Yakup Batlevi

2.0k total citations · 1 hit paper
7 papers, 1.4k citations indexed

About

Yakup Batlevi is a scholar working on Epidemiology, Molecular Biology and Cell Biology. According to data from OpenAlex, Yakup Batlevi has authored 7 papers receiving a total of 1.4k indexed citations (citations by other indexed papers that have themselves been cited), including 6 papers in Epidemiology, 3 papers in Molecular Biology and 2 papers in Cell Biology. Recurrent topics in Yakup Batlevi's work include Autophagy in Disease and Therapy (6 papers), Ubiquitin and proteasome pathways (2 papers) and Histone Deacetylase Inhibitors Research (2 papers). Yakup Batlevi is often cited by papers focused on Autophagy in Disease and Therapy (6 papers), Ubiquitin and proteasome pathways (2 papers) and Histone Deacetylase Inhibitors Research (2 papers). Yakup Batlevi collaborates with scholars based in United States and Brazil. Yakup Batlevi's co-authors include Eric H. Baehrecke, Albert R. La Spada, J. Paul Taylor, Deborah L. Berry, Melanie A. Knight, Oren Schuldiner, Natalia B. Nedelsky, Brett A. McCray, Tso-Pang Yao and Marc Hild and has published in prestigious journals such as Nature, Proceedings of the National Academy of Sciences and Nature Neuroscience.

In The Last Decade

Yakup Batlevi

6 papers receiving 1.4k citations

Hit Papers

HDAC6 rescues neurodegeneration and provides an essential... 2007 2026 2013 2019 2007 250 500 750

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Yakup Batlevi United States 6 878 660 334 288 251 7 1.4k
Misako Okuno Japan 12 874 1.0× 550 0.8× 428 1.3× 295 1.0× 227 0.9× 14 1.4k
Conrad C. Weihl United States 15 885 1.0× 970 1.5× 495 1.5× 158 0.5× 318 1.3× 17 1.7k
Hongfeng Wang China 25 1.0k 1.1× 651 1.0× 289 0.9× 316 1.1× 727 2.9× 48 1.8k
Laura C. Bott United States 10 812 0.9× 320 0.5× 370 1.1× 252 0.9× 179 0.7× 18 1.1k
Jeanne M.M. Tan Singapore 14 950 1.1× 594 0.9× 337 1.0× 352 1.2× 542 2.2× 17 1.6k
Eszter Zavodszky United Kingdom 12 743 0.8× 456 0.7× 480 1.4× 191 0.7× 231 0.9× 15 1.3k
Mei-Yao Lin United States 12 695 0.8× 253 0.4× 147 0.4× 219 0.8× 141 0.6× 14 1.2k
M.E. Cicardi Italy 18 659 0.8× 431 0.7× 319 1.0× 244 0.8× 438 1.7× 29 1.3k
Clara Penas Spain 19 759 0.9× 243 0.4× 248 0.7× 272 0.9× 92 0.4× 34 1.3k
Roger Belizaire United States 16 740 0.8× 194 0.3× 295 0.9× 400 1.4× 577 2.3× 30 1.7k

Countries citing papers authored by Yakup Batlevi

Since Specialization
Citations

This map shows the geographic impact of Yakup Batlevi's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Yakup Batlevi with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Yakup Batlevi more than expected).

Fields of papers citing papers by Yakup Batlevi

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Yakup Batlevi. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Yakup Batlevi. The network helps show where Yakup Batlevi may publish in the future.

Co-authorship network of co-authors of Yakup Batlevi

This figure shows the co-authorship network connecting the top 25 collaborators of Yakup Batlevi. A scholar is included among the top collaborators of Yakup Batlevi based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Yakup Batlevi. Yakup Batlevi is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

7 of 7 papers shown
1.
Jain, Tania, Craig S. Sauter, Gunjan L. Shah, et al.. (2019). Safety and Feasibility of Chimeric Antigen Receptor T Cell Therapy after Allogeneic Stem Cell Therapy in Relapsed/ Refractory B Cell Lymphomas. Biology of Blood and Marrow Transplantation. 25(3). S183–S184.
2.
Cortés, Constanza J., Helen C. Miranda, Harald Frankowski, et al.. (2014). Polyglutamine-expanded androgen receptor interferes with TFEB to elicit autophagy defects in SBMA. Nature Neuroscience. 17(9). 1180–1189. 138 indexed citations
3.
McPhee, Christina, Brian M. Balgley, Jahda H. Hill, et al.. (2012). Identification of factors that function in Drosophila salivary gland cell death during development using proteomics. Cell Death and Differentiation. 20(2). 218–225. 13 indexed citations
4.
Batlevi, Yakup & Albert R. La Spada. (2010). Mitochondrial autophagy in neural function, neurodegenerative disease, neuron cell death, and aging. Neurobiology of Disease. 43(1). 46–51. 107 indexed citations
5.
Batlevi, Yakup, Damali N. Martin, Claudio Simon, et al.. (2009). Dynein light chain 1 is required for autophagy, protein clearance, and cell death inDrosophila. Proceedings of the National Academy of Sciences. 107(2). 742–747. 47 indexed citations
6.
Nie, Zhiping, Yakup Batlevi, Brett A. McCray, et al.. (2007). HDAC6 rescues neurodegeneration and provides an essential link between autophagy and the UPS. Nature. 447(7146). 860–864. 979 indexed citations breakdown →
7.
Batlevi, Yakup, et al.. (2007). HDAC6 at the Intersection of Autophagy, the Ubiquitin-proteasome System, and Neurodegeneration. Autophagy. 3(6). 643–645. 97 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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