. The network helps show where Edward T. W. Bampton may publish in the future.
Co-authorship network
The 25 scholars most cited alongside Edward T. W. Bampton, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2013 | 23 | |
| 2 | 2012 | 58 | |
| 3 | 2012 | 5 | |
| 4 | 2011 | 152 | |
| 5 | 2010 | 167 | |
| 6 | 2010 | 30 | |
| 7 | 2009 | 19 | |
| 8 | 2009 | 24 | |
| 9 | 2009 | 100 | |
| 10 | 2007 | 126 | |
| 11 | 2007 | 34 | |
| 12 | 2005 | 321 | |
| 13 | 2005 | 40 | |
| 14 | 2005 | 30 | |
| 15 | 2003 | 36 | |
| 16 | 2001 | 27 | |
| 17 | 1999 | 24 | |
| 18 | 1999 | 43 |
About Edward T. W. Bampton
Edward T. W. Bampton is a scholar working on Developmental Neuroscience, Cellular and Molecular Neuroscience and Cell Biology, having authored 18 papers that have together received 1.3k indexed citations. Recurring topics across this work include Nerve injury and regeneration (6 papers), Autophagy in Disease and Therapy (3 papers), Cell death mechanisms and regulation (3 papers), Extracellular vesicles in disease (2 papers), Mitochondrial Function and Pathology (2 papers), ATP Synthase and ATPases Research (2 papers), Neuroscience and Neuropharmacology Research (2 papers) and Endoplasmic Reticulum Stress and Disease (2 papers). The work is most often cited by research in Physiology (103 citations), Cellular and Molecular Neuroscience (269 citations) and Developmental Neuroscience (60 citations). Edward T. W. Bampton has collaborated with scholars based in United Kingdom, United States and Italy. Frequent co-authors include Aviva M. Tolkovsky, Gerald M. Cohen, Christoph G. Goemans, Dhevahi Niranjan, Noboru Mizushima, J. S. H. Taylor, Laura F. Gumy, David Dinsdale, Pierluigi Nicotera and Hassan A. Hamali. Their work appears in journals such as Blood, PLoS ONE and Brain Research.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.